Potential role of signal transducer and activator of transcription (STAT)3 signaling pathway in inflammation, survival, proliferation and invasion of hepatocellular carcinoma

Hepatocellular carcinoma (HCC) is one of the most lethal malignancies, and is also the fourth most common cancer worldwide with around 700,000 new cases each year. Currently, first line chemotherapeutic drugs used for HCC include fluorouracil, cisplatin, doxorubicin, paclitaxel and mitomycin, but mo...

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Main Authors: Subramaniam, A., Shanmugam, M., Perumal, E., Li, F., Nachiyappan, A., Dai, X., Swamy, S., Anh, K., Kumar, Alan Prem, Tan, B., Hui, K., Sethi, G.
Format: Journal Article
Published: Elsevier 2012
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Online Access:http://hdl.handle.net/20.500.11937/3994
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author Subramaniam, A.
Shanmugam, M.
Perumal, E.
Li, F.
Nachiyappan, A.
Dai, X.
Swamy, S.
Anh, K.
Kumar, Alan Prem
Tan, B.
Hui, K.
Sethi, G.
author_facet Subramaniam, A.
Shanmugam, M.
Perumal, E.
Li, F.
Nachiyappan, A.
Dai, X.
Swamy, S.
Anh, K.
Kumar, Alan Prem
Tan, B.
Hui, K.
Sethi, G.
author_sort Subramaniam, A.
building Curtin Institutional Repository
collection Online Access
description Hepatocellular carcinoma (HCC) is one of the most lethal malignancies, and is also the fourth most common cancer worldwide with around 700,000 new cases each year. Currently, first line chemotherapeutic drugs used for HCC include fluorouracil, cisplatin, doxorubicin, paclitaxel and mitomycin, but most of these are non-selective cytotoxic molecules with significant side effects. Sorafenib is the only approved targeted therapy by the U.S. Food and Drug Administration for HCC treatment, but patients suffer from various kinds of adverse effects, including hypertension. The signal-transducer-and-activator-of-transcription 3 (STAT3) protein, one of the members of STATs transcription factor family, has been implicated in signal transduction by different cytokines, growth factors and oncogenes. In normal cells, STAT3 activation is tightly controlled to prevent dysregulated gene transcription, whereas constitutively activated STAT3 plays an important role in tumorigenesisthrough the upregulation of genes involved in anti-apoptosis, proliferation and angiogenesis. Thus, pharmacologically safe and effective agents that can block STAT3 activation have the potential both for the prevention and treatment of HCC. In the present review, we discuss the possible role of STAT3 signaling cascade and its interacting partners in the initiation of HCC and also analyze the role of various STAT3 regulated genes in HCC progression, inflammation, survival, invasion and angiogenesis
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spelling curtin-20.500.11937-39942018-03-29T09:05:22Z Potential role of signal transducer and activator of transcription (STAT)3 signaling pathway in inflammation, survival, proliferation and invasion of hepatocellular carcinoma Subramaniam, A. Shanmugam, M. Perumal, E. Li, F. Nachiyappan, A. Dai, X. Swamy, S. Anh, K. Kumar, Alan Prem Tan, B. Hui, K. Sethi, G. proliferation apoptosis invasion STAT3 hepatocellular carcinoma JAKs Hepatocellular carcinoma (HCC) is one of the most lethal malignancies, and is also the fourth most common cancer worldwide with around 700,000 new cases each year. Currently, first line chemotherapeutic drugs used for HCC include fluorouracil, cisplatin, doxorubicin, paclitaxel and mitomycin, but most of these are non-selective cytotoxic molecules with significant side effects. Sorafenib is the only approved targeted therapy by the U.S. Food and Drug Administration for HCC treatment, but patients suffer from various kinds of adverse effects, including hypertension. The signal-transducer-and-activator-of-transcription 3 (STAT3) protein, one of the members of STATs transcription factor family, has been implicated in signal transduction by different cytokines, growth factors and oncogenes. In normal cells, STAT3 activation is tightly controlled to prevent dysregulated gene transcription, whereas constitutively activated STAT3 plays an important role in tumorigenesisthrough the upregulation of genes involved in anti-apoptosis, proliferation and angiogenesis. Thus, pharmacologically safe and effective agents that can block STAT3 activation have the potential both for the prevention and treatment of HCC. In the present review, we discuss the possible role of STAT3 signaling cascade and its interacting partners in the initiation of HCC and also analyze the role of various STAT3 regulated genes in HCC progression, inflammation, survival, invasion and angiogenesis 2012 Journal Article http://hdl.handle.net/20.500.11937/3994 10.1016/j.bbcan.2012.10.002 Elsevier restricted
spellingShingle proliferation
apoptosis
invasion
STAT3
hepatocellular carcinoma
JAKs
Subramaniam, A.
Shanmugam, M.
Perumal, E.
Li, F.
Nachiyappan, A.
Dai, X.
Swamy, S.
Anh, K.
Kumar, Alan Prem
Tan, B.
Hui, K.
Sethi, G.
Potential role of signal transducer and activator of transcription (STAT)3 signaling pathway in inflammation, survival, proliferation and invasion of hepatocellular carcinoma
title Potential role of signal transducer and activator of transcription (STAT)3 signaling pathway in inflammation, survival, proliferation and invasion of hepatocellular carcinoma
title_full Potential role of signal transducer and activator of transcription (STAT)3 signaling pathway in inflammation, survival, proliferation and invasion of hepatocellular carcinoma
title_fullStr Potential role of signal transducer and activator of transcription (STAT)3 signaling pathway in inflammation, survival, proliferation and invasion of hepatocellular carcinoma
title_full_unstemmed Potential role of signal transducer and activator of transcription (STAT)3 signaling pathway in inflammation, survival, proliferation and invasion of hepatocellular carcinoma
title_short Potential role of signal transducer and activator of transcription (STAT)3 signaling pathway in inflammation, survival, proliferation and invasion of hepatocellular carcinoma
title_sort potential role of signal transducer and activator of transcription (stat)3 signaling pathway in inflammation, survival, proliferation and invasion of hepatocellular carcinoma
topic proliferation
apoptosis
invasion
STAT3
hepatocellular carcinoma
JAKs
url http://hdl.handle.net/20.500.11937/3994