Tumour-draining axillary lymph nodes in patients with large and locally advanced breast cancers undergoing neoadjuvant chemotherapy (NAC): the crucial contribution of immune cells (effector, regulatory) and cytokines (TH1, TH2) to immune-mediated tumour cell death induced by NAC

Background and Aim: The tumour microenvironment consists of malignant cells, stroma and immune cells. In women with large and locally advanced breast cancers (LLABCs) undergoing neoadjuvant chemotherapy (NAC), tumour-infiltrating lymphocytes (TILs), various subsets (effector, regulatory) and cytok...

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Main Authors: Kaewkangsadan, Viriya, Verma, Chandan, Eremin, Jennifer M., Cowley, Gerard P.
Format: Article
Published: BioMed Central 2016
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Online Access:https://eprints.nottingham.ac.uk/33127/
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author Kaewkangsadan, Viriya
Verma, Chandan
Eremin, Jennifer M.
Cowley, Gerard P.
author_facet Kaewkangsadan, Viriya
Verma, Chandan
Eremin, Jennifer M.
Cowley, Gerard P.
author_sort Kaewkangsadan, Viriya
building Nottingham Research Data Repository
collection Online Access
description Background and Aim: The tumour microenvironment consists of malignant cells, stroma and immune cells. In women with large and locally advanced breast cancers (LLABCs) undergoing neoadjuvant chemotherapy (NAC), tumour-infiltrating lymphocytes (TILs), various subsets (effector, regulatory) and cytokines in the primary tumour play a key role in the induction of tumour cell death and a pathological complete response (pCR) with NAC. The contribution of the cellular and humoral responses to a pCR in nodal metastases, however, is poorly studied and was investigated. Patients and Methods: Axillary lymph nodes (ALNs) (24 with and 9 without metastases) from women with LLABCs undergoing NAC were immunohistochemically assessed for TILs, T cell subsets, NK cells and cytokine expression using labelled antibodies, employing established semi-quantitative methods. Results: In ALN metastases high levels of TILs, CD4+ and CD8+ T and CD56+ NK cells were significantly associated with pCRs. High levels of TILs and CD56+ NK cells in ALN metastases were independent predictors (univariate analysis) of a pCR with NAC. Significantly higher levels of Tregs (FOXP3+, CTLA-4+) and CD56+ NK cells were documented in ALN metastases than in the corresponding primary breast tumours. CD8+ T and CD56+ NK cells showed a positive correlation between metastatic and primary tumours. A high % CD8+ and low % FOXP3+ T cells and high CD8+ :FOXP3+ ratio in metastatic ALNs (tumour-free paracortex) were associated with pCRs. Metastatic ALNs expressed high IL-10, low IL-2 and IFN-ϒ. Conclusion: Our study has provided new data characterising the possible contribution of T effector/regulatory and NK cells and cytokines to tumour cell death with NAC.
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spelling nottingham-331272020-05-04T20:02:43Z https://eprints.nottingham.ac.uk/33127/ Tumour-draining axillary lymph nodes in patients with large and locally advanced breast cancers undergoing neoadjuvant chemotherapy (NAC): the crucial contribution of immune cells (effector, regulatory) and cytokines (TH1, TH2) to immune-mediated tumour cell death induced by NAC Kaewkangsadan, Viriya Verma, Chandan Eremin, Jennifer M. Cowley, Gerard P. Background and Aim: The tumour microenvironment consists of malignant cells, stroma and immune cells. In women with large and locally advanced breast cancers (LLABCs) undergoing neoadjuvant chemotherapy (NAC), tumour-infiltrating lymphocytes (TILs), various subsets (effector, regulatory) and cytokines in the primary tumour play a key role in the induction of tumour cell death and a pathological complete response (pCR) with NAC. The contribution of the cellular and humoral responses to a pCR in nodal metastases, however, is poorly studied and was investigated. Patients and Methods: Axillary lymph nodes (ALNs) (24 with and 9 without metastases) from women