Variant identification using whole exome sequencing in a family suspected with Iron-refractory Iron Deficiency Anaemia

Iron-refractory iron deficiency anaemia (IRIDA) is a rare type of anaemia that affects about 1 in 1 million people globally. The inheritance pattern is autosomal recessive, which is caused by a defective Transmembrane Protease Serine 6 (TMPRSS6) gene. Patients with TMPRSS6 variants exhibit microc...

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Main Authors: Karuppiah, Thilakavathy, Mohamed Ibrahim, Noor Haliza, Musa, Nurul Huda, Shankar, Aissvarya, Ahmad Asnawi, Asral Wirda, Yap, Mandy Yee Yee, Sathar, Jameela, Selvaratnam, Veena, Noor Alif Wira, Nurul Ain Suraya
Format: Conference or Workshop Item
Language:English
Published: UPM 2024
Online Access:http://psasir.upm.edu.my/id/eprint/116509/
http://psasir.upm.edu.my/id/eprint/116509/1/116509.pdf
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author Karuppiah, Thilakavathy
Mohamed Ibrahim, Noor Haliza
Musa, Nurul Huda
Shankar, Aissvarya
Ahmad Asnawi, Asral Wirda
Yap, Mandy Yee Yee
Sathar, Jameela
Selvaratnam, Veena
Noor Alif Wira, Nurul Ain Suraya
author_facet Karuppiah, Thilakavathy
Mohamed Ibrahim, Noor Haliza
Musa, Nurul Huda
Shankar, Aissvarya
Ahmad Asnawi, Asral Wirda
Yap, Mandy Yee Yee
Sathar, Jameela
Selvaratnam, Veena
Noor Alif Wira, Nurul Ain Suraya
author_sort Karuppiah, Thilakavathy
building UPM Institutional Repository
collection Online Access
description Iron-refractory iron deficiency anaemia (IRIDA) is a rare type of anaemia that affects about 1 in 1 million people globally. The inheritance pattern is autosomal recessive, which is caused by a defective Transmembrane Protease Serine 6 (TMPRSS6) gene. Patients with TMPRSS6 variants exhibit microcytic hypochromicity, low serum iron and transferrin saturation (TSAT), relatively low serum ferritin, normal to high hepcidin, and unresponsive to oral iron. Based on our knowledge, no genetic studies have been conducted on IRIDA in Malaysia. This study aims to describe the molecular genetic findings in a family suspected of IRIDA using whole exome sequencing (WES). Two male siblings with persistent and unexplained anaemia and three male family members who were not having symptoms were recruited for this study at the Anaemia Clinic, Hospital Ampang. DNA was extracted from blood samples and sent for WES service. Bioinformatic analysis was performed to identify the presence of gene variants related to IRIDA. Gennext software (Version: 0.1.0) was utilised for variant annotation, and various databases were used for the variant filtering and prioritisation process such as NCBI, OMIM, HPO, etc. Haematological parameters revealed that the two suspected sons had microcytic hypochromic anaemia, low haemoglobin and TSAT, and normal serum hepcidin. The proband had low serum iron and normal ferritin, while the other suspected son had relatively low serum iron and ferritin. All the other three male family members showed normal haematological parameters. Pathogenic variants were not found in the family members; however, common SNPs related to IRIDA were identified. All five family members carried homozygous TMPRSS6/rs855791 (NM_001289000.1:c.2246T>C) and SLC11A2/rs445520 (NM_001174130.2:c.17A>C). The two suspected siblings carried heterozygous TMPRSS6/rs2235324 (NM_001374504.1:c.730A>G) but not the TMPRSS6/rs78174698 (NM_001374504.1:c.1636C>T) that was found in the father and first son. In addition, the proband was found to carry a homozygous splice-site deletion variant, TMPRSS6/rs60484081 (NM_001374504.1:c.1842-6_1842-2delCTGGGG). The haematological parameters of the suspected siblings point towards the IRIDA phenotype. rs78174698 was reported to increase serum ferritin, whereas the absence of this variant probably reduces serum ferritin in the suspected siblings, an indication of IRIDA. rs60484081 could be a modifier SNP that causes reduced penetrance in the proband, showing less severe symptoms compared to his brother. These findings emphasised that WES can be used in diagnosing underlying IRIDA that affect this family. Thus, advancements in diagnostic tools help families in need get better treatment for IRIDA.
