Insufficient OPC migration into demyelinated lesions is a cause of poor remyelination in MS and mouse models
Failure of remyelination of multiple sclerosis (MS) lesions contributes to neurodegeneration that correlates with chronic disability in patients. Currently, there are no available treatments to reduce neurodegeneration, but one therapeutic approach to fill this unmet need is to promote remyelination...
Main Authors: | , , |
---|---|
Format: | Online |
Language: | English |
Published: |
Springer-Verlag
2013
|
Online Access: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3661931/ |