Modulation of the immune response to Mycobacterium tuberculosis during malaria/M. tuberculosis co-infection

Tuberculosis (TB) causes significant morbidity and mortality on a global scale. The African region has 24% of the world's TB cases. TB overlaps with other infectious diseases such as malaria and HIV, which are also highly prevalent in the African region. TB is a leading cause of death among HIV...

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Main Authors: Chukwuanukwu, Rebecca C., Onyenekwe, Charles C., Martinez-Pomares, Luisa, Flynn, Robin J., Singh, Sonali, Amilo, Grace I., Agbakoba, Nneka R., Okoye, Jude O.
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Published: Wiley 2016
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Online Access:https://eprints.nottingham.ac.uk/37827/
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author Chukwuanukwu, Rebecca C.
Onyenekwe, Charles C.
Martinez-Pomares, Luisa
Flynn, Robin J.
Singh, Sonali
Amilo, Grace I.
Agbakoba, Nneka R.
Okoye, Jude O.
author_facet Chukwuanukwu, Rebecca C.
Onyenekwe, Charles C.
Martinez-Pomares, Luisa
Flynn, Robin J.
Singh, Sonali
Amilo, Grace I.
Agbakoba, Nneka R.
Okoye, Jude O.
author_sort Chukwuanukwu, Rebecca C.
building Nottingham Research Data Repository
collection Online Access
description Tuberculosis (TB) causes significant morbidity and mortality on a global scale. The African region has 24% of the world's TB cases. TB overlaps with other infectious diseases such as malaria and HIV, which are also highly prevalent in the African region. TB is a leading cause of death among HIV-positive patients and co-infection with HIV and TB has been described as a syndemic. In view of the overlapping epidemiology of these diseases, it is important to understand the dynamics of the immune response to TB in the context of co-infection. We investigated the cytokine response to purified protein derivative (PPD) in peripheral blood mononuclear cells from TB patients co-infected with HIV or malaria and compared it to that of malaria- and HIV-free TB patients. A total of 231 subjects were recruited for this study and classified into six groups; untreated TB-positive, TB positive subjects on TB drugs, TB- and HIV-positive, TB- and malaria-positive, latent TB and apparently healthy control subjects. Our results demonstrate maintenance of interferon (IFN)-γ production in HIV and malaria co-infected TB patients in spite of lower CD4 counts in the HIV-infected cohort. Malaria co-infection caused an increase in the production of the T helper type 2 (Th2)-associated cytokine interleukin (IL)-4 and the anti-inflammatory cytokine IL-10 in PPD-stimulated cultures. These results suggest that malaria co-infection diverts immune response against M. tuberculosis towards a Th-2/anti-inflammatory response which might have important consequences for disease progression.
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spelling nottingham-378272020-05-04T18:16:04Z https://eprints.nottingham.ac.uk/37827/ Modulation of the immune response to Mycobacterium tuberculosis during malaria/M. tuberculosis co-infection Chukwuanukwu, Rebecca C. Onyenekwe, Charles C. Martinez-Pomares, Luisa Flynn, Robin J. Singh, Sonali Amilo, Grace I. Agbakoba, Nneka R. Okoye, Jude O. Tuberculosis (TB) causes significant morbidity and mortality on a global scale. The African region has 24% of the world's TB cases. TB overlaps with other infectious diseases such as malaria and HIV, which are also highly prevalent in the African region. TB is a leading cause of death among HIV-positive patients and co-infection with HIV and TB has been described as a syndemic. In view of the overlapping epidemiology of these diseases, it is important to understand the dynamics of the immune response to TB in the context of co-infection. We investigated the cytokine response to purified protein derivative (PPD) in peripheral blood mononuclear cells from TB patients co-infected with HIV or malaria and compared it to that of malaria- and HIV-free TB patients. A total of 231 subjects were recruited for this study and classified into six groups; untreated TB-positive, TB positive subjects on TB drugs, TB- and HIV-positive, TB- and malaria-positive, latent TB and apparently healthy control subjects. Our results demonstrate maintenance of interferon (IFN)-γ production in HIV and malaria co-infected TB patients in spite of lower CD4 counts in the HIV-infected cohort. Malaria co-infection caused an increase in the production of the T helper type 2 (Th2)-associated cytokine interleukin (IL)-4 and the anti-inflammatory cytokine IL-10 in PPD-stimulated cultures. These results suggest that malaria co-infection diverts immune response against M. tuberculosis towards a Th-2/anti-inflammatory response which might have important consequences for disease progression. Wiley 2016-10-21 Article PeerReviewed Chukwuanukwu, Rebecca C., Onyenekwe, Charles C., Martinez-Pomares, Luisa, Flynn, Robin J., Singh, Sonali, Amilo, Grace I., Agbakoba, Nneka R. and Okoye, Jude O. (2016) Modulation of the immune response to Mycobacterium tuberculosis during malaria/M. tuberculosis co-infection. Clinical and Experimental Immunology, 187 (2). pp. 259-268. ISSN 1365-2249 co-infection cytokines HIV malaria tuberculosis http://onlinelibrary.wiley.com/doi/10.1111/cei.12861/abstract doi:10.1111/cei.12861 doi:10.1111/cei.12861
spellingShingle co-infection
cytokines
HIV
malaria
tuberculosis
Chukwuanukwu, Rebecca C.
Onyenekwe, Charles C.
Martinez-Pomares, Luisa
Flynn, Robin J.
Singh, Sonali
Amilo, Grace I.
Agbakoba, Nneka R.
Okoye, Jude O.
Modulation of the immune response to Mycobacterium tuberculosis during malaria/M. tuberculosis co-infection
title Modulation of the immune response to Mycobacterium tuberculosis during malaria/M. tuberculosis co-infection
title_full Modulation of the immune response to Mycobacterium tuberculosis during malaria/M. tuberculosis co-infection
title_fullStr Modulation of the immune response to Mycobacterium tuberculosis during malaria/M. tuberculosis co-infection
title_full_unstemmed Modulation of the immune response to Mycobacterium tuberculosis during malaria/M. tuberculosis co-infection
title_short Modulation of the immune response to Mycobacterium tuberculosis during malaria/M. tuberculosis co-infection
title_sort modulation of the immune response to mycobacterium tuberculosis during malaria/m. tuberculosis co-infection
topic co-infection
cytokines
HIV
malaria
tuberculosis
url https://eprints.nottingham.ac.uk/37827/
https://eprints.nottingham.ac.uk/37827/
https://eprints.nottingham.ac.uk/37827/