Biophysical analysis of a lethal laminin alpha-1 mutation reveals altered self-interaction

Laminins are key basement membrane molecules that influence several biological activities and are linked to a number of diseases. They are secreted as heterotrimeric proteins consisting of one α, one β, and one γ chain, followed by their assembly into a polymer-like sheet at the basement membrane. U...

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Main Authors: Patel, Trushar R., Nikodemus, Denise, Besong, Tabot M.D., Reuten, Raphael, Meier, Markus, Harding, Stephen E., Winzor, Donald J., Koch, Manuel, Stetefeld, Jörg
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Published: Elsevier 2016
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Online Access:https://eprints.nottingham.ac.uk/42730/
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author Patel, Trushar R.
Nikodemus, Denise
Besong, Tabot M.D.
Reuten, Raphael
Meier, Markus
Harding, Stephen E.
Winzor, Donald J.
Koch, Manuel
Stetefeld, Jörg
author_facet Patel, Trushar R.
Nikodemus, Denise
Besong, Tabot M.D.
Reuten, Raphael
Meier, Markus
Harding, Stephen E.
Winzor, Donald J.
Koch, Manuel
Stetefeld, Jörg
author_sort Patel, Trushar R.
building Nottingham Research Data Repository
collection Online Access
description Laminins are key basement membrane molecules that influence several biological activities and are linked to a number of diseases. They are secreted as heterotrimeric proteins consisting of one α, one β, and one γ chain, followed by their assembly into a polymer-like sheet at the basement membrane. Using sedimentation velocity, dynamic light scattering, and surface plasmon resonance experiments, we studied self-association of three laminin (LM) N-terminal fragments α-1 (hLM α-1 N), α-5 (hLM α-5 N) and β-3 (hLM β-3 N) originating from the short arms of the human laminin αβγ heterotrimer. Corresponding studies of the hLM α-1 N C49S mutant, equivalent to the larval lethal C56S mutant in zebrafish, have shown that this mutation causes enhanced self-association behavior, an observation that provides a plausible explanation for the inability of laminin bearing this mutation to fulfill functional roles in vivo, and hence for the deleterious pathological consequences of the mutation on lens function.
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spelling nottingham-427302020-05-04T20:04:43Z https://eprints.nottingham.ac.uk/42730/ Biophysical analysis of a lethal laminin alpha-1 mutation reveals altered self-interaction Patel, Trushar R. Nikodemus, Denise Besong, Tabot M.D. Reuten, Raphael Meier, Markus Harding, Stephen E. Winzor, Donald J. Koch, Manuel Stetefeld, Jörg Laminins are key basement membrane molecules that influence several biological activities and are linked to a number of diseases. They are secreted as heterotrimeric proteins consisting of one α, one β, and one γ chain, followed by their assembly into a polymer-like sheet at the basement membrane. Using sedimentation velocity, dynamic light scattering, and surface plasmon resonance experiments, we studied self-association of three laminin (LM) N-terminal fragments α-1 (hLM α-1 N), α-5 (hLM α-5 N) and β-3 (hLM β-3 N) originating from the short arms of the human laminin αβγ heterotrimer. Corresponding studies of the hLM α-1 N C49S mutant, equivalent to the larval lethal C56S mutant in zebrafish, have shown that this mutation causes enhanced self-association behavior, an observation that provides a plausible explanation for the inability of laminin bearing this mutation to fulfill functional roles in vivo, and hence for the deleterious pathological consequences of the mutation on lens function. Elsevier 2016-01 Article PeerReviewed Patel, Trushar R., Nikodemus, Denise, Besong, Tabot M.D., Reuten, Raphael, Meier, Markus, Harding, Stephen E., Winzor, Donald J., Koch, Manuel and Stetefeld, Jörg (2016) Biophysical analysis of a lethal laminin alpha-1 mutation reveals altered self-interaction. Matrix Biology, 49 . pp. 93-105. ISSN 1569-1802 Analytical ultracentrifugation; CD spectroscopy; Dynamic light scattering; Extracellular matrix; Laminin short arms; Protein self-association; Surface plasmon resonance http://www.sciencedirect.com/science/article/pii/S0945053X15001237 doi:10.1016/j.matbio.2015.06.005 doi:10.1016/j.matbio.2015.06.005
spellingShingle Analytical ultracentrifugation; CD spectroscopy; Dynamic light scattering; Extracellular matrix; Laminin short arms; Protein self-association; Surface plasmon resonance
Patel, Trushar R.
Nikodemus, Denise
Besong, Tabot M.D.
Reuten, Raphael
Meier, Markus
Harding, Stephen E.
Winzor, Donald J.
Koch, Manuel
Stetefeld, Jörg
Biophysical analysis of a lethal laminin alpha-1 mutation reveals altered self-interaction
title Biophysical analysis of a lethal laminin alpha-1 mutation reveals altered self-interaction
title_full Biophysical analysis of a lethal laminin alpha-1 mutation reveals altered self-interaction
title_fullStr Biophysical analysis of a lethal laminin alpha-1 mutation reveals altered self-interaction
title_full_unstemmed Biophysical analysis of a lethal laminin alpha-1 mutation reveals altered self-interaction
title_short Biophysical analysis of a lethal laminin alpha-1 mutation reveals altered self-interaction
title_sort biophysical analysis of a lethal laminin alpha-1 mutation reveals altered self-interaction
topic Analytical ultracentrifugation; CD spectroscopy; Dynamic light scattering; Extracellular matrix; Laminin short arms; Protein self-association; Surface plasmon resonance
url https://eprints.nottingham.ac.uk/42730/
https://eprints.nottingham.ac.uk/42730/
https://eprints.nottingham.ac.uk/42730/