Cold atmospheric plasma induces ATP-dependent endocytosis of nanoparticles and synergistic U373MG cancer cell death

Gold nanoparticles (AuNP) have potential as both diagnostic and therapeutic vehicles. However, selective targeting and uptake in cancer cells remains challenging. Cold atmospheric plasma (CAP) can be combined with AuNP to achieve synergistic anti-cancer cytotoxicity. To explore synergistic mechanism...

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Main Authors: He, Zhonglei, Liu, Kangze, Manaloto, Eline, Casey, Alan, Cribaro, George P., Byrne, Hugh J., Tian, Furong, Barcia, Carlos, Conway, Gillian E., Cullen, P.J., Curtin, James F.
Format: Article
Language:English
Published: Nature Publishing Group 2018
Online Access:https://eprints.nottingham.ac.uk/50993/
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author He, Zhonglei
Liu, Kangze
Manaloto, Eline
Casey, Alan
Cribaro, George P.
Byrne, Hugh J.
Tian, Furong
Barcia, Carlos
Conway, Gillian E.
Cullen, P.J.
Curtin, James F.
author_facet He, Zhonglei
Liu, Kangze
Manaloto, Eline
Casey, Alan
Cribaro, George P.
Byrne, Hugh J.
Tian, Furong
Barcia, Carlos
Conway, Gillian E.
Cullen, P.J.
Curtin, James F.
author_sort He, Zhonglei
building Nottingham Research Data Repository
collection Online Access
description Gold nanoparticles (AuNP) have potential as both diagnostic and therapeutic vehicles. However, selective targeting and uptake in cancer cells remains challenging. Cold atmospheric plasma (CAP) can be combined with AuNP to achieve synergistic anti-cancer cytotoxicity. To explore synergistic mechanisms, we demonstrate both rate of AuNP uptake and total amount accumulated in U373MG Glioblastoma multiforme (GBM) cells are significantly increased when exposed to 75 kV CAP generated by dielectric barrier discharge. No significant changes in the physical parameters of AuNP were caused by CAP but active transport mechanisms were stimulated in cells. Unlike many other biological effects of CAP, long-lived reactive species were not involved, and plasma-activated liquids did not replicate the effect. Chemical effects induced by direct and indirect exposure to CAP appears the dominant mediator of enhanced uptake. Transient physical alterations of membrane integrity played a minor role. 3D-reconstruction of deconvoluted confocal images confirmed AuNP accumulation in lysosomes and other acidic vesicles, which will be useful for future drug delivery and diagnostic strategies. Toxicity of AuNP significantly increased by 25-fold when combined with CAP. Our data indicate that direct exposure to CAP activates AuNP-dependent cytotoxicity by increasing AuNP endocytosis and trafficking to lysosomes in U373MG cells.
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spelling nottingham-509932018-04-09T13:23:21Z https://eprints.nottingham.ac.uk/50993/ Cold atmospheric plasma induces ATP-dependent endocytosis of nanoparticles and synergistic U373MG cancer cell death He, Zhonglei Liu, Kangze Manaloto, Eline Casey, Alan Cribaro, George P. Byrne, Hugh J. Tian, Furong Barcia, Carlos Conway, Gillian E. Cullen, P.J. Curtin, James F. Gold nanoparticles (AuNP) have potential as both diagnostic and therapeutic vehicles. However, selective targeting and uptake in cancer cells remains challenging. Cold atmospheric plasma (CAP) can be combined with AuNP to achieve synergistic anti-cancer cytotoxicity. To explore synergistic mechanisms, we demonstrate both rate of AuNP uptake and total amount accumulated in U373MG Glioblastoma multiforme (GBM) cells are significantly increased when exposed to 75 kV CAP generated by dielectric barrier discharge. No significant changes in the physical parameters of AuNP were caused by CAP but active transport mechanisms were stimulated in cells. Unlike many other biological effects of CAP, long-lived reactive species were not involved, and plasma-activated liquids did not replicate the effect. Chemical effects induced by direct and indirect exposure to CAP appears the dominant mediator of enhanced uptake. Transient physical alterations of membrane integrity played a minor role. 3D-reconstruction of deconvoluted confocal images confirmed AuNP accumulation in lysosomes and other acidic vesicles, which will be useful for future drug delivery and diagnostic strategies. Toxicity of AuNP significantly increased by 25-fold when combined with CAP. Our data indicate that direct exposure to CAP activates AuNP-dependent cytotoxicity by increasing AuNP endocytosis and trafficking to lysosomes in U373MG cells. Nature Publishing Group 2018-03-28 Article PeerReviewed application/pdf en cc_by https://eprints.nottingham.ac.uk/50993/8/Plasma%20s41598-018-23262-0.pdf He, Zhonglei, Liu, Kangze, Manaloto, Eline, Casey, Alan, Cribaro, George P., Byrne, Hugh J., Tian, Furong, Barcia, Carlos, Conway, Gillian E., Cullen, P.J. and Curtin, James F. (2018) Cold atmospheric plasma induces ATP-dependent endocytosis of nanoparticles and synergistic U373MG cancer cell death. Scientific Reports, 8 (5298). 5298/1-5298/11. ISSN 2045-2322 https://www.nature.com/articles/s41598-018-23262-0 doi:10.1038/s41598-018-23262-0 doi:10.1038/s41598-018-23262-0
spellingShingle He, Zhonglei
Liu, Kangze
Manaloto, Eline
Casey, Alan
Cribaro, George P.
Byrne, Hugh J.
Tian, Furong
Barcia, Carlos
Conway, Gillian E.
Cullen, P.J.
Curtin, James F.
Cold atmospheric plasma induces ATP-dependent endocytosis of nanoparticles and synergistic U373MG cancer cell death
title Cold atmospheric plasma induces ATP-dependent endocytosis of nanoparticles and synergistic U373MG cancer cell death
title_full Cold atmospheric plasma induces ATP-dependent endocytosis of nanoparticles and synergistic U373MG cancer cell death
title_fullStr Cold atmospheric plasma induces ATP-dependent endocytosis of nanoparticles and synergistic U373MG cancer cell death
title_full_unstemmed Cold atmospheric plasma induces ATP-dependent endocytosis of nanoparticles and synergistic U373MG cancer cell death
title_short Cold atmospheric plasma induces ATP-dependent endocytosis of nanoparticles and synergistic U373MG cancer cell death
title_sort cold atmospheric plasma induces atp-dependent endocytosis of nanoparticles and synergistic u373mg cancer cell death
url https://eprints.nottingham.ac.uk/50993/
https://eprints.nottingham.ac.uk/50993/
https://eprints.nottingham.ac.uk/50993/