Phenotypic and pharmacogenetic evaluation of patients with thiazide-induced hyponatremia

Thiazide diuretics are among the most widely used treatments for hypertension, but thiazide-induced hyponatremia (TIH), a clinically significant adverse effect, is poorly understood. Here, we have studied the phenotypic and genetic characteristics of patients hospitalized with TIH. In a cohort of 10...

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Main Authors: Ware, James S., Wain, Louise V., Channavajjhala, Sarath K., Jackson, Victoria E., Edwards, Elizabeth, Lu, Run, Siew, Keith, Jia, Wenjing, Shrine, Nick, Kinnear, Sue, Jalland, Mahli, Henry, Amanda P., Clayton, Jenny, O’Shaughnessy, Kevin M., Tobin, Martin D., Schuster, Victor, Cook, Stuart, Hall, Ian P., Glover, Mark
Format: Article
Published: American Society for Clinical Investigation 2017
Online Access:https://eprints.nottingham.ac.uk/44729/
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author Ware, James S.
Wain, Louise V.
Channavajjhala, Sarath K.
Jackson, Victoria E.
Edwards, Elizabeth
Lu, Run
Siew, Keith
Jia, Wenjing
Shrine, Nick
Kinnear, Sue
Jalland, Mahli
Henry, Amanda P.
Clayton, Jenny
O’Shaughnessy, Kevin M.
Tobin, Martin D.
Schuster, Victor
Cook, Stuart
Hall, Ian P.
Glover, Mark
author_facet Ware, James S.
Wain, Louise V.
Channavajjhala, Sarath K.
Jackson, Victoria E.
Edwards, Elizabeth
Lu, Run
Siew, Keith
Jia, Wenjing
Shrine, Nick
Kinnear, Sue
Jalland, Mahli
Henry, Amanda P.
Clayton, Jenny
O’Shaughnessy, Kevin M.
Tobin, Martin D.
Schuster, Victor
Cook, Stuart
Hall, Ian P.
Glover, Mark
author_sort Ware, James S.
building Nottingham Research Data Repository
collection Online Access
description Thiazide diuretics are among the most widely used treatments for hypertension, but thiazide-induced hyponatremia (TIH), a clinically significant adverse effect, is poorly understood. Here, we have studied the phenotypic and genetic characteristics of patients hospitalized with TIH. In a cohort of 109 TIH patients, those with severe TIH displayed an extended phenotype of intravascular volume expansion, increased free water reabsorption, urinary prostaglandin E2 excretion, and reduced excretion of serum chloride, magnesium, zinc, and antidiuretic hormone. GWAS in a separate cohort of 48 TIH patients and 2,922 controls from the 1958 British birth cohort identified an additional 14 regions associated with TIH. We identified a suggestive association with a variant in SLCO2A1, which encodes a prostaglandin transporter in the distal nephron. Resequencing of SLCO2A1 revealed a nonsynonymous variant, rs34550074 (p.A396T), and association with this SNP was replicated in a second cohort of TIH cases. TIH patients with the p.A396T variant demonstrated increased urinary excretion of prostaglandin E2 and metabolites. Moreover, the SLCO2A1 phospho-mimic p.A396E showed loss of transporter function in vitro. These findings indicate that the phenotype of TIH involves a more extensive metabolic derangement than previously recognized. We propose one mechanism underlying TIH development in a subgroup of patients in which SLCO2A1 regulation is altered.
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spelling nottingham-447292020-05-04T18:59:46Z https://eprints.nottingham.ac.uk/44729/ Phenotypic and pharmacogenetic evaluation of patients with thiazide-induced hyponatremia Ware, James S. Wain, Louise V. Channavajjhala, Sarath K. Jackson, Victoria E. Edwards, Elizabeth Lu, Run Siew, Keith Jia, Wenjing Shrine, Nick Kinnear, Sue Jalland, Mahli Henry, Amanda P. Clayton, Jenny O’Shaughnessy, Kevin M. Tobin, Martin D. Schuster, Victor Cook, Stuart Hall, Ian P. Glover, Mark Thiazide diuretics are among the most widely used treatments for hypertension, but thiazide-induced hyponatremia (TIH), a clinically significant adverse effect, is poorly understood. Here, we have studied the phenotypic and genetic characteristics of patients hospitalized with TIH. In a cohort of 109 TIH patients, those with severe TIH displayed an extended phenotype of intravascular volume expansion, increased free water reabsorption, urinary prostaglandin E2 excretion, and reduced excretion of serum chloride, magnesium, zinc, and antidiuretic hormone. GWAS in a separate cohort of 48 TIH patients and 2,922 controls from the 1958 British birth cohort identified an additional 14 regions associated with TIH. We identified a suggestive association with a variant in SLCO2A1, which encodes a prostaglandin transporter in the distal nephron. Resequencing of SLCO2A1 revealed a nonsynonymous variant, rs34550074 (p.A396T), and association with this SNP was replicated in a second cohort of TIH cases. TIH patients with the p.A396T variant demonstrated increased urinary excretion of prostaglandin E2 and metabolites. Moreover, the SLCO2A1 phospho-mimic p.A396E showed loss of transporter function in vitro. These findings indicate that the phenotype of TIH involves a more extensive metabolic derangement than previously recognized. We propose one mechanism underlying TIH development in a subgroup of patients in which SLCO2A1 regulation is altered. American Society for Clinical Investigation 2017-08-07 Article PeerReviewed Ware, James S., Wain, Louise V., Channavajjhala, Sarath K., Jackson, Victoria E., Edwards, Elizabeth, Lu, Run, Siew, Keith, Jia, Wenjing, Shrine, Nick, Kinnear, Sue, Jalland, Mahli, Henry, Amanda P., Clayton, Jenny, O’Shaughnessy, Kevin M., Tobin, Martin D., Schuster, Victor, Cook, Stuart, Hall, Ian P. and Glover, Mark (2017) Phenotypic and pharmacogenetic evaluation of patients with thiazide-induced hyponatremia. Journal of Clinical Investigation, 127 (9). pp. 3367-3374. ISSN 1558-8238 https://www.jci.org/articles/view/89812 doi:10.1172/JCI89812 doi:10.1172/JCI89812
spellingShingle Ware, James S.
Wain, Louise V.
Channavajjhala, Sarath K.
Jackson, Victoria E.
Edwards, Elizabeth
Lu, Run
Siew, Keith
Jia, Wenjing
Shrine, Nick
Kinnear, Sue
Jalland, Mahli
Henry, Amanda P.
Clayton, Jenny
O’Shaughnessy, Kevin M.
Tobin, Martin D.
Schuster, Victor
Cook, Stuart
Hall, Ian P.
Glover, Mark
Phenotypic and pharmacogenetic evaluation of patients with thiazide-induced hyponatremia
title Phenotypic and pharmacogenetic evaluation of patients with thiazide-induced hyponatremia
title_full Phenotypic and pharmacogenetic evaluation of patients with thiazide-induced hyponatremia
title_fullStr Phenotypic and pharmacogenetic evaluation of patients with thiazide-induced hyponatremia
title_full_unstemmed Phenotypic and pharmacogenetic evaluation of patients with thiazide-induced hyponatremia
title_short Phenotypic and pharmacogenetic evaluation of patients with thiazide-induced hyponatremia
title_sort phenotypic and pharmacogenetic evaluation of patients with thiazide-induced hyponatremia
url https://eprints.nottingham.ac.uk/44729/
https://eprints.nottingham.ac.uk/44729/
https://eprints.nottingham.ac.uk/44729/