Hoxa9 collaborates with E2A-PBX1 in mouse B cell leukemia in association with Flt3 activation and decrease of B cell gene expression

Background: The fusion protein E2A-PBX1 induces pediatric B cell leukemia in human. Previously, we reported oncogenic interactions between homeobox (Hox) genes and E2A-PBX1 in murine T cell leukemia. A proviral insertional mutagenesis screen with our E2A-PBX1 B cell leukemia mouse model identified H...

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Main Authors: Hassawi, Mona, Shestakova, Elena A., Fournier, Marilaine, Lebert-Ghali, Charles-Étienne, Vaisson, Gratianne, Frison, Héloïse, Sinnett, Daniel, Vidal, Ramon, Thompson, Alexander, Bijl, Janet J.
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Published: Wiley 2014
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Online Access:https://eprints.nottingham.ac.uk/42822/
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author Hassawi, Mona
Shestakova, Elena A.
Fournier, Marilaine
Lebert-Ghali, Charles-Étienne
Vaisson, Gratianne
Frison, Héloïse
Sinnett, Daniel
Vidal, Ramon
Thompson, Alexander
Bijl, Janet J.
author_facet Hassawi, Mona
Shestakova, Elena A.
Fournier, Marilaine
Lebert-Ghali, Charles-Étienne
Vaisson, Gratianne
Frison, Héloïse
Sinnett, Daniel
Vidal, Ramon
Thompson, Alexander
Bijl, Janet J.
author_sort Hassawi, Mona
building Nottingham Research Data Repository
collection Online Access
description Background: The fusion protein E2A-PBX1 induces pediatric B cell leukemia in human. Previously, we reported oncogenic interactions between homeobox (Hox) genes and E2A-PBX1 in murine T cell leukemia. A proviral insertional mutagenesis screen with our E2A-PBX1 B cell leukemia mouse model identified Hoxa genes as potential collaborators to E2A-PBX1. Here we studied whether Hoxa9 could enhance E2A-PBX1 leukemogenesis. Results: We show that Hoxa9 confers a proliferative advantage to E2A-PBX1 B cells. Transplantation experiments with E2A-PBX1 transgenic B cells overexpressing Hoxa9 isolated from bone marrow chimeras showed that Hoxa9 accelerates the generation of E2A-PBX1 B cell leukemia, but Hoxa9 is unable to transform B cells alone. Quantitative-reverse transcriptase polymerase chain reaction analysis demonstrated a strong repression of B cell specific genes in these E2A-PBX1/Hoxa9 leukemias in addition to Flt3 activation, indicating inhibition of B cell differentiation in combination with enhanced proliferation. Overexpression of Hoxa9 in established E2A-PBX1 mouse leukemic B cells resulted in a growth advantage in vitro, which was also characterized by an enhanced expression of Flt3. Conclusions: we show for the first time that Hoxa9 collaborates with E2A-PBX1 in the oncogenic transformation of B cells in a mouse model that involves Flt3 signaling, which is potentially relevant to human disease.
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spelling nottingham-428222020-05-04T20:15:44Z https://eprints.nottingham.ac.uk/42822/ Hoxa9 collaborates with E2A-PBX1 in mouse B cell leukemia in association with Flt3 activation and decrease of B cell gene expression Hassawi, Mona Shestakova, Elena A. Fournier, Marilaine Lebert-Ghali, Charles-Étienne Vaisson, Gratianne Frison, Héloïse Sinnett, Daniel Vidal, Ramon Thompson, Alexander Bijl, Janet J. Background: The fusion protein E2A-PBX1 induces pediatric B cell leukemia in human. Previously, we reported oncogenic interactions between homeobox (Hox) genes and E2A-PBX1 in murine T cell leukemia. A proviral insertional mutagenesis screen with our E2A-PBX1 B cell leukemia mouse model identified Hoxa genes as potential collaborators to E2A-PBX1. Here we studied whether Hoxa9 could enhance E2A-PBX1 leukemogenesis. Results: We show that Hoxa9 confers a proliferative advantage to E2A-PBX1 B cells. Transplantation experiments with E2A-PBX1 transgenic B cells overexpressing Hoxa9 isolated from bone marrow chimeras showed that Hoxa9 accelerates the generation of E2A-PBX1 B cell leukemia, but Hoxa9 is unable to transform B cells alone. Quantitative-reverse transcriptase polymerase chain reaction analysis demonstrated a strong repression of B cell specific genes in these E2A-PBX1/Hoxa9 leukemias in addition to Flt3 activation, indicating inhibition of B cell differentiation in combination with enhanced proliferation. Overexpression of Hoxa9 in established E2A-PBX1 mouse leukemic B cells resulted in a growth advantage in vitro, which was also characterized by an enhanced expression of Flt3. Conclusions: we show for the first time that Hoxa9 collaborates with E2A-PBX1 in the oncogenic transformation of B cells in a mouse model that involves Flt3 signaling, which is potentially relevant to human disease. Wiley 2014-01 Article PeerReviewed Hassawi, Mona, Shestakova, Elena A., Fournier, Marilaine, Lebert-Ghali, Charles-Étienne, Vaisson, Gratianne, Frison, Héloïse, Sinnett, Daniel, Vidal, Ramon, Thompson, Alexander and Bijl, Janet J. (2014) Hoxa9 collaborates with E2A-PBX1 in mouse B cell leukemia in association with Flt3 activation and decrease of B cell gene expression. Developmental Dynamics, 243 (1). pp. 145-158. ISSN 1097-0177 Hox genes; oncogenes; transgenic mouse model; transcription factors http://onlinelibrary.wiley.com/doi/10.1002/dvdy.24056/abstract doi:10.1002/dvdy.24056 doi:10.1002/dvdy.24056
spellingShingle Hox genes; oncogenes; transgenic mouse model; transcription factors
Hassawi, Mona
Shestakova, Elena A.
Fournier, Marilaine
Lebert-Ghali, Charles-Étienne
Vaisson, Gratianne
Frison, Héloïse
Sinnett, Daniel
Vidal, Ramon
Thompson, Alexander
Bijl, Janet J.
Hoxa9 collaborates with E2A-PBX1 in mouse B cell leukemia in association with Flt3 activation and decrease of B cell gene expression
title Hoxa9 collaborates with E2A-PBX1 in mouse B cell leukemia in association with Flt3 activation and decrease of B cell gene expression
title_full Hoxa9 collaborates with E2A-PBX1 in mouse B cell leukemia in association with Flt3 activation and decrease of B cell gene expression
title_fullStr Hoxa9 collaborates with E2A-PBX1 in mouse B cell leukemia in association with Flt3 activation and decrease of B cell gene expression
title_full_unstemmed Hoxa9 collaborates with E2A-PBX1 in mouse B cell leukemia in association with Flt3 activation and decrease of B cell gene expression
title_short Hoxa9 collaborates with E2A-PBX1 in mouse B cell leukemia in association with Flt3 activation and decrease of B cell gene expression
title_sort hoxa9 collaborates with e2a-pbx1 in mouse b cell leukemia in association with flt3 activation and decrease of b cell gene expression
topic Hox genes; oncogenes; transgenic mouse model; transcription factors
url https://eprints.nottingham.ac.uk/42822/
https://eprints.nottingham.ac.uk/42822/
https://eprints.nottingham.ac.uk/42822/