Cytokine signals through PI-3 kinase pathway modulate Th17 cytokine production by CCR6+ human memory T cells
PI-3K–mediated repression of FOXO1 and KLF2 promotes proinflammatory cytokine expression by lineage-committed human CCR6+ Th17/Th22 memory cells.
Main Authors: | Wan, Qi, Kozhaya, Lina, ElHed, Aimee, Ramesh, Radha, Carlson, Thaddeus J., Djuretic, Ivana M., Sundrud, Mark S., Unutmaz, Derya |
---|---|
Format: | Online |
Language: | English |
Published: |
The Rockefeller University Press
2011
|
Online Access: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3171088/ |
Similar Items
-
Pro-inflammatory human Th17 cells selectively express P-glycoprotein and are refractory to glucocorticoids
by: Ramesh, Radha, et al.
Published: (2014) -
The Metalloprotease ADAM12 Regulates the Effector Function of Human Th17 Cells
by: Zhou, Angela X., et al.
Published: (2013) -
Th17 Cytokines and Barrier Functions
by: Wang, Guansong, et al.
Published: (2016) -
Th17 cytokines regulate profibrotic cytokines release by human eosinophils
by: Halwani, Rabih, et al.
Published: (2012) -
The Role of TH17-Associated Cytokines in Health and Disease
by: O'Connor, William, et al.
Published: (2014)