Development of Clostridium difficile R20291ΔPaLoc model strains and in vitro methodologies reveals CdtR is required for the production of CDT to cytotoxic levels

Assessing the regulation of Clostridium difficile transferase (CDT), is complicated by the presence of a Pathogenicity locus (PaLoc) which encodes Toxins A and B. Here we developed R20291ΔPaLoc model strains and cell-based assays to quantify CDT-mediated virulence. Their application demonstrated tha...

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Main Authors: Bilverstone, T.W., Kinsmore, N.L., Minton, Nigel P., Kuehne, Sarah A.
Format: Article
Published: Elsevier 2017
Subjects:
Online Access:https://eprints.nottingham.ac.uk/39999/
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author Bilverstone, T.W.
Kinsmore, N.L.
Minton, Nigel P.
Kuehne, Sarah A.
author_facet Bilverstone, T.W.
Kinsmore, N.L.
Minton, Nigel P.
Kuehne, Sarah A.
author_sort Bilverstone, T.W.
building Nottingham Research Data Repository
collection Online Access
description Assessing the regulation of Clostridium difficile transferase (CDT), is complicated by the presence of a Pathogenicity locus (PaLoc) which encodes Toxins A and B. Here we developed R20291ΔPaLoc model strains and cell-based assays to quantify CDT-mediated virulence. Their application demonstrated that the transcriptional regulator, CdtR, was required for CDT-mediated cytotoxicity.
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institution University of Nottingham Malaysia Campus
institution_category Local University
last_indexed 2025-11-14T19:40:25Z
publishDate 2017
publisher Elsevier
recordtype eprints
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spelling nottingham-399992020-05-04T18:30:23Z https://eprints.nottingham.ac.uk/39999/ Development of Clostridium difficile R20291ΔPaLoc model strains and in vitro methodologies reveals CdtR is required for the production of CDT to cytotoxic levels Bilverstone, T.W. Kinsmore, N.L. Minton, Nigel P. Kuehne, Sarah A. Assessing the regulation of Clostridium difficile transferase (CDT), is complicated by the presence of a Pathogenicity locus (PaLoc) which encodes Toxins A and B. Here we developed R20291ΔPaLoc model strains and cell-based assays to quantify CDT-mediated virulence. Their application demonstrated that the transcriptional regulator, CdtR, was required for CDT-mediated cytotoxicity. Elsevier 2017-01-17 Article PeerReviewed Bilverstone, T.W., Kinsmore, N.L., Minton, Nigel P. and Kuehne, Sarah A. (2017) Development of Clostridium difficile R20291ΔPaLoc model strains and in vitro methodologies reveals CdtR is required for the production of CDT to cytotoxic levels. Anaerobe, 44 . pp. 51-54. ISSN 1095-8274 C. difficile; PaLoc; Binary toxin; CdtR; Virulence http://www.sciencedirect.com/science/article/pii/S1075996417300173 doi:10.1016/j.anaerobe.2017.01.009 doi:10.1016/j.anaerobe.2017.01.009
spellingShingle C. difficile; PaLoc; Binary toxin; CdtR; Virulence
Bilverstone, T.W.
Kinsmore, N.L.
Minton, Nigel P.
Kuehne, Sarah A.
Development of Clostridium difficile R20291ΔPaLoc model strains and in vitro methodologies reveals CdtR is required for the production of CDT to cytotoxic levels
title Development of Clostridium difficile R20291ΔPaLoc model strains and in vitro methodologies reveals CdtR is required for the production of CDT to cytotoxic levels
title_full Development of Clostridium difficile R20291ΔPaLoc model strains and in vitro methodologies reveals CdtR is required for the production of CDT to cytotoxic levels
title_fullStr Development of Clostridium difficile R20291ΔPaLoc model strains and in vitro methodologies reveals CdtR is required for the production of CDT to cytotoxic levels
title_full_unstemmed Development of Clostridium difficile R20291ΔPaLoc model strains and in vitro methodologies reveals CdtR is required for the production of CDT to cytotoxic levels
title_short Development of Clostridium difficile R20291ΔPaLoc model strains and in vitro methodologies reveals CdtR is required for the production of CDT to cytotoxic levels
title_sort development of clostridium difficile r20291δpaloc model strains and in vitro methodologies reveals cdtr is required for the production of cdt to cytotoxic levels
topic C. difficile; PaLoc; Binary toxin; CdtR; Virulence
url https://eprints.nottingham.ac.uk/39999/
https://eprints.nottingham.ac.uk/39999/
https://eprints.nottingham.ac.uk/39999/