Hepatitis B virus genome replication triggers toll-like receptor 3-dependent interferon responses in the absence of hepatitis B surface antigen

Hepatitis B virus genome replication triggers toll-like receptor 3-dependent interferon responses in the absence of hepatitis B surface antigen

The hepatitis B virus (HBV) has been described as stealth virus subverting immune responses initially upon infection. Impaired toll-like receptor signaling by the HBV surface antigen (HBsAg) attenuates immune responses to facilitate chronic infection. This implies that HBV replication may trigger ho...

Full description

Bibliographic Details
Main Authors: Real, Catherine Isabell, Lu, Mengji, Liu, Jia, Huang, Xuan, Trippler, Martin, Hossbach, Markus, Deckert, Jochen, Jahn-Hofmann, Kerstin, Ickenstein, Ludger Markus, John, Matthias Johannes, Gibbert, Kathrin, Dittmer, Ulf, Vornlocher, Hans-Peter, Schirmbeck, Reinhold, Gerken, Guido, Schlaak, Joerg Friedrich, Broering, Ruth
Format: Online
Language:English
Published: Nature Publishing Group 2016
Online Access:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4848479/

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4848479/

Similar Items