Control of targeting ligand display by pH-responsive polymers on gold nanoparticles mediates selective entry into cancer cells

Selective targeting of cells for intracellular delivery of therapeutics represents a major challenge for pharmaceutical intervention in disease. Here we show pH triggered receptor-mediated endocytosis of nanoparticles via surface ligand exposure. Gold nanoparticles were decorated with two polymers:...

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Main Authors: Brazzale, C., Mastrotto, Francesca, Moody, P., Watson, P.D., Balasso, A., Malfanti, A., Mantovani, Giuseppe, Caliceti, Paolo, Alexander, Cameron, Jones, A.T., Salmaso, Stefano
Format: Article
Published: Royal Society of Chemistry 2017
Online Access:https://eprints.nottingham.ac.uk/44261/
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author Brazzale, C.
Mastrotto, Francesca
Moody, P.
Watson, P.D.
Balasso, A.
Malfanti, A.
Mantovani, Giuseppe
Caliceti, Paolo
Alexander, Cameron
Jones, A.T.
Salmaso, Stefano
author_facet Brazzale, C.
Mastrotto, Francesca
Moody, P.
Watson, P.D.
Balasso, A.
Malfanti, A.
Mantovani, Giuseppe
Caliceti, Paolo
Alexander, Cameron
Jones, A.T.
Salmaso, Stefano
author_sort Brazzale, C.
building Nottingham Research Data Repository
collection Online Access
description Selective targeting of cells for intracellular delivery of therapeutics represents a major challenge for pharmaceutical intervention in disease. Here we show pH triggered receptor-mediated endocytosis of nanoparticles via surface ligand exposure. Gold nanoparticles were decorated with two polymers: a 2 kDa PEG with a terminal folate targeting ligand, and a di-block copolymer including a pH-responsive and a hydrophilic block. At the normal serum pH of 7.4, the pH-responsive block (apparent pKa of 7.1) displayed a hydrophilic extended conformation, shielding the PEG-folate ligands, which inhibited cellular uptake of the nanoparticles. Under pH conditions resembling those of the extracellular matrix around solid tumours (pH 6.5), protonation of the pH-responsive polymer triggered a coil-to-globule polymer chain contraction, exposing folate residues on the PEG chains. In line with this, endocytosis of folate-decorated polymer-coated gold nanoparticles in cancer cells overexpressing folate receptor was significantly increased at pH 6.5, compared with pH 7.4. Thus, the tumour acidic environment and high folate receptor expression was effectively exploited to activate cell binding and endocytosis of these nanoparticles. These data provide proof-of-concept for strategies enabling extracellular pH stimuli to selectively enhance cellular uptake of drug delivery vectors and their associated therapeutic cargo.
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spelling nottingham-442612020-05-04T18:55:26Z https://eprints.nottingham.ac.uk/44261/ Control of targeting ligand display by pH-responsive polymers on gold nanoparticles mediates selective entry into cancer cells Brazzale, C. Mastrotto, Francesca Moody, P. Watson, P.D. Balasso, A. Malfanti, A. Mantovani, Giuseppe Caliceti, Paolo Alexander, Cameron Jones, A.T. Salmaso, Stefano Selective targeting of cells for intracellular delivery of therapeutics represents a major challenge for pharmaceutical intervention in disease. Here we show pH triggered receptor-mediated endocytosis of nanoparticles via surface ligand exposure. Gold nanoparticles were decorated with two polymers: a 2 kDa PEG with a terminal folate targeting ligand, and a di-block copolymer including a pH-responsive and a hydrophilic block. At the normal serum pH of 7.4, the pH-responsive block (apparent pKa of 7.1) displayed a hydrophilic extended conformation, shielding the PEG-folate ligands, which inhibited cellular uptake of the nanoparticles. Under pH conditions resembling those of the extracellular matrix around solid tumours (pH 6.5), protonation of the pH-responsive polymer triggered a coil-to-globule polymer chain contraction, exposing folate residues on the PEG chains. In line with this, endocytosis of folate-decorated polymer-coated gold nanoparticles in cancer cells overexpressing folate receptor was significantly increased at pH 6.5, compared with pH 7.4. Thus, the tumour acidic environment and high folate receptor expression was effectively exploited to activate cell binding and endocytosis of these nanoparticles. These data provide proof-of-concept for strategies enabling extracellular pH stimuli to selectively enhance cellular uptake of drug delivery vectors and their associated therapeutic cargo. Royal Society of Chemistry 2017-07-14 Article PeerReviewed Brazzale, C., Mastrotto, Francesca, Moody, P., Watson, P.D., Balasso, A., Malfanti, A., Mantovani, Giuseppe, Caliceti, Paolo, Alexander, Cameron, Jones, A.T. and Salmaso, Stefano (2017) Control of targeting ligand display by pH-responsive polymers on gold nanoparticles mediates selective entry into cancer cells. Nanoscale, 9 (31). pp. 11137-11147. ISSN 2040-3372 http://pubs.rsc.org/en/Content/ArticleLanding/2017/NR/C7NR02595E#!divAbstract doi:10.1039/C7NR02595E doi:10.1039/C7NR02595E
spellingShingle Brazzale, C.
Mastrotto, Francesca
Moody, P.
Watson, P.D.
Balasso, A.
Malfanti, A.
Mantovani, Giuseppe
Caliceti, Paolo
Alexander, Cameron
Jones, A.T.
Salmaso, Stefano
Control of targeting ligand display by pH-responsive polymers on gold nanoparticles mediates selective entry into cancer cells
title Control of targeting ligand display by pH-responsive polymers on gold nanoparticles mediates selective entry into cancer cells
title_full Control of targeting ligand display by pH-responsive polymers on gold nanoparticles mediates selective entry into cancer cells
title_fullStr Control of targeting ligand display by pH-responsive polymers on gold nanoparticles mediates selective entry into cancer cells
title_full_unstemmed Control of targeting ligand display by pH-responsive polymers on gold nanoparticles mediates selective entry into cancer cells
title_short Control of targeting ligand display by pH-responsive polymers on gold nanoparticles mediates selective entry into cancer cells
title_sort control of targeting ligand display by ph-responsive polymers on gold nanoparticles mediates selective entry into cancer cells
url https://eprints.nottingham.ac.uk/44261/
https://eprints.nottingham.ac.uk/44261/
https://eprints.nottingham.ac.uk/44261/