An anthraquinone derivative, emodin sensitizes hepatocellular carcinoma cells to TRAIL induced apoptosis through the induction of death receptors and downregulation of cell survival proteins

Recombinant tumor necrosis factor-related apoptosis-inducing ligand (TRAIL) is currently under clinical trials for cancer, however many tumor cells, including hepatocellular carcinoma (HCC) develop resistance to TRAIL-induced apoptosis. Hence, novel agents that can alleviate TRAIL-induced resistance...

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Main Authors: Subramaniam, Aruljothi, Loo, Ser, Rajendran, P., Manu, K., Permul, E., Li, F., Shanmugam, M., Siveen, K., Park, J., Ahn, K., Hui, K., Kumar, Alan Prem, Sethi, G.
Format: Journal Article
Published: Springer 2013
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Online Access:http://hdl.handle.net/20.500.11937/35723
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author Subramaniam, Aruljothi
Loo, Ser
Rajendran, P.
Manu, K.
Permul, E.
Li, F.
Shanmugam, M.
Siveen, K.
Park, J.
Ahn, K.
Hui, K.
Kumar, Alan Prem
Sethi, G.
author_facet Subramaniam, Aruljothi
Loo, Ser
Rajendran, P.
Manu, K.
Permul, E.
Li, F.
Shanmugam, M.
Siveen, K.
Park, J.
Ahn, K.
Hui, K.
Kumar, Alan Prem
Sethi, G.
author_sort Subramaniam, Aruljothi
building Curtin Institutional Repository
collection Online Access
description Recombinant tumor necrosis factor-related apoptosis-inducing ligand (TRAIL) is currently under clinical trials for cancer, however many tumor cells, including hepatocellular carcinoma (HCC) develop resistance to TRAIL-induced apoptosis. Hence, novel agents that can alleviate TRAIL-induced resistance are urgently needed. In the present report, we investigated the potential of emodin to enhance apoptosis induced by TRAIL in HCC cells. As observed by MTT cytotoxicity assay and the externalization of the membrane phospholipid phosphatidylserine, we found that emodin can significantly potentiate TRAIL-induced apoptosis in HCC cells. When investigated for the mechanism(s), we observed that emodin can downregulate the expression of various cell survival proteins, and induce the cell surface expression of both TRAIL receptors, death receptors (DR) 4 as well as 5. In addition, emodin increased the expression of C/EBP homologous protein (CHOP) in a time-dependent manner. Knockdown of CHOP by siRNA decreased the induction of emodin-induced DR5 expression and apoptosis. Emodin-induced induction of DR5 was mediated through the generation of reactive oxygen species (ROS), as N-acetylcysteine blocked the induction of DR5 and the induction of apoptosis. Also, the knockdown of X-linked inhibitor of apoptosis protein by siRNA significantly reduced the sensitization effect of emodin on TRAIL-induced apoptosis. Overall, our experimental results clearly indicate that emodin can indeed potentiate TRAIL-induced apoptosis through the downregulation of antiapoptotic proteins, increased expression of apoptotic proteins, and ROS mediated upregulation of DR in HCC cells.
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institution Curtin University Malaysia
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publishDate 2013
publisher Springer
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spelling curtin-20.500.11937-357232018-03-29T09:09:00Z An anthraquinone derivative, emodin sensitizes hepatocellular carcinoma cells to TRAIL induced apoptosis through the induction of death receptors and downregulation of cell survival proteins Subramaniam, Aruljothi Loo, Ser Rajendran, P. Manu, K. Permul, E. Li, F. Shanmugam, M. Siveen, K. Park, J. Ahn, K. Hui, K. Kumar, Alan Prem Sethi, G. TRAIL DR5 DR4 Apoptosis Emodin CHOP HCC Recombinant tumor necrosis factor-related apoptosis-inducing ligand (TRAIL) is currently under clinical trials for cancer, however many tumor cells, including hepatocellular carcinoma (HCC) develop resistance to TRAIL-induced apoptosis. Hence, novel agents that can alleviate TRAIL-induced resistance are urgently needed. In the present report, we investigated the potential of emodin to enhance apoptosis induced by TRAIL in HCC cells. As observed by MTT cytotoxicity assay and the externalization of the membrane phospholipid phosphatidylserine, we found that emodin can significantly potentiate TRAIL-induced apoptosis in HCC cells. When investigated for the mechanism(s), we observed that emodin can downregulate the expression of various cell survival proteins, and induce the cell surface expression of both TRAIL receptors, death receptors (DR) 4 as well as 5. In addition, emodin increased the expression of C/EBP homologous protein (CHOP) in a time-dependent manner. Knockdown of CHOP by siRNA decreased the induction of emodin-induced DR5 expression and apoptosis. Emodin-induced induction of DR5 was mediated through the generation of reactive oxygen species (ROS), as N-acetylcysteine blocked the induction of DR5 and the induction of apoptosis. Also, the knockdown of X-linked inhibitor of apoptosis protein by siRNA significantly reduced the sensitization effect of emodin on TRAIL-induced apoptosis. Overall, our experimental results clearly indicate that emodin can indeed potentiate TRAIL-induced apoptosis through the downregulation of antiapoptotic proteins, increased expression of apoptotic proteins, and ROS mediated upregulation of DR in HCC cells. 2013 Journal Article http://hdl.handle.net/20.500.11937/35723 10.1007/s10495-013-0851-5 Springer restricted
spellingShingle TRAIL
DR5
DR4
Apoptosis
Emodin
CHOP
HCC
Subramaniam, Aruljothi
Loo, Ser
Rajendran, P.
Manu, K.
Permul, E.
Li, F.
Shanmugam, M.
Siveen, K.
Park, J.
Ahn, K.
Hui, K.
Kumar, Alan Prem
Sethi, G.
An anthraquinone derivative, emodin sensitizes hepatocellular carcinoma cells to TRAIL induced apoptosis through the induction of death receptors and downregulation of cell survival proteins
title An anthraquinone derivative, emodin sensitizes hepatocellular carcinoma cells to TRAIL induced apoptosis through the induction of death receptors and downregulation of cell survival proteins
title_full An anthraquinone derivative, emodin sensitizes hepatocellular carcinoma cells to TRAIL induced apoptosis through the induction of death receptors and downregulation of cell survival proteins
title_fullStr An anthraquinone derivative, emodin sensitizes hepatocellular carcinoma cells to TRAIL induced apoptosis through the induction of death receptors and downregulation of cell survival proteins
title_full_unstemmed An anthraquinone derivative, emodin sensitizes hepatocellular carcinoma cells to TRAIL induced apoptosis through the induction of death receptors and downregulation of cell survival proteins
title_short An anthraquinone derivative, emodin sensitizes hepatocellular carcinoma cells to TRAIL induced apoptosis through the induction of death receptors and downregulation of cell survival proteins
title_sort anthraquinone derivative, emodin sensitizes hepatocellular carcinoma cells to trail induced apoptosis through the induction of death receptors and downregulation of cell survival proteins
topic TRAIL
DR5
DR4
Apoptosis
Emodin
CHOP
HCC
url http://hdl.handle.net/20.500.11937/35723