Blockade of eosinophil-induced bronchial epithelial-mesenchymal transition with a geranyl acetophenone in a coculture model

Epithelial-mesenchymal transition (EMT) is currently recognized as the main cellular event that contributes to airway remodeling. Eosinophils can induce EMT in airway epithelial cells via increased transforming growth factor (TGF)-β production. We assessed the effect of synthetic 2,4,6-trihydroxy-3-...

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Main Authors: Lee, Yu Zhao, Yap, Hui Min, Shaari, Khozirah, Tham, Chau Ling, Sulaiman, Mohd Roslan, Israf Ali, Daud Ahmad
Format: Article
Language:English
Published: Frontiers Research Foundation 2017
Online Access:http://psasir.upm.edu.my/id/eprint/61018/
http://psasir.upm.edu.my/id/eprint/61018/1/Blockade%20of%20eosinophil-induced%20bronchial%20epithelial-mesenchymal%20transition%20with%20a%20geranyl%20acetophenone%20in%20a%20coculture%20model.pdf
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author Lee, Yu Zhao
Yap, Hui Min
Shaari, Khozirah
Tham, Chau Ling
Sulaiman, Mohd Roslan
Israf Ali, Daud Ahmad
author_facet Lee, Yu Zhao
Yap, Hui Min
Shaari, Khozirah
Tham, Chau Ling
Sulaiman, Mohd Roslan
Israf Ali, Daud Ahmad
author_sort Lee, Yu Zhao
building UPM Institutional Repository
collection Online Access
description Epithelial-mesenchymal transition (EMT) is currently recognized as the main cellular event that contributes to airway remodeling. Eosinophils can induce EMT in airway epithelial cells via increased transforming growth factor (TGF)-β production. We assessed the effect of synthetic 2,4,6-trihydroxy-3-geranyl acetophenone (tHGA) upon eosinophil-induced EMT in a cellular model. The human eosinophil cell line EoL-1 was used to induce EMT in BEAS-2B human bronchial epithelial cells. The induction of EMT was dose-dependently suppressed following tHGA treatment in which the epithelial morphology and E-cadherin expression were not altered. Protein and mRNA expression of vimentin, collagen I and fibronectin in eosinophil-induced epithelial cells were also significantly suppressed by tHGA treatment. Following pathway analysis, we showed that tHGA suppressed eosinophil-induced activator protein-1-mediated TGF-β production by targeting c-Jun N-terminal kinase and phosphoinositide 3-kinase signaling pathways. These findings corroborated previous findings on the ability of tHGA to inhibit experimental murine airway remodeling.
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spelling upm-610182019-04-30T03:45:34Z http://psasir.upm.edu.my/id/eprint/61018/ Blockade of eosinophil-induced bronchial epithelial-mesenchymal transition with a geranyl acetophenone in a coculture model Lee, Yu Zhao Yap, Hui Min Shaari, Khozirah Tham, Chau Ling Sulaiman, Mohd Roslan Israf Ali, Daud Ahmad Epithelial-mesenchymal transition (EMT) is currently recognized as the main cellular event that contributes to airway remodeling. Eosinophils can induce EMT in airway epithelial cells via increased transforming growth factor (TGF)-β production. We assessed the effect of synthetic 2,4,6-trihydroxy-3-geranyl acetophenone (tHGA) upon eosinophil-induced EMT in a cellular model. The human eosinophil cell line EoL-1 was used to induce EMT in BEAS-2B human bronchial epithelial cells. The induction of EMT was dose-dependently suppressed following tHGA treatment in which the epithelial morphology and E-cadherin expression were not altered. Protein and mRNA expression of vimentin, collagen I and fibronectin in eosinophil-induced epithelial cells were also significantly suppressed by tHGA treatment. Following pathway analysis, we showed that tHGA suppressed eosinophil-induced activator protein-1-mediated TGF-β production by targeting c-Jun N-terminal kinase and phosphoinositide 3-kinase signaling pathways. These findings corroborated previous findings on the ability of tHGA to inhibit experimental murine airway remodeling. Frontiers Research Foundation 2017 Article PeerReviewed text en http://psasir.upm.edu.my/id/eprint/61018/1/Blockade%20of%20eosinophil-induced%20bronchial%20epithelial-mesenchymal%20transition%20with%20a%20geranyl%20acetophenone%20in%20a%20coculture%20model.pdf Lee, Yu Zhao and Yap, Hui Min and Shaari, Khozirah and Tham, Chau Ling and Sulaiman, Mohd Roslan and Israf Ali, Daud Ahmad (2017) Blockade of eosinophil-induced bronchial epithelial-mesenchymal transition with a geranyl acetophenone in a coculture model. Frontiers in Pharmacology, 8. pp. 1-13. ISSN ESSN: 1663-9812 https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5696322/pdf/fphar-08-00837.pdf 10.3389/fphar.2017.00837
spellingShingle Lee, Yu Zhao
Yap, Hui Min
Shaari, Khozirah
Tham, Chau Ling
Sulaiman, Mohd Roslan
Israf Ali, Daud Ahmad
Blockade of eosinophil-induced bronchial epithelial-mesenchymal transition with a geranyl acetophenone in a coculture model
title Blockade of eosinophil-induced bronchial epithelial-mesenchymal transition with a geranyl acetophenone in a coculture model
title_full Blockade of eosinophil-induced bronchial epithelial-mesenchymal transition with a geranyl acetophenone in a coculture model
title_fullStr Blockade of eosinophil-induced bronchial epithelial-mesenchymal transition with a geranyl acetophenone in a coculture model
title_full_unstemmed Blockade of eosinophil-induced bronchial epithelial-mesenchymal transition with a geranyl acetophenone in a coculture model
title_short Blockade of eosinophil-induced bronchial epithelial-mesenchymal transition with a geranyl acetophenone in a coculture model
title_sort blockade of eosinophil-induced bronchial epithelial-mesenchymal transition with a geranyl acetophenone in a coculture model
url http://psasir.upm.edu.my/id/eprint/61018/
http://psasir.upm.edu.my/id/eprint/61018/
http://psasir.upm.edu.my/id/eprint/61018/
http://psasir.upm.edu.my/id/eprint/61018/1/Blockade%20of%20eosinophil-induced%20bronchial%20epithelial-mesenchymal%20transition%20with%20a%20geranyl%20acetophenone%20in%20a%20coculture%20model.pdf