N-n-Butyl Haloperidol Iodide Ameliorates Cardiomyocytes Hypoxia/Reoxygenation Injury by Extracellular Calcium-Dependent and -Independent Mechanisms

N-n-Butyl Haloperidol Iodide Ameliorates Cardiomyocytes Hypoxia/Reoxygenation Injury by Extracellular Calcium-Dependent and -Independent Mechanisms

N-n-butyl haloperidol iodide (F2) has been shown to antagonize myocardial ischemia/reperfusion injury by blocking calcium channels. This study explores the biological functions of ERK pathway in cardiomyocytes hypoxia/reoxygenation injury and clarifies the mechanisms by which F2 ameliorates cardiom...

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Bibliographic Details
Main Authors: Zhang, Yanmei, Chen, Gaoyong, Zhong, Shuping, Zheng, Fuchun, Gao, Fenfei, Chen, Yicun, Huang, Zhanqin, Cai, Wenfeng, Li, Weiqiu, Liu, Xingping, Zheng, Yanshan, Xu, Han, Shi, Ganggang
Format: Online
Language:English
Published: Hindawi Publishing Corporation 2013
Online Access:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3857550/

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https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3857550/

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