Understanding the ZEB1 interactome during lymphangiogenesis

Endothelial cells (ECs) constitute the crucial lining of blood and lymphatic vessels, controlling vascular integrity and function. During quiescence, ECs regulate junctional integrity and barrier stability. Recently, ZEB1 was hypothesised to contribute to endothelial quiescence. Integrating mass spe...

Full description

Bibliographic Details
Main Author: Ahmed, Nada
Format: Thesis (University of Nottingham only)
Language:English
Published: 2025
Subjects:
Online Access:https://eprints.nottingham.ac.uk/80747/
_version_ 1848801268983660544
author Ahmed, Nada
author_facet Ahmed, Nada
author_sort Ahmed, Nada
building Nottingham Research Data Repository
collection Online Access
description Endothelial cells (ECs) constitute the crucial lining of blood and lymphatic vessels, controlling vascular integrity and function. During quiescence, ECs regulate junctional integrity and barrier stability. Recently, ZEB1 was hypothesised to contribute to endothelial quiescence. Integrating mass spectrometric and transcriptomic analyses in lymphatic ECs uncovered that ZEB1 contributes to junctional reorganization. Silencing ZEB1 significantly reduced VE-cadherin phosphorylation at Y731 and Y685 (59%±5.39, 71%±15,p<0.05, respectively), consequently compromising barrier integrity. Silencing ZEB1 downregulated YES and YAP1 suggesting that altered VE-cadherin signalling was a consequence of ZEB1 mediated-YES/YAP1 signalling. In LECs, silencing YAP1 induced cytoskeletal remodelling, and functionally compromised EC barrier phenocopying ZEB1 loss. Consistently, YAP1 loss showed dephosphorylation of Y685 and Y731.
first_indexed 2025-11-14T21:04:46Z
format Thesis (University of Nottingham only)
id nottingham-80747
institution University of Nottingham Malaysia Campus
institution_category Local University
language English
last_indexed 2025-11-14T21:04:46Z
publishDate 2025
recordtype eprints
repository_type Digital Repository
spelling nottingham-807472025-07-23T04:40:11Z https://eprints.nottingham.ac.uk/80747/ Understanding the ZEB1 interactome during lymphangiogenesis Ahmed, Nada Endothelial cells (ECs) constitute the crucial lining of blood and lymphatic vessels, controlling vascular integrity and function. During quiescence, ECs regulate junctional integrity and barrier stability. Recently, ZEB1 was hypothesised to contribute to endothelial quiescence. Integrating mass spectrometric and transcriptomic analyses in lymphatic ECs uncovered that ZEB1 contributes to junctional reorganization. Silencing ZEB1 significantly reduced VE-cadherin phosphorylation at Y731 and Y685 (59%±5.39, 71%±15,p<0.05, respectively), consequently compromising barrier integrity. Silencing ZEB1 downregulated YES and YAP1 suggesting that altered VE-cadherin signalling was a consequence of ZEB1 mediated-YES/YAP1 signalling. In LECs, silencing YAP1 induced cytoskeletal remodelling, and functionally compromised EC barrier phenocopying ZEB1 loss. Consistently, YAP1 loss showed dephosphorylation of Y685 and Y731. 2025-07-23 Thesis (University of Nottingham only) NonPeerReviewed application/pdf en cc_by https://eprints.nottingham.ac.uk/80747/1/AHMED%2CNADA%2C20217798%2CFinal%20Thesis.pdf Ahmed, Nada (2025) Understanding the ZEB1 interactome during lymphangiogenesis. PhD thesis, University of Nottingham. Lymphangiogenesis ZEB1 Vascular biology Interactome Junctional integrity
spellingShingle Lymphangiogenesis
ZEB1
Vascular biology
Interactome
Junctional integrity
Ahmed, Nada
Understanding the ZEB1 interactome during lymphangiogenesis
title Understanding the ZEB1 interactome during lymphangiogenesis
title_full Understanding the ZEB1 interactome during lymphangiogenesis
title_fullStr Understanding the ZEB1 interactome during lymphangiogenesis
title_full_unstemmed Understanding the ZEB1 interactome during lymphangiogenesis
title_short Understanding the ZEB1 interactome during lymphangiogenesis
title_sort understanding the zeb1 interactome during lymphangiogenesis
topic Lymphangiogenesis
ZEB1
Vascular biology
Interactome
Junctional integrity
url https://eprints.nottingham.ac.uk/80747/