Gliclazide reduces MKC intestinal transport in healthy but not diabetic rats

The aim is to investigate the influence of the antidiabetic drug gliclazide on the ileal permeation of the semisynthetic bile acid, MKC, in tissues from healthy and diabetic rats. Sixteen Wistar rats (350 +/- 50 g) were randomly allocated into four groups (4 rats per group, 8 chambers per rat, i.e.,...

Full description

Bibliographic Details
Main Authors: Al-Salami, Hani, Butt, G., Tucker, I., Fawcett, P., Golocorbin-Kon, S., Mikov, I., Mikov, M.
Format: Journal Article
Published: Springer France 2009
Subjects:
Online Access:http://hdl.handle.net/20.500.11937/25466
_version_ 1848751717200429056
author Al-Salami, Hani
Butt, G.
Tucker, I.
Fawcett, P.
Golocorbin-Kon, S.
Mikov, I.
Mikov, M.
author_facet Al-Salami, Hani
Butt, G.
Tucker, I.
Fawcett, P.
Golocorbin-Kon, S.
Mikov, I.
Mikov, M.
author_sort Al-Salami, Hani
building Curtin Institutional Repository
collection Online Access
description The aim is to investigate the influence of the antidiabetic drug gliclazide on the ileal permeation of the semisynthetic bile acid, MKC, in tissues from healthy and diabetic rats. Sixteen Wistar rats (350 +/- 50 g) were randomly allocated into four groups (4 rats per group, 8 chambers per rat, i.e., n=32) two of which were made diabetic (given alloxan i.v. 30 mg/kg). Group 1 was used to measure the permeation of MKC (50 microg/ml) alone (control) while group 2 to measure MKC permeation in the presence of gliclazide (200 microg/ml). The diabetic groups 3 (gliclazide) and 4 (MKC+gliclazide) were treated in the same way. Rats were sacrificed and tissues were mounted into the Ussing chamber for the measurement of MKC mucosal to serosal (absorptive) and serosal to mucosal (secretory) fluxes. In healthy tissues, gliclazide reduced MKC absorptive flux (p < 0.01) and increased its secretory flux (p < 0.01). In diabetic tissues, gliclazide had no effect on either the absorptive or the secretory fluxes of MKC. The lack of effect of gliclazide on MKC permeation in diabetic tissues suggests the absence or suppressed drug transporters. Furthermore, gliclazide inhibition of MKC absorptive flux and induction of MKC secretory flux in healthy tissues may result from the selective inhibition of an efflux drug transporter.
first_indexed 2025-11-14T07:57:10Z
format Journal Article
id curtin-20.500.11937-25466
institution Curtin University Malaysia
institution_category Local University
last_indexed 2025-11-14T07:57:10Z
publishDate 2009
publisher Springer France
recordtype eprints
repository_type Digital Repository
spelling curtin-20.500.11937-254662017-01-30T12:48:38Z Gliclazide reduces MKC intestinal transport in healthy but not diabetic rats Al-Salami, Hani Butt, G. Tucker, I. Fawcett, P. Golocorbin-Kon, S. Mikov, I. Mikov, M. permeation Mrp3 transporters gliclazide diabetes bile acid The aim is to investigate the influence of the antidiabetic drug gliclazide on the ileal permeation of the semisynthetic bile acid, MKC, in tissues from healthy and diabetic rats. Sixteen Wistar rats (350 +/- 50 g) were randomly allocated into four groups (4 rats per group, 8 chambers per rat, i.e., n=32) two of which were made diabetic (given alloxan i.v. 30 mg/kg). Group 1 was used to measure the permeation of MKC (50 microg/ml) alone (control) while group 2 to measure MKC permeation in the presence of gliclazide (200 microg/ml). The diabetic groups 3 (gliclazide) and 4 (MKC+gliclazide) were treated in the same way. Rats were sacrificed and tissues were mounted into the Ussing chamber for the measurement of MKC mucosal to serosal (absorptive) and serosal to mucosal (secretory) fluxes. In healthy tissues, gliclazide reduced MKC absorptive flux (p < 0.01) and increased its secretory flux (p < 0.01). In diabetic tissues, gliclazide had no effect on either the absorptive or the secretory fluxes of MKC. The lack of effect of gliclazide on MKC permeation in diabetic tissues suggests the absence or suppressed drug transporters. Furthermore, gliclazide inhibition of MKC absorptive flux and induction of MKC secretory flux in healthy tissues may result from the selective inhibition of an efflux drug transporter. 2009 Journal Article http://hdl.handle.net/20.500.11937/25466 Springer France restricted
spellingShingle permeation
Mrp3
transporters
gliclazide
diabetes
bile acid
Al-Salami, Hani
Butt, G.
Tucker, I.
Fawcett, P.
Golocorbin-Kon, S.
Mikov, I.
Mikov, M.
Gliclazide reduces MKC intestinal transport in healthy but not diabetic rats
title Gliclazide reduces MKC intestinal transport in healthy but not diabetic rats
title_full Gliclazide reduces MKC intestinal transport in healthy but not diabetic rats
title_fullStr Gliclazide reduces MKC intestinal transport in healthy but not diabetic rats
title_full_unstemmed Gliclazide reduces MKC intestinal transport in healthy but not diabetic rats
title_short Gliclazide reduces MKC intestinal transport in healthy but not diabetic rats
title_sort gliclazide reduces mkc intestinal transport in healthy but not diabetic rats
topic permeation
Mrp3
transporters
gliclazide
diabetes
bile acid
url http://hdl.handle.net/20.500.11937/25466