Glucose regulates clathrin adaptors at the trans-Golgi network and endosomes
Traffic at the trans-Golgi network (TGN) and endosomes is regulated by glucose via an unknown mechanism that depends on protein kinase A (PKA). TGN–endosomal clathrin adaptors exhibit specific responses to glucose starvation that likely are coordinated with other cell behaviors regulated by PKA.
| Main Authors: | , , |
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| Format: | Online |
| Language: | English |
| Published: |
The American Society for Cell Biology
2011
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| Online Access: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3183021/ |
| Summary: | Traffic at the trans-Golgi network (TGN) and endosomes is regulated by glucose via an unknown mechanism that depends on protein kinase A (PKA). TGN–endosomal clathrin adaptors exhibit specific responses to glucose starvation that likely are coordinated with other cell behaviors regulated by PKA. |
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