The desferrithiocin (DFT) class of iron chelators: Potential as antineoplastic agents

Iron (Fe) is essential for the proliferation of cancer cells. A subgroup of the orthosubstituted phenolate class of Fe chelators, desferrithiocin [2-(3′-hydroxypyrid-2′-yl)-4-methyl- Δ2-thiazoline-4(S)-carboxylic acid; DFT] and its analogues, have potential application in short-term chemotherapy for...

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Main Authors: Kicic, Anthony, Chua, A.C.G., Baker, E.
Format: Journal Article
Language:English
Published: OXFORD UNIV PRESS 2001
Subjects:
Online Access:http://hdl.handle.net/20.500.11937/76831
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author Kicic, Anthony
Chua, A.C.G.
Baker, E.
author_facet Kicic, Anthony
Chua, A.C.G.
Baker, E.
author_sort Kicic, Anthony
building Curtin Institutional Repository
collection Online Access
description Iron (Fe) is essential for the proliferation of cancer cells. A subgroup of the orthosubstituted phenolate class of Fe chelators, desferrithiocin [2-(3′-hydroxypyrid-2′-yl)-4-methyl- Δ2-thiazoline-4(S)-carboxylic acid; DFT] and its analogues, have potential application in short-term chemotherapy for cancer by Fe deprivation. Their effects on cell proliferation, cell cycle progression, Fe uptake and toxicity were therefore examined in adult and fetal rat and human hepatocellular carcinoma (HCC) cell lines, as well as in normal cells. DFT was more active than desferrioxamine, in clinical trials as an antineoplastic agent, consistently inhibiting cell proliferation in all cell lines (IC50 = 40 μM). 2-(2′-hydroxyphenyl-2′-yl)- Δ2-thiazoline-4(S)-carboxylic acid was the most active analogue (IC50 = 55-90 μM). Inhibition was affected by chelator concentration and ability to prevent Fe uptake. The fetal-cell-derived HCC was more susceptible than adult HCC. Structure-activity studies revealed that thiazol methyl deletion greatly diminished antiproliferative activity of the chelators but stereochemical orientation of COOH around C4 had no effect. Removal of the N from the pyridine ring restored antiproliferative activity. Chelators inhibited DNA synthesis in the S phase. The chelators at their IC50 concentration had little or no effect on Fe uptake in normal cells. This apparent selectivity of these chelators for cancer cells, coupled with their high activity, suggests that further investigation is warranted.
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spelling curtin-20.500.11937-768312019-11-12T08:29:01Z The desferrithiocin (DFT) class of iron chelators: Potential as antineoplastic agents Kicic, Anthony Chua, A.C.G. Baker, E. Science & Technology Life Sciences & Biomedicine Biochemistry & Molecular Biology Oncology Chemistry, Medicinal Pharmacology & Pharmacy cancer desferrioxamine desferrithiocin Fe chelators fetal and adult derived hepatoma cells PYRIDOXAL ISONICOTINOYL HYDRAZONE EFFECTIVE ANTIPROLIFERATIVE AGENTS HUMAN NEUROBLASTOMA CELL MALIGNANT MELANOMA CELL TRANSFERRIN RECEPTOR MONOCLONAL-ANTIBODY IRON(III) CHELATOR DNA-SYNTHESIS HL-60 CELLS DESFERRIOXAMINE Iron (Fe) is essential for the proliferation of cancer cells. A subgroup of the orthosubstituted phenolate class of Fe chelators, desferrithiocin [2-(3′-hydroxypyrid-2′-yl)-4-methyl- Δ2-thiazoline-4(S)-carboxylic acid; DFT] and its analogues, have potential application in short-term chemotherapy for cancer by Fe deprivation. Their effects on cell proliferation, cell cycle progression, Fe uptake and toxicity were therefore examined in adult and fetal rat and human hepatocellular carcinoma (HCC) cell lines, as well as in normal cells. DFT was more active than desferrioxamine, in clinical trials as an antineoplastic agent, consistently inhibiting cell proliferation in all cell lines (IC50 = 40 μM). 2-(2′-hydroxyphenyl-2′-yl)- Δ2-thiazoline-4(S)-carboxylic acid was the most active analogue (IC50 = 55-90 μM). Inhibition was affected by chelator concentration and ability to prevent Fe uptake. The fetal-cell-derived HCC was more susceptible than adult HCC. Structure-activity studies revealed that thiazol methyl deletion greatly diminished antiproliferative activity of the chelators but stereochemical orientation of COOH around C4 had no effect. Removal of the N from the pyridine ring restored antiproliferative activity. Chelators inhibited DNA synthesis in the S phase. The chelators at their IC50 concentration had little or no effect on Fe uptake in normal cells. This apparent selectivity of these chelators for cancer cells, coupled with their high activity, suggests that further investigation is warranted. 2001 Journal Article http://hdl.handle.net/20.500.11937/76831 English OXFORD UNIV PRESS restricted
spellingShingle Science & Technology
Life Sciences & Biomedicine
Biochemistry & Molecular Biology
Oncology
Chemistry, Medicinal
Pharmacology & Pharmacy
cancer
desferrioxamine
desferrithiocin
Fe chelators
fetal and adult derived hepatoma cells
PYRIDOXAL ISONICOTINOYL HYDRAZONE
EFFECTIVE ANTIPROLIFERATIVE AGENTS
HUMAN NEUROBLASTOMA CELL
MALIGNANT MELANOMA CELL
TRANSFERRIN RECEPTOR
MONOCLONAL-ANTIBODY
IRON(III) CHELATOR
DNA-SYNTHESIS
HL-60 CELLS
DESFERRIOXAMINE
Kicic, Anthony
Chua, A.C.G.
Baker, E.
The desferrithiocin (DFT) class of iron chelators: Potential as antineoplastic agents
title The desferrithiocin (DFT) class of iron chelators: Potential as antineoplastic agents
title_full The desferrithiocin (DFT) class of iron chelators: Potential as antineoplastic agents
title_fullStr The desferrithiocin (DFT) class of iron chelators: Potential as antineoplastic agents
title_full_unstemmed The desferrithiocin (DFT) class of iron chelators: Potential as antineoplastic agents
title_short The desferrithiocin (DFT) class of iron chelators: Potential as antineoplastic agents
title_sort desferrithiocin (dft) class of iron chelators: potential as antineoplastic agents
topic Science & Technology
Life Sciences & Biomedicine
Biochemistry & Molecular Biology
Oncology
Chemistry, Medicinal
Pharmacology & Pharmacy
cancer
desferrioxamine
desferrithiocin
Fe chelators
fetal and adult derived hepatoma cells
PYRIDOXAL ISONICOTINOYL HYDRAZONE
EFFECTIVE ANTIPROLIFERATIVE AGENTS
HUMAN NEUROBLASTOMA CELL
MALIGNANT MELANOMA CELL
TRANSFERRIN RECEPTOR
MONOCLONAL-ANTIBODY
IRON(III) CHELATOR
DNA-SYNTHESIS
HL-60 CELLS
DESFERRIOXAMINE
url http://hdl.handle.net/20.500.11937/76831