Immuno-pathophysiological responses of mouse model to experimental infection with Brucella melitensis and its lipopolysaccharides via intraperitoneal route

Brucella melitensis is one of the major zoonotic pathogens with significant economic implications worldwide. The pathogenicity is complex and not always well understood. Lipopolysaccharide (LPS) remains the major virulent factor of B. melitensis and responsible for the mechanism by which the pathoge...

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Main Authors: Osman, Abdinasir Yusuf, Abdullah, Faez Firdaus Jesse, Abdul Kadir, Arifah, Saharee, Abdul Aziz
Format: Article
Language:English
Published: Elsevier 2016
Subjects:
Online Access:http://psasir.upm.edu.my/id/eprint/54498/
http://psasir.upm.edu.my/id/eprint/54498/1/Immuno-pathophysiological%20responses%20of%20mouse%20model%20to%20experimental%20infection%20with%20Brucella%20melitensis%20and%20its%20lipopolysaccharides%20via%20intraperitoneal%20route.pdf
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author Osman, Abdinasir Yusuf
Abdullah, Faez Firdaus Jesse
Abdul Kadir, Arifah
Saharee, Abdul Aziz
author_facet Osman, Abdinasir Yusuf
Abdullah, Faez Firdaus Jesse
Abdul Kadir, Arifah
Saharee, Abdul Aziz
author_sort Osman, Abdinasir Yusuf
building UPM Institutional Repository
collection Online Access
description Brucella melitensis is one of the major zoonotic pathogens with significant economic implications worldwide. The pathogenicity is complex and not always well understood. Lipopolysaccharide (LPS) remains the major virulent factor of B. melitensis and responsible for the mechanism by which the pathogen causes its deleterious effects. In this study, 84 mice of 6–8 weeks old of both sexes were divided equally into 3 groups; namely Brucella melitensis infected group, lipopolysaccharide (LPS) infected group and control group. The former two groups contained 36 mice each with equal gender distribution. The control group consisted of 12 mice only. Animals in B. melitensis infected group, a single inoculum of 0.4 ml containing 109 of B. melitensis were intraperitoneally challenged while animals in LPS group, a single dose of 0.4 ml containing LPS extracted from the B. melitensis were intraperitoneally inoculated. Animals in control group received intraperitoneally, a single dose of 0.4 ml phosphate buffered saline (PBS) of pH7. Animals that were infected intraperitoneally with B. melitensis demonstrated significant clinical presentation; gross and histo-pathological evidence than LPS infected group. However, both infected groups showed elevated levels of interleukins (IL-1β and IL6), antibody levels (IgM an IgG) as early as 3 days post-infection with predominance in LPS infected group. In contrast, low levels of sex related hormonal changes in which LPS infected group showed the least concentration were also detected throughout the experimental period. In conclusion, B. melitensis can be transmitted via gastrointestinal, respiratory and reproductive tract. Moreover, LPS stimulated significantly the innate and acquired immune system without significant systemic dysfunction, suggesting potentiality of the protective properties of this component as alternative vaccine for brucellosis infection.
