Peptide hydrogels — a tissue engineering strategy for the prevention of oesophageal strictures

Endoscopic treatment of Barrett’s oesophagus often leads to further damage of healthy tissue causing fibrotic tissue formation termed as strictures. This study shows that synthetic, self-assembling peptide hydrogels (PeptiGelDesign) support the activity and function of primary oesophageal cells, lea...

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Main Authors: Kumar, Deepak, Workman, Victoria, O'Brien, Marie Claire, McLaren, Jane S., White, Lisa J., Ragunath, Krish, Saiani, Alberto, Gough, Julie, Rose, Felicity R.A.J.
Format: Article
Published: Wiley 2017
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Online Access:https://eprints.nottingham.ac.uk/45558/
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author Kumar, Deepak
Workman, Victoria
O'Brien, Marie Claire
McLaren, Jane S.
White, Lisa J.
Ragunath, Krish
Saiani, Alberto
Gough, Julie
Rose, Felicity R.A.J.
author_facet Kumar, Deepak
Workman, Victoria
O'Brien, Marie Claire
McLaren, Jane S.
White, Lisa J.
Ragunath, Krish
Saiani, Alberto
Gough, Julie
Rose, Felicity R.A.J.
author_sort Kumar, Deepak
building Nottingham Research Data Repository
collection Online Access
description Endoscopic treatment of Barrett’s oesophagus often leads to further damage of healthy tissue causing fibrotic tissue formation termed as strictures. This study shows that synthetic, self-assembling peptide hydrogels (PeptiGelDesign) support the activity and function of primary oesophageal cells, leading to epithelialisation and stratification during in vitro 3D co-culture. Following buffering in culture media, oesophageal stromal fibroblasts (rOSFs) were incorporated into a library of peptide hydrogels, whereas oesophageal epithelial cells (mOECs) were seeded on the surface. Optimal hydrogels (PGD-AlphaProC and PGD-CGD2) supported mOEC viability (>95 %), typical cell morphology (cobblestone-like), a migration rate of 17.4 μm/hr and a migration distance of 364 μm, at 48 hours. Positive expression of typical epithelial markers (ZO-1 and cytokeratins) was witnessed detected using immunocytochemistry at day 3 in culture. Furthermore, optimal hydrogels were identified which supported rOSF viability (> 95%) with homogenous distribution when incorporated into the hydrogels and also promoted the secretion of collagen type I detected using ELISA, at day 7. 3D co-culture model using optimal hydrogels for both cell types supported a stratified epithelial layer (expressing involucrin and AE1/AE3 markers). Findings from this study could lead to the use of peptide hydrogels as a minimally invasive endoscopic therapy to manage oesophageal strictures.
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spelling nottingham-455582020-05-04T19:12:26Z https://eprints.nottingham.ac.uk/45558/ Peptide hydrogels — a tissue engineering strategy for the prevention of oesophageal strictures Kumar, Deepak Workman, Victoria O'Brien, Marie Claire McLaren, Jane S. White, Lisa J. Ragunath, Krish Saiani, Alberto Gough, Julie Rose, Felicity R.A.J. Endoscopic treatment of Barrett’s oesophagus often leads to further damage of healthy tissue causing fibrotic tissue formation termed as strictures. This study shows that synthetic, self-assembling peptide hydrogels (PeptiGelDesign) support the activity and function of primary oesophageal cells, leading to epithelialisation and stratification during in vitro 3D co-culture. Following buffering in culture media, oesophageal stromal fibroblasts (rOSFs) were incorporated into a library of peptide hydrogels, whereas oesophageal epithelial cells (mOECs) were seeded on the surface. Optimal hydrogels (PGD-AlphaProC and PGD-CGD2) supported mOEC viability (>95 %), typical cell morphology (cobblestone-like), a migration rate of 17.4 μm/hr and a migration distance of 364 μm, at 48 hours. Positive expression of typical epithelial markers (ZO-1 and cytokeratins) was witnessed detected using immunocytochemistry at day 3 in culture. Furthermore, optimal hydrogels were identified which supported rOSF viability (> 95%) with homogenous distribution when incorporated into the hydrogels and also promoted the secretion of collagen type I detected using ELISA, at day 7. 3D co-culture model using optimal hydrogels for both cell types supported a stratified epithelial layer (expressing involucrin and AE1/AE3 markers). Findings from this study could lead to the use of peptide hydrogels as a minimally invasive endoscopic therapy to manage oesophageal strictures. Wiley 2017-10-12 Article PeerReviewed Kumar, Deepak, Workman, Victoria, O'Brien, Marie Claire, McLaren, Jane S., White, Lisa J., Ragunath, Krish, Saiani, Alberto, Gough, Julie and Rose, Felicity R.A.J. (2017) Peptide hydrogels — a tissue engineering strategy for the prevention of oesophageal strictures. Advanced Functional Materials, 27 (38). 1702424/1-1702424/12. ISSN 1616-3028 Barrett's oesophagus; co-culture model; stiffness; synthetic peptide hydrogels http://onlinelibrary.wiley.com/doi/10.1002/adfm.201702424/full doi:10.1002/adfm.201702424 doi:10.1002/adfm.201702424
spellingShingle Barrett's oesophagus; co-culture model; stiffness; synthetic peptide hydrogels
Kumar, Deepak
Workman, Victoria
O'Brien, Marie Claire
McLaren, Jane S.
White, Lisa J.
Ragunath, Krish
Saiani, Alberto
Gough, Julie
Rose, Felicity R.A.J.
Peptide hydrogels — a tissue engineering strategy for the prevention of oesophageal strictures
title Peptide hydrogels — a tissue engineering strategy for the prevention of oesophageal strictures
title_full Peptide hydrogels — a tissue engineering strategy for the prevention of oesophageal strictures
title_fullStr Peptide hydrogels — a tissue engineering strategy for the prevention of oesophageal strictures
title_full_unstemmed Peptide hydrogels — a tissue engineering strategy for the prevention of oesophageal strictures
title_short Peptide hydrogels — a tissue engineering strategy for the prevention of oesophageal strictures
title_sort peptide hydrogels — a tissue engineering strategy for the prevention of oesophageal strictures
topic Barrett's oesophagus; co-culture model; stiffness; synthetic peptide hydrogels
url https://eprints.nottingham.ac.uk/45558/
https://eprints.nottingham.ac.uk/45558/
https://eprints.nottingham.ac.uk/45558/