Pharmacology of modulators of alternative splicing

More than 95% of genes in the human genome are alternatively spliced to form multiple transcripts, often encoding proteins with differing or opposing function. The control of alternative splicing is now being elucidated, and with this comes the opportunity to develop modulators of alternative splici...

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Main Authors: Bates, David O., Morris, Jonathan C., Oltean, Sebastian, Donaldson, Lucy F.
Format: Article
Published: American Society for Pharmacology and Experimental Therapeutics 2017
Online Access:https://eprints.nottingham.ac.uk/39444/
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author Bates, David O.
Morris, Jonathan C.
Oltean, Sebastian
Donaldson, Lucy F.
author_facet Bates, David O.
Morris, Jonathan C.
Oltean, Sebastian
Donaldson, Lucy F.
author_sort Bates, David O.
building Nottingham Research Data Repository
collection Online Access
description More than 95% of genes in the human genome are alternatively spliced to form multiple transcripts, often encoding proteins with differing or opposing function. The control of alternative splicing is now being elucidated, and with this comes the opportunity to develop modulators of alternative splicing that can control cellular function. A number of approaches have been taken to develop compounds that can experimentally, and sometimes clinically, affect splicing control resulting in potential novel therapeutics. Here we develop the concepts that targeting alternative splicing can result in relatively specific pathway inhibitors/activators that result in dampening down of physiological or pathological processes, from changes in muscle physiology, to altering angiogenesis or pain. The targets and pharmacology of some of the current inhibitors/activators of alternative splicing are demonstrated and future directions discussed.
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spelling nottingham-394442020-05-04T18:22:44Z https://eprints.nottingham.ac.uk/39444/ Pharmacology of modulators of alternative splicing Bates, David O. Morris, Jonathan C. Oltean, Sebastian Donaldson, Lucy F. More than 95% of genes in the human genome are alternatively spliced to form multiple transcripts, often encoding proteins with differing or opposing function. The control of alternative splicing is now being elucidated, and with this comes the opportunity to develop modulators of alternative splicing that can control cellular function. A number of approaches have been taken to develop compounds that can experimentally, and sometimes clinically, affect splicing control resulting in potential novel therapeutics. Here we develop the concepts that targeting alternative splicing can result in relatively specific pathway inhibitors/activators that result in dampening down of physiological or pathological processes, from changes in muscle physiology, to altering angiogenesis or pain. The targets and pharmacology of some of the current inhibitors/activators of alternative splicing are demonstrated and future directions discussed. American Society for Pharmacology and Experimental Therapeutics 2017-01-01 Article PeerReviewed Bates, David O., Morris, Jonathan C., Oltean, Sebastian and Donaldson, Lucy F. (2017) Pharmacology of modulators of alternative splicing. Pharmacological Reviews, 69 (1). pp. 63-79. ISSN 1521-0081 http://pharmrev.aspetjournals.org/content/69/1/63 doi:10.1124/pr.115.011239 doi:10.1124/pr.115.011239
spellingShingle Bates, David O.
Morris, Jonathan C.
Oltean, Sebastian
Donaldson, Lucy F.
Pharmacology of modulators of alternative splicing
title Pharmacology of modulators of alternative splicing
title_full Pharmacology of modulators of alternative splicing
title_fullStr Pharmacology of modulators of alternative splicing
title_full_unstemmed Pharmacology of modulators of alternative splicing
title_short Pharmacology of modulators of alternative splicing
title_sort pharmacology of modulators of alternative splicing
url https://eprints.nottingham.ac.uk/39444/
https://eprints.nottingham.ac.uk/39444/
https://eprints.nottingham.ac.uk/39444/