Platelets convert peripheral blood circulating monocytes to regulatory cells via immunoglobulin G and activating-type Fcγ receptors
Main Authors: | Inui, Masanori, Tazawa, Kino, Kishi, Yoshiro, Takai, Toshiyuki |
---|---|
Format: | Online |
Language: | English |
Published: |
BioMed Central
2015
|
Online Access: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4407389/ |
Similar Items
-
Fcγ receptor targeting in RA
by: Takai, Toshiyuki, et al.
Published: (2012) -
Dichotomy in FcγRIIB deficiency and autoimmune-prone SLAM haplotype clarifies the roles of the Fc receptor in development of autoantibodies and glomerulonephritis
by: Kanari, Yasuyoshi, et al.
Published: (2014) -
Association between monocyte Fcγ subclass expression and acute coronary syndrome
by: Calverley, David C, et al.
Published: (2004) -
Monocyte-Platelet Interaction Induces a Pro-Inflammatory Phenotype in Circulating Monocytes
by: Passacquale, Gabriella, et al.
Published: (2011) -
Selective blockade of the inhibitory Fcγ receptor (FcγRIIB) in human dendritic cells and monocytes induces a type I interferon response program
by: Dhodapkar, Kavita M., et al.
Published: (2007)