Neuroprotection and its molecular mechanism following spinal cord injury☆
Acute spinal cord injury initiates a complex cascade of molecular events termed ‘secondary injury’, which leads to progressive degeneration ranging from early neuronal apoptosis at the lesion site to delayed degeneration of intact white matter tracts, and, ultimately, expansion of the initial injury...
Main Authors: | Liu, Nai-Kui, Xu, Xiao-Ming |
---|---|
Format: | Online |
Language: | English |
Published: |
Medknow Publications & Media Pvt Ltd
2012
|
Online Access: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4296426/ |
Similar Items
-
Neuroprotection by diarylpropionitrile in mice with spinal cord injury
by: Suwanna, Nirut, et al.
Published: (2014) -
Neuroprotective Effect of Ulinastatin on Spinal Cord Ischemia-Reperfusion Injury in Rabbits
by: Liu, Bingbing, et al.
Published: (2015) -
Systemic Bisperoxovanadium Activates Akt/mTOR, Reduces Autophagy, and Enhances Recovery following Cervical Spinal Cord Injury
by: Walker, Chandler L., et al.
Published: (2012) -
Ambulation following spinal cord injury and its correlates
by: Menon, Nitin, et al.
Published: (2015) -
Neuroprotection by Estrogen and Progesterone in Traumatic Brain Injury and Spinal Cord Injury
by: Brotfain, Evgeni, et al.
Published: (2016)