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spelling upm-544982018-03-21T03:37:30Z http://psasir.upm.edu.my/id/eprint/54498/ Immuno-pathophysiological responses of mouse model to experimental infection with Brucella melitensis and its lipopolysaccharides via intraperitoneal route Osman, Abdinasir Yusuf Abdullah, Faez Firdaus Jesse Abdul Kadir, Arifah Saharee, Abdul Aziz Brucella melitensis is one of the major zoonotic pathogens with significant economic implications worldwide. The pathogenicity is complex and not always well understood. Lipopolysaccharide (LPS) remains the major virulent factor of B. melitensis and responsible for the mechanism by which the pathogen causes its deleterious effects. In this study, 84 mice of 6–8 weeks old of both sexes were divided equally into 3 groups; namely Brucella melitensis infected group, lipopolysaccharide (LPS) infected group and control group. The former two groups contained 36 mice each with equal gender distribution. The control group consisted of 12 mice only. Animals in B. melitensis infected group, a single inoculum of 0.4 ml containing 109 of B. melitensis were intraperitoneally challenged while animals in LPS group, a single dose of 0.4 ml containing LPS extracted from the B. melitensis were intraperitoneally inoculated. Animals in control group received intraperitoneally, a single dose of 0.4 ml phosphate buffered saline (PBS) of pH7. Animals that were infected intraperitoneally with B. melitensis demonstrated significant clinical presentation; gross and histo-pathological evidence than LPS infected group. However, both infected groups showed elevated levels of interleukins (IL-1β and IL6), antibody levels (IgM an IgG) as early as 3 days post-infection with predominance in LPS infected group. In contrast, low levels of sex related hormonal changes in which LPS infected group showed the least concentration were also detected throughout the experimental period. In conclusion, B. melitensis can be transmitted via gastrointestinal, respiratory and reproductive tract. Moreover, LPS stimulated significantly the innate and acquired immune system without significant systemic dysfunction, suggesting potentiality of the protective properties of this component as alternative vaccine for brucellosis infection. Elsevier 2016-11 Article PeerReviewed text en http://psasir.upm.edu.my/id/eprint/54498/1/Immuno-pathophysiological%20responses%20of%20mouse%20model%20to%20experimental%20infection%20with%20Brucella%20melitensis%20and%20its%20lipopolysaccharides%20via%20intraperitoneal%20route.pdf Osman, Abdinasir Yusuf and Abdullah, Faez Firdaus Jesse and Abdul Kadir, Arifah and Saharee, Abdul Aziz (2016) Immuno-pathophysiological responses of mouse model to experimental infection with Brucella melitensis and its lipopolysaccharides via intraperitoneal route. Microbial Pathogenesis, 100. pp. 17-29. ISSN 0882-4010; ESSN: 1096-1208 https://www.sciencedirect.com/science/article/pii/S0882401016300602?via%3Dihub Brucella melitensis; Lipopolysaccharide; Mouse model; Intraperitoneal route of infection; Clinico-pathological; Histopathology; Cytokine; Antibody; Hormone; PCR 10.1016/j.micpath.2016.08.019
spellingShingle Brucella melitensis; Lipopolysaccharide; Mouse model; Intraperitoneal route of infection; Clinico-pathological; Histopathology; Cytokine; Antibody; Hormone; PCR
Osman, Abdinasir Yusuf
Abdullah, Faez Firdaus Jesse
Abdul Kadir, Arifah
Saharee, Abdul Aziz
Immuno-pathophysiological responses of mouse model to experimental infection with Brucella melitensis and its lipopolysaccharides via intraperitoneal route
title Immuno-pathophysiological responses of mouse model to experimental infection with Brucella melitensis and its lipopolysaccharides via intraperitoneal route
title_full Immuno-pathophysiological responses of mouse model to experimental infection with Brucella melitensis and its lipopolysaccharides via intraperitoneal route
title_fullStr Immuno-pathophysiological responses of mouse model to experimental infection with Brucella melitensis and its lipopolysaccharides via intraperitoneal route
title_full_unstemmed Immuno-pathophysiological responses of mouse model to experimental infection with Brucella melitensis and its lipopolysaccharides via intraperitoneal route
title_short Immuno-pathophysiological responses of mouse model to experimental infection with Brucella melitensis and its lipopolysaccharides via intraperitoneal route
title_sort immuno-pathophysiological responses of mouse model to experimental infection with brucella melitensis and its lipopolysaccharides via intraperitoneal route
topic Brucella melitensis; Lipopolysaccharide; Mouse model; Intraperitoneal route of infection; Clinico-pathological; Histopathology; Cytokine; Antibody; Hormone; PCR
url http://psasir.upm.edu.my/id/eprint/54498/
http://psasir.upm.edu.my/id/eprint/54498/
http://psasir.upm.edu.my/id/eprint/54498/
http://psasir.upm.edu.my/id/eprint/54498/1/Immuno-pathophysiological%20responses%20of%20mouse%20model%20to%20experimental%20infection%20with%20Brucella%20melitensis%20and%20its%20lipopolysaccharides%20via%20intraperitoneal%20route.pdf