Compromised autophagy by MIR30B benefits the intracellular survival of Helicobacter pylori

Compromised autophagy by MIR30B benefits the intracellular survival of Helicobacter pylori

Helicobacter pylori evade immune responses and achieve persistent colonization in the stomach. However, the mechanism by which H. pylori infections persist is not clear. In this study, we showed that MIR30B is upregulated during H. pylori infection of an AGS cell line and human gastric tissues. Upre...

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Bibliographic Details
Main Authors: Tang, Bin, Li, Na, Gu, Jiang, Zhuang, Yuan, Li, Qian, Wang, Hai-Guang, Fang, Yao, Yu, Bo, Zhang, Jin-Yu, Xie, Qing-Hua, Chen, Li, Jiang, Xue-Jun, Xiao, Bin, Zou, Quan-Ming, Mao, Xu-Hu
Format: Online
Language:English
Published: Landes Bioscience 2012
Online Access:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3429542/

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3429542/

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