Disruption of Slc52a3 gene causes neonatal lethality with riboflavin deficiency in mice

Disruption of Slc52a3 gene causes neonatal lethality with riboflavin deficiency in mice

Homeostasis of riboflavin should be maintained by transporters. Previous in vitro studies have elucidated basic information about riboflavin transporter RFVT3 encoded by SLC52A3 gene. However, the contribution of RFVT3 to the maintenance of riboflavin homeostasis and the significance in vivo remain...

Full description

Bibliographic Details
Main Authors: Yoshimatsu, Hiroki, Yonezawa, Atsushi, Yamanishi, Kaori, Yao, Yoshiaki, Sugano, Kumiko, Nakagawa, Shunsaku, Imai, Satoshi, Omura, Tomohiro, Nakagawa, Takayuki, Yano, Ikuko, Masuda, Satohiro, Inui, Ken-ichi, Matsubara, Kazuo
Format: Online
Language:English
Published: Nature Publishing Group 2016
Online Access:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4897618/

Similar Items

Similar Items