Global miRNA expression profiling of domestic cat livers following acute Toxoplasma gondii infection

Although microRNAs (miRNAs) play an important role in liver homeostasis, the extent to which they can be altered by Toxoplasma gondii infection is unknown. Here, we utilized small RNA sequencing and bioinformatic analyses to characterize miRNA expression profiles in the liver of domestic cats at 7 d...

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Main Authors: Cong, Wei, Zhang, Xiao-Xuan, He, Jun-Jun, Li, Fa-Cai, Elsheikha, Hany M., Zhu, Xing-Quan
Format: Article
Published: Impact Journals 2017
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Online Access:https://eprints.nottingham.ac.uk/40648/
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author Cong, Wei
Zhang, Xiao-Xuan
He, Jun-Jun
Li, Fa-Cai
Elsheikha, Hany M.
Zhu, Xing-Quan
author_facet Cong, Wei
Zhang, Xiao-Xuan
He, Jun-Jun
Li, Fa-Cai
Elsheikha, Hany M.
Zhu, Xing-Quan
author_sort Cong, Wei
building Nottingham Research Data Repository
collection Online Access
description Although microRNAs (miRNAs) play an important role in liver homeostasis, the extent to which they can be altered by Toxoplasma gondii infection is unknown. Here, we utilized small RNA sequencing and bioinformatic analyses to characterize miRNA expression profiles in the liver of domestic cats at 7 days after oral infection with T. gondii (Type II) strain. A total of 384 miRNAs were identified and 82 were differentially expressed, of which 33 were up-regulated and 49 down-regulated. Also, 5690 predicted host gene targets for the differentially expressed miRNAs were identified using the bioinformatic algorithm miRanda. Gene ontology analysis revealed that the predicted gene targets of the dysregulated miRNAs were significantly enriched in apoptosis. Kyoto Encyclopedia of Genes and Genomes analysis showed that the predicted gene targets were involved in several pathways, including acute myeloid leukemia, central carbon metabolism in cancer, choline metabolism in cancer, estrogen signaling pathway, fatty acid degradation, lysosome, nucleotide excision repair, progesterone-mediated oocyte maturation, and VEGF signaling pathway. The expression level of 6 upregulated miRNAs (mmu-miR-21a-5p, mmu-miR-20a-5p, mmu-miR-17-5p, mmu-miR-30e-3p, mmu-miR-142a-3p, and mmu-miR-106b-3p) was confirmed by stem-loop quantitative reverse transcription PCR, which yielded results consistent with the sequencing data. These findings expand our understanding of the regulatory mechanisms of miRNAs underlying T. gondii pathogenesis and contribute new database information on cat miRNAs, opening a new perspective on the prevention and treatment of T. gondii infection.
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spelling nottingham-406482020-05-04T18:37:22Z https://eprints.nottingham.ac.uk/40648/ Global miRNA expression profiling of domestic cat livers following acute Toxoplasma gondii infection Cong, Wei Zhang, Xiao-Xuan He, Jun-Jun Li, Fa-Cai Elsheikha, Hany M. Zhu, Xing-Quan Although microRNAs (miRNAs) play an important role in liver homeostasis, the extent to which they can be altered by Toxoplasma gondii infection is unknown. Here, we utilized small RNA sequencing and bioinformatic analyses to characterize miRNA expression profiles in the liver of domestic cats at 7 days after oral infection with T. gondii (Type II) strain. A total of 384 miRNAs were identified and 82 were differentially expressed, of which 33 were up-regulated and 49 down-regulated. Also, 5690 predicted host gene targets for the differentially expressed miRNAs were identified using the bioinformatic algorithm miRanda. Gene ontology analysis revealed that the predicted gene targets of the dysregulated miRNAs were significantly enriched in apoptosis. Kyoto Encyclopedia of Genes and Genomes analysis showed that the predicted gene targets were involved in several pathways, including acute myeloid leukemia, central carbon metabolism in cancer, choline metabolism in cancer, estrogen signaling pathway, fatty acid degradation, lysosome, nucleotide excision repair, progesterone-mediated oocyte maturation, and VEGF signaling pathway. The expression level of 6 upregulated miRNAs (mmu-miR-21a-5p, mmu-miR-20a-5p, mmu-miR-17-5p, mmu-miR-30e-3p, mmu-miR-142a-3p, and mmu-miR-106b-3p) was confirmed by stem-loop quantitative reverse transcription PCR, which yielded results consistent with the sequencing data. These findings expand our understanding of the regulatory mechanisms of miRNAs underlying T. gondii pathogenesis and contribute new database information on cat miRNAs, opening a new perspective on the prevention and treatment of T. gondii infection. Impact Journals 2017-03-10 Article PeerReviewed Cong, Wei, Zhang, Xiao-Xuan, He, Jun-Jun, Li, Fa-Cai, Elsheikha, Hany M. and Zhu, Xing-Quan (2017) Global miRNA expression profiling of domestic cat livers following acute Toxoplasma gondii infection. Oncotarget, 8 . pp. 25599-25611. ISSN 1949-2553 Toxoplasma gondii Domestic cat Liver MircoRNA RNA-seq http://www.impactjournals.com/oncotarget/index.php?journal=oncotarget&page=article&op=view&path[]=16108&author-preview=cfg doi:10.18632/oncotarget.16108 doi:10.18632/oncotarget.16108
spellingShingle Toxoplasma gondii
Domestic cat
Liver
MircoRNA
RNA-seq
Cong, Wei
Zhang, Xiao-Xuan
He, Jun-Jun
Li, Fa-Cai
Elsheikha, Hany M.
Zhu, Xing-Quan
Global miRNA expression profiling of domestic cat livers following acute Toxoplasma gondii infection
title Global miRNA expression profiling of domestic cat livers following acute Toxoplasma gondii infection
title_full Global miRNA expression profiling of domestic cat livers following acute Toxoplasma gondii infection
title_fullStr Global miRNA expression profiling of domestic cat livers following acute Toxoplasma gondii infection
title_full_unstemmed Global miRNA expression profiling of domestic cat livers following acute Toxoplasma gondii infection
title_short Global miRNA expression profiling of domestic cat livers following acute Toxoplasma gondii infection
title_sort global mirna expression profiling of domestic cat livers following acute toxoplasma gondii infection
topic Toxoplasma gondii
Domestic cat
Liver
MircoRNA
RNA-seq
url https://eprints.nottingham.ac.uk/40648/
https://eprints.nottingham.ac.uk/40648/
https://eprints.nottingham.ac.uk/40648/