Impaired cortical mitochondrial function following TBI precedes behavioral changes
Traumatic brain injury (TBI) pathophysiology can be attributed to either the immediate, primary physical injury, or the delayed, secondary injury which begins minutes to hours after the initial injury and can persist for several months or longer. Because these secondary cascades are delayed and last...
Main Authors: | Watson, William D., Buonora, John E., Yarnell, Angela M., Lucky, Jessica J., D’Acchille, Michaela I., McMullen, David C., Boston, Andrew G., Kuczmarski, Andrew V., Kean, William S., Verma, Ajay, Grunberg, Neil E., Cole, Jeffrey T. |
---|---|
Format: | Online |
Language: | English |
Published: |
Frontiers Media S.A.
2014
|
Online Access: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3912469/ |
Similar Items
-
A microplate technique to simultaneously assay calcium accumulation in endoplasmic reticulum and SERCA release of inorganic phosphate
by: McMullen, David C, et al.
Published: (2012) -
The cytokine temporal profile in rat cortex after controlled cortical impact
by: Dalgard, Clifton L., et al.
Published: (2012) -
Glucose-6-phosphate reduces calcium accumulation in rat brain endoplasmic reticulum
by: Cole, Jeffrey T., et al.
Published: (2012) -
Statistical Issues in TBI Clinical Studies
by: Rapp, Paul E., et al.
Published: (2013) -
Altered Mitochondrial Dynamics and TBI Pathophysiology
by: Fischer, Tara D., et al.
Published: (2016)