Effects of plasticizer diisobutyl adipate on the Japanese medaka (Oryzias latipes) endocrine system

Plasticizer pollution of the water environment is one of the world's most serious environmental issues. Phthalate plasticizers can disrupt endocrine function in vertebrates. Therefore, this study analyzed thyroid-related, reproduction-related, and estrogenresponsive genes in Japanese medaka (Or...

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Main Authors: Horie, Yoshifumi, Ramaswamy, Babu Rajendran, Ríos, Juan Manuel, Chee, Kong Yap, Okamura, Hideo
Format: Article
Language:English
Published: John Wiley and Sons 2023
Online Access:http://psasir.upm.edu.my/id/eprint/118432/
http://psasir.upm.edu.my/id/eprint/118432/1/118432.pdf
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author Horie, Yoshifumi
Ramaswamy, Babu Rajendran
Ríos, Juan Manuel
Chee, Kong Yap
Okamura, Hideo
author_facet Horie, Yoshifumi
Ramaswamy, Babu Rajendran
Ríos, Juan Manuel
Chee, Kong Yap
Okamura, Hideo
author_sort Horie, Yoshifumi
building UPM Institutional Repository
collection Online Access
description Plasticizer pollution of the water environment is one of the world's most serious environmental issues. Phthalate plasticizers can disrupt endocrine function in vertebrates. Therefore, this study analyzed thyroid-related, reproduction-related, and estrogenresponsive genes in Japanese medaka (Oryzias latipes) to determine whether nonphthalate diisobutyl adipate (DIBA) plasticizer could affect endocrine hormone activity or not. Developmental toxicity during fish embryogenesis was also evaluated. At a concentration of 11.57 mg/l, embryonic exposure to DIBA increased the mortality rate. Although abnormal development, including body curvature, edema, and lack of swim bladder inflation, was observed at 3.54 and 11.57 mg/l DIBA, growth inhibition and reduced swimming performance were also observed. In addition, DIBA exposure increased the levels of thyroid-stimulating hormone beta-subunit (tshβ) and deiodinase 1(dio1) but decreased the levels of thyroid hormone receptor alpha (trα) and beta (trβ). These results suggest that DIBA has thyroid hormone-disrupting activities in fish. However, kisspeptin (kiss1 and kiss2), gonadotropin-releasing hormone (gnrh1), folliclestimulating hormone beta (fshβ), luteinizing hormone beta (lhβ), choriogenin H (chgH), and vitellogenin (vtg1) expression did not change dose-dependently in response to DIBA exposure, whereas gnrh2 and vtg2 expression was elevated. These results indicate that DIBA has low estrogenic activity and does not disrupt the endocrine reproduction system in fish. Overall, this is the first report indicating that non-phthalate DIBA plasticizer is embryotoxic and disrupt thyroid hormone activity in fish.
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spelling upm-1184322025-08-05T08:39:35Z http://psasir.upm.edu.my/id/eprint/118432/ Effects of plasticizer diisobutyl adipate on the Japanese medaka (Oryzias latipes) endocrine system Horie, Yoshifumi Ramaswamy, Babu Rajendran Ríos, Juan Manuel Chee, Kong Yap Okamura, Hideo Plasticizer pollution of the water environment is one of the world's most serious environmental issues. Phthalate plasticizers can disrupt endocrine function in vertebrates. Therefore, this study analyzed thyroid-related, reproduction-related, and estrogenresponsive genes in Japanese medaka (Oryzias latipes) to determine whether nonphthalate diisobutyl adipate (DIBA) plasticizer could affect endocrine hormone activity or not. Developmental toxicity during fish embryogenesis was also evaluated. At a concentration of 11.57 mg/l, embryonic exposure to DIBA increased the mortality rate. Although abnormal development, including body curvature, edema, and lack of swim bladder inflation, was observed at 3.54 and 11.57 mg/l DIBA, growth inhibition and reduced swimming performance were also observed. In addition, DIBA exposure increased the levels of thyroid-stimulating hormone beta-subunit (tshβ) and deiodinase 1(dio1) but decreased the levels of thyroid hormone receptor alpha (trα) and beta (trβ). These results suggest that DIBA has thyroid hormone-disrupting activities in fish. However, kisspeptin (kiss1 and kiss2), gonadotropin-releasing hormone (gnrh1), folliclestimulating hormone beta (fshβ), luteinizing hormone beta (lhβ), choriogenin H (chgH), and vitellogenin (vtg1) expression did not change dose-dependently in response to DIBA exposure, whereas gnrh2 and vtg2 expression was elevated. These results indicate that DIBA has low estrogenic activity and does not disrupt the endocrine reproduction system in fish. Overall, this is the first report indicating that non-phthalate DIBA plasticizer is embryotoxic and disrupt thyroid hormone activity in fish. John Wiley and Sons 2023 Article PeerReviewed text en http://psasir.upm.edu.my/id/eprint/118432/1/118432.pdf Horie, Yoshifumi and Ramaswamy, Babu Rajendran and Ríos, Juan Manuel and Chee, Kong Yap and Okamura, Hideo (2023) Effects of plasticizer diisobutyl adipate on the Japanese medaka (Oryzias latipes) endocrine system. Journal of Applied Toxicology, 43 (7). pp. 982-992. ISSN 0260-437X; eISSN: 1099-1263 https://analyticalsciencejournals.onlinelibrary.wiley.com/doi/10.1002/jat.4437 10.1002/jat.4437
spellingShingle Horie, Yoshifumi
Ramaswamy, Babu Rajendran
Ríos, Juan Manuel
Chee, Kong Yap
Okamura, Hideo
Effects of plasticizer diisobutyl adipate on the Japanese medaka (Oryzias latipes) endocrine system
title Effects of plasticizer diisobutyl adipate on the Japanese medaka (Oryzias latipes) endocrine system
title_full Effects of plasticizer diisobutyl adipate on the Japanese medaka (Oryzias latipes) endocrine system
title_fullStr Effects of plasticizer diisobutyl adipate on the Japanese medaka (Oryzias latipes) endocrine system
title_full_unstemmed Effects of plasticizer diisobutyl adipate on the Japanese medaka (Oryzias latipes) endocrine system
title_short Effects of plasticizer diisobutyl adipate on the Japanese medaka (Oryzias latipes) endocrine system
title_sort effects of plasticizer diisobutyl adipate on the japanese medaka (oryzias latipes) endocrine system
url http://psasir.upm.edu.my/id/eprint/118432/
http://psasir.upm.edu.my/id/eprint/118432/
http://psasir.upm.edu.my/id/eprint/118432/
http://psasir.upm.edu.my/id/eprint/118432/1/118432.pdf