with LLABCs undergoing NAC were immunohistochemically assessed for TILs, T cell subsets, NK cells and cytokine expression using labelled antibodies, employing established semi-quantitative methods. Results: In ALN metastases high levels of TILs, CD4+ and CD8+ T and CD56+ NK cells were significantly associated with pCRs. High levels of TILs and CD56+ NK cells in ALN metastases were independent predictors (univariate analysis) of a pCR with NAC. Significantly higher levels of Tregs (FOXP3+, CTLA-4+) and CD56+ NK cells were documented in ALN metastases than in the corresponding primary breast tumours. CD8+ T and CD56+ NK cells showed a positive correlation between metastatic and primary tumours. A high % CD8+ and low % FOXP3+ T cells and high CD8+ :FOXP3+ ratio in metastatic ALNs (tumour-free paracortex) were associated with pCRs. Metastatic ALNs expressed high IL-10, low IL-2 and IFN-ϒ. Conclusion: Our study has provided new data characterising the possible contribution of T effector/regulatory and NK cells and cytokines to tumour cell death with NAC. BioMed Central 2016-05 Article NonPeerReviewed Kaewkangsadan, Viriya, Verma, Chandan, Eremin, Jennifer M. and Cowley, Gerard P. (2016) Tumour-draining axillary lymph nodes in patients with large and locally advanced breast cancers undergoing neoadjuvant chemotherapy (NAC): the crucial contribution of immune cells (effector, regulatory) and cytokines (TH1, TH2) to immune-mediated tumour cell death induced by NAC. Breast Cancer Research . ISSN 1465-542X (Submitted) Axillary lymph node; breast cancer neoadjuvant chemotherapy tumour microenvironment tumour-infiltrating lymphocyte subsets cytokines.
spellingShingle Axillary lymph node; breast cancer
neoadjuvant chemotherapy
tumour microenvironment
tumour-infiltrating lymphocyte subsets
cytokines.
Kaewkangsadan, Viriya
Verma, Chandan
Eremin, Jennifer M.
Cowley, Gerard P.
Tumour-draining axillary lymph nodes in patients with large and locally advanced breast cancers undergoing neoadjuvant chemotherapy (NAC): the crucial contribution of immune cells (effector, regulatory) and cytokines (TH1, TH2) to immune-mediated tumour cell death induced by NAC
title Tumour-draining axillary lymph nodes in patients with large and locally advanced breast cancers undergoing neoadjuvant chemotherapy (NAC): the crucial contribution of immune cells (effector, regulatory) and cytokines (TH1, TH2) to immune-mediated tumour cell death induced by NAC
title_full Tumour-draining axillary lymph nodes in patients with large and locally advanced breast cancers undergoing neoadjuvant chemotherapy (NAC): the crucial contribution of immune cells (effector, regulatory) and cytokines (TH1, TH2) to immune-mediated tumour cell death induced by NAC
title_fullStr Tumour-draining axillary lymph nodes in patients with large and locally advanced breast cancers undergoing neoadjuvant chemotherapy (NAC): the crucial contribution of immune cells (effector, regulatory) and cytokines (TH1, TH2) to immune-mediated tumour cell death induced by NAC
title_full_unstemmed Tumour-draining axillary lymph nodes in patients with large and locally advanced breast cancers undergoing neoadjuvant chemotherapy (NAC): the crucial contribution of immune cells (effector, regulatory) and cytokines (TH1, TH2) to immune-mediated tumour cell death induced by NAC
title_short Tumour-draining axillary lymph nodes in patients with large and locally advanced breast cancers undergoing neoadjuvant chemotherapy (NAC): the crucial contribution of immune cells (effector, regulatory) and cytokines (TH1, TH2) to immune-mediated tumour cell death induced by NAC
title_sort tumour-draining axillary lymph nodes in patients with large and locally advanced breast cancers undergoing neoadjuvant chemotherapy (nac): the crucial contribution of immune cells (effector, regulatory) and cytokines (th1, th2) to immune-mediated tumour cell death induced by nac
topic Axillary lymph node; breast cancer
neoadjuvant chemotherapy
tumour microenvironment
tumour-infiltrating lymphocyte subsets
cytokines.
url https://eprints.nottingham.ac.uk/33127/