Prenatal inflammation-induced NF-κB dyshomeostasis contributes to renin-angiotensin system over-activity resulting in prenatally programmed hypertension in offspring

Prenatal inflammation-induced NF-κB dyshomeostasis contributes to renin-angiotensin system over-activity resulting in prenatally programmed hypertension in offspring

Studies involving the use of prenatally programmed hypertension have been shown to potentially contribute to prevention of essential hypertension (EH). Our previous research has demonstrated that prenatal inflammatory stimulation leads to offspring’s aortic dysfunction and hypertension in pregnant S...

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Bibliographic Details
Main Authors: Deng, Youcai, Deng, Yafei, He, Xiaoyan, Chu, Jianhong, Zhou, Jianzhi, Zhang, Qi, Guo, Wei, Huang, Pei, Guan, Xiao, Tang, Yuan, Wei, Yanling, Zhao, Shanyu, Zhang, Xingxing, Wei, Chiming, Namaka, Michael, Yi, Ping, Yu, Jianhua, Li, Xiaohui
Format: Online
Language:English
Published: Nature Publishing Group 2016
Online Access:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4753429/

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https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4753429/

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