A rapid IL-17 response to Cryptosporidium parvum in the bovine intestine

Cryptosporidium parvum causes diarrhoea, due to villi damage, in livestock and humans globally. Immunity develops after repeated infections but initial infections can be severe, highlighting the importance of early infection dynamics. We have modelled early C. parvum infection in bovine jejunum biop...

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Main Authors: Drinkall, Emma, Wass, Matthew J., Coffey, Tracey J., Flynn, Robin J.
Format: Article
Published: Elsevier 2017
Subjects:
Online Access:https://eprints.nottingham.ac.uk/44427/
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author Drinkall, Emma
Wass, Matthew J.
Coffey, Tracey J.
Flynn, Robin J.
author_facet Drinkall, Emma
Wass, Matthew J.
Coffey, Tracey J.
Flynn, Robin J.
author_sort Drinkall, Emma
building Nottingham Research Data Repository
collection Online Access
description Cryptosporidium parvum causes diarrhoea, due to villi damage, in livestock and humans globally. Immunity develops after repeated infections but initial infections can be severe, highlighting the importance of early infection dynamics. We have modelled early C. parvum infection in bovine jejunum biopsies. IL-17A accumulated over time peaking at 9 h post-infection, with no effect of infection on IL-1β; antibiotics positively influenced IL-17A as higher levels were found in cultures with antibiotics. Infection of primary fibroblasts resulted in lower plaque formation when fibroblasts were primed with IL-17A. Our results indicate a role for IL-17A in reducing C. parvum-dependent host cell damage.
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spelling nottingham-444272020-05-04T18:56:33Z https://eprints.nottingham.ac.uk/44427/ A rapid IL-17 response to Cryptosporidium parvum in the bovine intestine Drinkall, Emma Wass, Matthew J. Coffey, Tracey J. Flynn, Robin J. Cryptosporidium parvum causes diarrhoea, due to villi damage, in livestock and humans globally. Immunity develops after repeated infections but initial infections can be severe, highlighting the importance of early infection dynamics. We have modelled early C. parvum infection in bovine jejunum biopsies. IL-17A accumulated over time peaking at 9 h post-infection, with no effect of infection on IL-1β; antibiotics positively influenced IL-17A as higher levels were found in cultures with antibiotics. Infection of primary fibroblasts resulted in lower plaque formation when fibroblasts were primed with IL-17A. Our results indicate a role for IL-17A in reducing C. parvum-dependent host cell damage. Elsevier 2017-07-23 Article PeerReviewed Drinkall, Emma, Wass, Matthew J., Coffey, Tracey J. and Flynn, Robin J. (2017) A rapid IL-17 response to Cryptosporidium parvum in the bovine intestine. Veterinary Immunology and Immunopathology, 191 . pp. 1-4. ISSN 1873-2534 Cryptosporidium parvum; Biopsy; IL-17A; Intestine; Villi; Bovine http://www.sciencedirect.com/science/article/pii/S0165242717302945 doi:10.1016/j.vetimm.2017.07.009 doi:10.1016/j.vetimm.2017.07.009
spellingShingle Cryptosporidium parvum; Biopsy; IL-17A; Intestine; Villi; Bovine
Drinkall, Emma
Wass, Matthew J.
Coffey, Tracey J.
Flynn, Robin J.
A rapid IL-17 response to Cryptosporidium parvum in the bovine intestine
title A rapid IL-17 response to Cryptosporidium parvum in the bovine intestine
title_full A rapid IL-17 response to Cryptosporidium parvum in the bovine intestine
title_fullStr A rapid IL-17 response to Cryptosporidium parvum in the bovine intestine
title_full_unstemmed A rapid IL-17 response to Cryptosporidium parvum in the bovine intestine
title_short A rapid IL-17 response to Cryptosporidium parvum in the bovine intestine
title_sort rapid il-17 response to cryptosporidium parvum in the bovine intestine
topic Cryptosporidium parvum; Biopsy; IL-17A; Intestine; Villi; Bovine
url https://eprints.nottingham.ac.uk/44427/
https://eprints.nottingham.ac.uk/44427/
https://eprints.nottingham.ac.uk/44427/