Transcriptomic gene signatures associated with persistent airflow limitation in patients with severe asthma

A proportion of severe asthma patients suffers from persistent airflow limitation (PAL), often associated with more symptoms and exacerbations. Little is known about the underlying mechanisms. Here, our aim was to discover unexplored potential mechanisms using Gene Set Variation Analysis (GSVA), a s...

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Main Authors: Hekking, Pieter-Paul, Loza, Matthew J., Pavlidis, Stelios, De Meulder, Bertrand, Lefaudeux, Diane, Baribaud, Frederic, Auffray, Charles, Wagener, Ariane H., Brinkman, Paul, Lutter, René, Bansal, Aruna T., Sousa, Ana R., Bates, Stewart A., Pandis, Ioannis, Fleming, Louise J., Shaw, Dominick E., Fowler, Stephen J., Guo, Yike, Meiser, Andrea, Sun, Kai, Corfield, Julie, Howarth, Peter, Bel, Elisabeth H., Adcock, Ian M., Chung, Kian Fan, Djukanovic, Ratko, Sterk, Peter J.
Format: Article
Published: European Respiratory Society 2017
Online Access:https://eprints.nottingham.ac.uk/51852/
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author Hekking, Pieter-Paul
Loza, Matthew J.
Pavlidis, Stelios
De Meulder, Bertrand
Lefaudeux, Diane
Baribaud, Frederic
Auffray, Charles
Wagener, Ariane H.
Brinkman, Paul
Lutter, René
Bansal, Aruna T.
Sousa, Ana R.
Bates, Stewart A.
Pandis, Ioannis
Fleming, Louise J.
Shaw, Dominick E.
Fowler, Stephen J.
Guo, Yike
Meiser, Andrea
Sun, Kai
Corfield, Julie
Howarth, Peter
Bel, Elisabeth H.
Adcock, Ian M.
Chung, Kian Fan
Djukanovic, Ratko
Sterk, Peter J.
author_facet Hekking, Pieter-Paul
Loza, Matthew J.
Pavlidis, Stelios
De Meulder, Bertrand
Lefaudeux, Diane
Baribaud, Frederic
Auffray, Charles
Wagener, Ariane H.
Brinkman, Paul
Lutter, René
Bansal, Aruna T.
Sousa, Ana R.
Bates, Stewart A.
Pandis, Ioannis
Fleming, Louise J.
Shaw, Dominick E.
Fowler, Stephen J.
Guo, Yike
Meiser, Andrea
Sun, Kai
Corfield, Julie
Howarth, Peter
Bel, Elisabeth H.
Adcock, Ian M.
Chung, Kian Fan
Djukanovic, Ratko
Sterk, Peter J.
author_sort Hekking, Pieter-Paul
building Nottingham Research Data Repository
collection Online Access
description A proportion of severe asthma patients suffers from persistent airflow limitation (PAL), often associated with more symptoms and exacerbations. Little is known about the underlying mechanisms. Here, our aim was to discover unexplored potential mechanisms using Gene Set Variation Analysis (GSVA), a sensitive technique that can detect underlying pathways in heterogeneous samples. Severe asthma patients from the U-BIOPRED cohort with PAL (post-bronchodilator forced expiratory volume in 1 s/forced vital capacity ratio below the lower limit of normal) were compared with those without PAL. Gene expression was assessed on the total RNA of sputum cells, nasal brushings, and endobronchial brushings and biopsies. GSVA was applied to identify differentially enriched predefined gene signatures based on all available gene expression publications and data on airways disease. Differentially enriched gene signatures were identified in nasal brushings (n=1), sputum (n=9), bronchial brushings (n=1) and bronchial biopsies (n=4) that were associated with response to inhaled steroids, eosinophils, interleukin-13, interferon-α, specific CD4+ T-cells and airway remodelling. PAL in severe asthma has distinguishable underlying gene networks that are associated with treatment, inflammatory pathways and airway remodelling. These findings point towards targets for the therapy of PAL in severe asthma.
