The HIF-1/glial TIM-3 axis controls inflammation-associated brain damage under hypoxia

The HIF-1/glial TIM-3 axis controls inflammation-associated brain damage under hypoxia

Inflammation is closely related to the extent of damage following cerebral ischaemia, and the targeting of this inflammation has emerged as a promising therapeutic strategy. Here, we present that hypoxia-induced glial T-cell immunoglobulin and mucin domain protein (TIM)-3 can function as a modulator...

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Bibliographic Details
Main Authors: Koh, Han Seok, Chang, Chi Young, Jeon, Sae-Bom, Yoon, Hee Jung, Ahn, Ye-Hyeon, Kim, Hyung-Seok, Kim, In-Hoo, Jeon, Sung Ho, Johnson, Randall S., Park, Eun Jung
Format: Online
Language:English
Published: Nature Pub. Group 2015
Online Access:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4383004/

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https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4383004/

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