first_indexed 2025-11-15T14:29:52Z
format Conference or Workshop Item
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institution Universiti Putra Malaysia
institution_category Local University
language English
last_indexed 2025-11-15T14:29:52Z
publishDate 2024
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spelling upm-1165092025-04-09T07:02:10Z http://psasir.upm.edu.my/id/eprint/116509/ Variant identification using whole exome sequencing in a family suspected with Iron-refractory Iron Deficiency Anaemia Karuppiah, Thilakavathy Mohamed Ibrahim, Noor Haliza Musa, Nurul Huda Shankar, Aissvarya Ahmad Asnawi, Asral Wirda Yap, Mandy Yee Yee Sathar, Jameela Selvaratnam, Veena Noor Alif Wira, Nurul Ain Suraya Iron-refractory iron deficiency anaemia (IRIDA) is a rare type of anaemia that affects about 1 in 1 million people globally. The inheritance pattern is autosomal recessive, which is caused by a defective Transmembrane Protease Serine 6 (TMPRSS6) gene. Patients with TMPRSS6 variants exhibit microcytic hypochromicity, low serum iron and transferrin saturation (TSAT), relatively low serum ferritin, normal to high hepcidin, and unresponsive to oral iron. Based on our knowledge, no genetic studies have been conducted on IRIDA in Malaysia. This study aims to describe the molecular genetic findings in a family suspected of IRIDA using whole exome sequencing (WES). Two male siblings with persistent and unexplained anaemia and three male family members who were not having symptoms were recruited for this study at the Anaemia Clinic, Hospital Ampang. DNA was extracted from blood samples and sent for WES service. Bioinformatic analysis was performed to identify the presence of gene variants related to IRIDA. Gennext software (Version: 0.1.0) was utilised for variant annotation, and various databases were used for the variant filtering and prioritisation process such as NCBI, OMIM, HPO, etc. Haematological parameters revealed that the two suspected sons had microcytic hypochromic anaemia, low haemoglobin and TSAT, and normal serum hepcidin. The proband had low serum iron and normal ferritin, while the other suspected son had relatively low serum iron and ferritin. All the other three male family members showed normal haematological parameters. Pathogenic variants were not found in the family members; however, common SNPs related to IRIDA were identified. All five family members carried homozygous TMPRSS6/rs855791 (NM_001289000.1:c.2246T>C) and SLC11A2/rs445520 (NM_001174130.2:c.17A>C). The two suspected siblings carried heterozygous TMPRSS6/rs2235324 (NM_001374504.1:c.730A>G) but not the TMPRSS6/rs78174698 (NM_001374504.1:c.1636C>T) that was found in the father and first son. In addition, the proband was found to carry a homozygous splice-site deletion variant, TMPRSS6/rs60484081 (NM_001374504.1:c.1842-6_1842-2delCTGGGG). The haematological parameters of the suspected siblings point towards the IRIDA phenotype. rs78174698 was reported to increase serum ferritin, whereas the absence of this variant probably reduces serum ferritin in the suspected siblings, an indication of IRIDA. rs60484081 could be a modifier SNP that causes reduced penetrance in the proband, showing less severe symptoms compared to his brother. These findings emphasised that WES can be used in diagnosing underlying IRIDA that affect this family. Thus, advancements in diagnostic tools help families in need get better treatment for IRIDA. UPM 2024 Conference or Workshop Item PeerReviewed text en http://psasir.upm.edu.my/id/eprint/116509/1/116509.pdf Karuppiah, Thilakavathy and Mohamed Ibrahim, Noor Haliza and Musa, Nurul Huda and Shankar, Aissvarya and Ahmad Asnawi, Asral Wirda and Yap, Mandy Yee Yee and Sathar, Jameela and Selvaratnam, Veena and Noor Alif Wira, Nurul Ain Suraya (2024) Variant identification using whole exome sequencing in a family suspected with Iron-refractory Iron Deficiency Anaemia. In: 14th Malaysian Symposium of Biomedical Science 2024, 1-2 Jun 2024, Universiti Putra Malaysia, Serdang. (p. 40).
spellingShingle Karuppiah, Thilakavathy
Mohamed Ibrahim, Noor Haliza
Musa, Nurul Huda
Shankar, Aissvarya
Ahmad Asnawi, Asral Wirda
Yap, Mandy Yee Yee
Sathar, Jameela
Selvaratnam, Veena
Noor Alif Wira, Nurul Ain Suraya
Variant identification using whole exome sequencing in a family suspected with Iron-refractory Iron Deficiency Anaemia
title Variant identification using whole exome sequencing in a family suspected with Iron-refractory Iron Deficiency Anaemia
title_full Variant identification using whole exome sequencing in a family suspected with Iron-refractory Iron Deficiency Anaemia
title_fullStr Variant identification using whole exome sequencing in a family suspected with Iron-refractory Iron Deficiency Anaemia
title_full_unstemmed Variant identification using whole exome sequencing in a family suspected with Iron-refractory Iron Deficiency Anaemia
title_short Variant identification using whole exome sequencing in a family suspected with Iron-refractory Iron Deficiency Anaemia
title_sort variant identification using whole exome sequencing in a family suspected with iron-refractory iron deficiency anaemia
url http://psasir.upm.edu.my/id/eprint/116509/
http://psasir.upm.edu.my/id/eprint/116509/1/116509.pdf