Anti-GAD65 Containing Cerebrospinal Fluid Does not Alter GABAergic Transmission
Glutamic acid decarboxylase of 65 kDa (GAD65) antibodies have been reported in a variety of neurological disorders such as stiff-person syndrome (SPS), sporadic ataxia and some cases of epilepsy. Since the target is believed to be the cytoplasmic enzyme GAD65, the key enzyme of γ-aminobutyric acid (...
Main Authors: | Hackert, Jana K., Müller, Lorenz, Rohde, Marco, Bien, Christian G., Köhling, Rüdiger, Kirschstein, Timo |
---|---|
Format: | Online |
Language: | English |
Published: |
Frontiers Media S.A.
2016
|
Online Access: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4870265/ |
Similar Items
-
NMDA Receptor-Dependent Metaplasticity by High-Frequency Magnetic Stimulation
by: Tokay, Tursonjan, et al.
Published: (2014) -
Decline in Titers of Anti-Idiotypic Antibodies Specific to Autoantibodies to GAD65 (GAD65Ab) Precedes Development of GAD65Ab and Type 1 Diabetes
by: Larsson, Helena Elding, et al.
Published: (2013) -
An Analysis of the Cross-Reactivity of Autoantibodies to GAD65 and GAD67 in Diabetes
by: Jayakrishnan, Bindu, et al.
Published: (2011) -
Elevated Serum GAD65 and GAD65-GADA Immune Complexes in Stiff Person Syndrome
by: Gu Urban, Gucci Jijuan, et al.
Published: (2015) -
Status Epilepticus Enhances Depotentiation after Fully Established LTP in an NMDAR-Dependent but GluN2B-Independent Manner
by: Guli, Xiati, et al.
Published: (2016)