Methionine-aromatic interactions in ABCG2 influence mitoxantrone efflux

The ATP-Binding Cassette (ABC) transporters are one of many families of membrane transport proteins that utilise the hydrolysis of ATP to provide the energy required to actively transport substrates against a concentration gradient. Some ABC transporters have been implicated in cancer multidrug resi...

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Main Author: Azmir, Nur Amirah Hannan
Format: Thesis (University of Nottingham only)
Language:English
Published: 2020
Subjects:
Online Access:https://eprints.nottingham.ac.uk/63776/
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author Azmir, Nur Amirah Hannan
author_facet Azmir, Nur Amirah Hannan
author_sort Azmir, Nur Amirah Hannan
building Nottingham Research Data Repository
collection Online Access
description The ATP-Binding Cassette (ABC) transporters are one of many families of membrane transport proteins that utilise the hydrolysis of ATP to provide the energy required to actively transport substrates against a concentration gradient. Some ABC transporters have been implicated in cancer multidrug resistance (MDR), where some solid tumours and blood cancers develop reduced chemosensitivity to the antineoplastic agents used to treat them due to these MDR-type ABC transporters having a very diverse substrate specificity incorporating different chemistries. One of these ABC transporters implicated in human cancer MDR is ABCG2. The mechanism behind the ability for ABCG2 to recognise a wide variety of structurally dissimilar substrates is a key question that has yet to be fully answered, with many studies trying to identify both the location of drug binding sites and the translocation pathway for allocrite recognition and efflux. In this study, an attempt was made to understand the role of a predicted drug-binding site on the surface of ABCG2. This site is rich in methionine- aromatic interactions and it was predicted that disruption of these interac- tions would influence drug transport. Mutagenesis of 12 residues (6 methi- onines and 6 phenylalanines) was performed and mutants successfully ex- pressed and trafficked to the plasma membrane. In drug transport assays using the native substrate mitoxantrone, 3 of the 12 mutants showed a significant decrease in drug efflux compared to the wild type From this work it was predicted that the pair of residues M541 and F545 are engaged in a methionine-aromatic interaction that is critical for drug recognition and efflux. Further future studies such as transport assays with another native substrate and a non-native substrate can be performed to further validate this claim.
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format Thesis (University of Nottingham only)
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institution University of Nottingham Malaysia Campus
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language English
last_indexed 2025-11-14T20:45:29Z
publishDate 2020
recordtype eprints
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spelling nottingham-637762025-02-28T15:07:00Z https://eprints.nottingham.ac.uk/63776/ Methionine-aromatic interactions in ABCG2 influence mitoxantrone efflux Azmir, Nur Amirah Hannan The ATP-Binding Cassette (ABC) transporters are one of many families of membrane transport proteins that utilise the hydrolysis of ATP to provide the energy required to actively transport substrates against a concentration gradient. Some ABC transporters have been implicated in cancer multidrug resistance (MDR), where some solid tumours and blood cancers develop reduced chemosensitivity to the antineoplastic agents used to treat them due to these MDR-type ABC transporters having a very diverse substrate specificity incorporating different chemistries. One of these ABC transporters implicated in human cancer MDR is ABCG2. The mechanism behind the ability for ABCG2 to recognise a wide variety of structurally dissimilar substrates is a key question that has yet to be fully answered, with many studies trying to identify both the location of drug binding sites and the translocation pathway for allocrite recognition and efflux. In this study, an attempt was made to understand the role of a predicted drug-binding site on the surface of ABCG2. This site is rich in methionine- aromatic interactions and it was predicted that disruption of these interac- tions would influence drug transport. Mutagenesis of 12 residues (6 methi- onines and 6 phenylalanines) was performed and mutants successfully ex- pressed and trafficked to the plasma membrane. In drug transport assays using the native substrate mitoxantrone, 3 of the 12 mutants showed a significant decrease in drug efflux compared to the wild type From this work it was predicted that the pair of residues M541 and F545 are engaged in a methionine-aromatic interaction that is critical for drug recognition and efflux. Further future studies such as transport assays with another native substrate and a non-native substrate can be performed to further validate this claim. 2020-12-11 Thesis (University of Nottingham only) NonPeerReviewed application/pdf en arr https://eprints.nottingham.ac.uk/63776/1/NAH%20Binti%20Azmir%20%20-%2018023418.pdf Azmir, Nur Amirah Hannan (2020) Methionine-aromatic interactions in ABCG2 influence mitoxantrone efflux. MRes thesis, University of Nottingham. ATP-binding cassette transporters; Membrane proteins; Multidrug resistance; Mitoxantrone efflux
spellingShingle ATP-binding cassette transporters; Membrane proteins; Multidrug resistance; Mitoxantrone efflux
Azmir, Nur Amirah Hannan
Methionine-aromatic interactions in ABCG2 influence mitoxantrone efflux
title Methionine-aromatic interactions in ABCG2 influence mitoxantrone efflux
title_full Methionine-aromatic interactions in ABCG2 influence mitoxantrone efflux
title_fullStr Methionine-aromatic interactions in ABCG2 influence mitoxantrone efflux
title_full_unstemmed Methionine-aromatic interactions in ABCG2 influence mitoxantrone efflux
title_short Methionine-aromatic interactions in ABCG2 influence mitoxantrone efflux
title_sort methionine-aromatic interactions in abcg2 influence mitoxantrone efflux
topic ATP-binding cassette transporters; Membrane proteins; Multidrug resistance; Mitoxantrone efflux
url https://eprints.nottingham.ac.uk/63776/