Antioxidant-mediated protective role of Hericium erinaceus (Bull.: Fr.) Pers. against oxidative damage in fibroblasts from Friedreich’s ataxia patient

Friedreich’s ataxia (FRDA) is a progressive neuromuscular disorder caused by substantial decrease of mitochondrial protein frataxin responsible for biogenesis of iron-sulphur clusters and protection from oxidative damage. In this study, we investigated the antioxidant activities of a standardized aq...

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Main Authors: Lew, S. Y., Yow, Yoon Yen *, Lim, L. W., Wong, K. H.
Format: Article
Language:English
Published: Sociedade Brasileira de Ciência e Tecnologia de Alimentos 2020
Subjects:
Online Access:http://eprints.sunway.edu.my/1721/
http://eprints.sunway.edu.my/1721/1/Yow%20Yoon%20Yen%20Antioxidant%20mediated.pdf
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author Lew, S. Y.
Yow, Yoon Yen *
Lim, L. W.
Wong, K. H.
author_facet Lew, S. Y.
Yow, Yoon Yen *
Lim, L. W.
Wong, K. H.
author_sort Lew, S. Y.
building SU Institutional Repository
collection Online Access
description Friedreich’s ataxia (FRDA) is a progressive neuromuscular disorder caused by substantial decrease of mitochondrial protein frataxin responsible for biogenesis of iron-sulphur clusters and protection from oxidative damage. In this study, we investigated the antioxidant activities of a standardized aqueous extract from fruiting bodies of Hericium erinaceus mushroom (HESAE) and its protective effects against oxidative damage induced by L-Buthionine sulfoximine (BSO) in fibroblasts derived from FRDA patient. The lactate dehydrogenase-based viability assay showed that FRDA fibroblast was sensitive to 12.5 mM BSO with a reduction of viability to 52.51 ± 13.92% after 24 h of BSO exposure. Interestingly, co-incubation with 32 mg/mL HESAE increased the viability to 85.35 ± 3.4%. Further, 12.5 mM BSO caused a decrease in the ratio of cellular reduced glutathione (GSH) to oxidised GSH (GSSG) that leads to cell death. Nevertheless, the damage was reduced by co-incubation with 32 mg/mL HESAE. Nuclear fluorescence staining revealed that 12.5 mM BSO induced cell death and the apoptosis was decreased by co-incubation with HESAE. These findings suggest the ability of HESAE in attenuating BSO-mediated cytotoxicity through maintenance of membrane integrity and optimal GSH/GSSG ratio, that are closely linked to its antioxidant activities. Further in vivo trials are highly warranted to clarify its potential benefits in management of FRDA.
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spelling sunway-17212021-04-14T07:37:21Z http://eprints.sunway.edu.my/1721/ Antioxidant-mediated protective role of Hericium erinaceus (Bull.: Fr.) Pers. against oxidative damage in fibroblasts from Friedreich’s ataxia patient Lew, S. Y. Yow, Yoon Yen * Lim, L. W. Wong, K. H. QH301 Biology Friedreich’s ataxia (FRDA) is a progressive neuromuscular disorder caused by substantial decrease of mitochondrial protein frataxin responsible for biogenesis of iron-sulphur clusters and protection from oxidative damage. In this study, we investigated the antioxidant activities of a standardized aqueous extract from fruiting bodies of Hericium erinaceus mushroom (HESAE) and its protective effects against oxidative damage induced by L-Buthionine sulfoximine (BSO) in fibroblasts derived from FRDA patient. The lactate dehydrogenase-based viability assay showed that FRDA fibroblast was sensitive to 12.5 mM BSO with a reduction of viability to 52.51 ± 13.92% after 24 h of BSO exposure. Interestingly, co-incubation with 32 mg/mL HESAE increased the viability to 85.35 ± 3.4%. Further, 12.5 mM BSO caused a decrease in the ratio of cellular reduced glutathione (GSH) to oxidised GSH (GSSG) that leads to cell death. Nevertheless, the damage was reduced by co-incubation with 32 mg/mL HESAE. Nuclear fluorescence staining revealed that 12.5 mM BSO induced cell death and the apoptosis was decreased by co-incubation with HESAE. These findings suggest the ability of HESAE in attenuating BSO-mediated cytotoxicity through maintenance of membrane integrity and optimal GSH/GSSG ratio, that are closely linked to its antioxidant activities. Further in vivo trials are highly warranted to clarify its potential benefits in management of FRDA. Sociedade Brasileira de Ciência e Tecnologia de Alimentos 2020 Article PeerReviewed text en cc_by_nc_4 http://eprints.sunway.edu.my/1721/1/Yow%20Yoon%20Yen%20Antioxidant%20mediated.pdf Lew, S. Y. and Yow, Yoon Yen * and Lim, L. W. and Wong, K. H. (2020) Antioxidant-mediated protective role of Hericium erinaceus (Bull.: Fr.) Pers. against oxidative damage in fibroblasts from Friedreich’s ataxia patient. Food Science and Technology, 40 (suppl). pp. 264-272. ISSN 0101-2061 http://doi.org/10.1590/fst.09919 doi:10.1590/fst.09919
spellingShingle QH301 Biology
Lew, S. Y.
Yow, Yoon Yen *
Lim, L. W.
Wong, K. H.
Antioxidant-mediated protective role of Hericium erinaceus (Bull.: Fr.) Pers. against oxidative damage in fibroblasts from Friedreich’s ataxia patient
title Antioxidant-mediated protective role of Hericium erinaceus (Bull.: Fr.) Pers. against oxidative damage in fibroblasts from Friedreich’s ataxia patient
title_full Antioxidant-mediated protective role of Hericium erinaceus (Bull.: Fr.) Pers. against oxidative damage in fibroblasts from Friedreich’s ataxia patient
title_fullStr Antioxidant-mediated protective role of Hericium erinaceus (Bull.: Fr.) Pers. against oxidative damage in fibroblasts from Friedreich’s ataxia patient
title_full_unstemmed Antioxidant-mediated protective role of Hericium erinaceus (Bull.: Fr.) Pers. against oxidative damage in fibroblasts from Friedreich’s ataxia patient
title_short Antioxidant-mediated protective role of Hericium erinaceus (Bull.: Fr.) Pers. against oxidative damage in fibroblasts from Friedreich’s ataxia patient
title_sort antioxidant-mediated protective role of hericium erinaceus (bull.: fr.) pers. against oxidative damage in fibroblasts from friedreich’s ataxia patient
topic QH301 Biology
url http://eprints.sunway.edu.my/1721/
http://eprints.sunway.edu.my/1721/
http://eprints.sunway.edu.my/1721/
http://eprints.sunway.edu.my/1721/1/Yow%20Yoon%20Yen%20Antioxidant%20mediated.pdf