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spelling nottingham-518522020-05-04T19:08:40Z https://eprints.nottingham.ac.uk/51852/ Transcriptomic gene signatures associated with persistent airflow limitation in patients with severe asthma Hekking, Pieter-Paul Loza, Matthew J. Pavlidis, Stelios De Meulder, Bertrand Lefaudeux, Diane Baribaud, Frederic Auffray, Charles Wagener, Ariane H. Brinkman, Paul Lutter, René Bansal, Aruna T. Sousa, Ana R. Bates, Stewart A. Pandis, Ioannis Fleming, Louise J. Shaw, Dominick E. Fowler, Stephen J. Guo, Yike Meiser, Andrea Sun, Kai Corfield, Julie Howarth, Peter Bel, Elisabeth H. Adcock, Ian M. Chung, Kian Fan Djukanovic, Ratko Sterk, Peter J. A proportion of severe asthma patients suffers from persistent airflow limitation (PAL), often associated with more symptoms and exacerbations. Little is known about the underlying mechanisms. Here, our aim was to discover unexplored potential mechanisms using Gene Set Variation Analysis (GSVA), a sensitive technique that can detect underlying pathways in heterogeneous samples. Severe asthma patients from the U-BIOPRED cohort with PAL (post-bronchodilator forced expiratory volume in 1 s/forced vital capacity ratio below the lower limit of normal) were compared with those without PAL. Gene expression was assessed on the total RNA of sputum cells, nasal brushings, and endobronchial brushings and biopsies. GSVA was applied to identify differentially enriched predefined gene signatures based on all available gene expression publications and data on airways disease. Differentially enriched gene signatures were identified in nasal brushings (n=1), sputum (n=9), bronchial brushings (n=1) and bronchial biopsies (n=4) that were associated with response to inhaled steroids, eosinophils, interleukin-13, interferon-α, specific CD4+ T-cells and airway remodelling. PAL in severe asthma has distinguishable underlying gene networks that are associated with treatment, inflammatory pathways and airway remodelling. These findings point towards targets for the therapy of PAL in severe asthma. European Respiratory Society 2017-09-27 Article PeerReviewed Hekking, Pieter-Paul, Loza, Matthew J., Pavlidis, Stelios, De Meulder, Bertrand, Lefaudeux, Diane, Baribaud, Frederic, Auffray, Charles, Wagener, Ariane H., Brinkman, Paul, Lutter, René, Bansal, Aruna T., Sousa, Ana R., Bates, Stewart A., Pandis, Ioannis, Fleming, Louise J., Shaw, Dominick E., Fowler, Stephen J., Guo, Yike, Meiser, Andrea, Sun, Kai, Corfield, Julie, Howarth, Peter, Bel, Elisabeth H., Adcock, Ian M., Chung, Kian Fan, Djukanovic, Ratko and Sterk, Peter J. (2017) Transcriptomic gene signatures associated with persistent airflow limitation in patients with severe asthma. European Respiratory Journal, 50 (3). p. 1602298. ISSN 1399-3003 https://doi.org/10.1183/13993003.02298-2016 doi:10.1183/13993003.02298-2016 doi:10.1183/13993003.02298-2016
spellingShingle Hekking, Pieter-Paul
Loza, Matthew J.
Pavlidis, Stelios
De Meulder, Bertrand
Lefaudeux, Diane
Baribaud, Frederic
Auffray, Charles
Wagener, Ariane H.
Brinkman, Paul
Lutter, René
Bansal, Aruna T.
Sousa, Ana R.
Bates, Stewart A.
Pandis, Ioannis
Fleming, Louise J.
Shaw, Dominick E.
Fowler, Stephen J.
Guo, Yike
Meiser, Andrea
Sun, Kai
Corfield, Julie
Howarth, Peter
Bel, Elisabeth H.
Adcock, Ian M.
Chung, Kian Fan
Djukanovic, Ratko
Sterk, Peter J.
Transcriptomic gene signatures associated with persistent airflow limitation in patients with severe asthma
title Transcriptomic gene signatures associated with persistent airflow limitation in patients with severe asthma
title_full Transcriptomic gene signatures associated with persistent airflow limitation in patients with severe asthma
title_fullStr Transcriptomic gene signatures associated with persistent airflow limitation in patients with severe asthma
title_full_unstemmed Transcriptomic gene signatures associated with persistent airflow limitation in patients with severe asthma
title_short Transcriptomic gene signatures associated with persistent airflow limitation in patients with severe asthma
title_sort transcriptomic gene signatures associated with persistent airflow limitation in patients with severe asthma
url https://eprints.nottingham.ac.uk/51852/
https://eprints.nottingham.ac.uk/51852/
https://eprints.nottingham.ac.uk/51852/