Loss of endogenous thymosin β4 accelerates glomerular disease

Loss of endogenous thymosin β4 accelerates glomerular disease

Glomerular disease is characterized by morphologic changes in podocyte cells accompanied by inflammation and fibrosis. Thymosin β4 regulates cell morphology, inflammation, and fibrosis in several organs and administration of exogenous thymosin β4 improves animal models of unilateral ureteral obstruc...

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Bibliographic Details
Main Authors: Vasilopoulou, Elisavet, Kolatsi-Joannou, Maria, Lindenmeyer, Maja T., White, Kathryn E., Robson, Michael G., Cohen, Clemens D., Sebire, Neil J., Riley, Paul R., Winyard, Paul J., Long, David A.
Format: Online
Language:English
Published: Elsevier 2016
Online Access:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5073078/

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https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5073078/

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