Computational modeling of the effects of auditory nerve dysmyelination

Our previous study showed that exposure to loud sound leading to hearing loss elongated the auditory nerve (AN) nodes of Ranvier and triggered notable morphological changes at paranodes and juxtaparanodes. Here we used computational modeling to examine how theoretical redistribution of voltage gated...

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Main Authors: Brown, Angus M., Hamann, Martine
Format: Article
Published: Frontiers Media 2014
Subjects:
Online Access:https://eprints.nottingham.ac.uk/35377/
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author Brown, Angus M.
Hamann, Martine
author_facet Brown, Angus M.
Hamann, Martine
author_sort Brown, Angus M.
building Nottingham Research Data Repository
collection Online Access
description Our previous study showed that exposure to loud sound leading to hearing loss elongated the auditory nerve (AN) nodes of Ranvier and triggered notable morphological changes at paranodes and juxtaparanodes. Here we used computational modeling to examine how theoretical redistribution of voltage gated Na+, Kv3.1, and Kv1.1 channels along the AN may be responsible for the alterations of conduction property following acoustic over-exposure. Our modeling study infers that changes related to Na+ channel density (rather than the redistribution of voltage gated Na+, Kv3.1, and Kv1.1 channels) is the likely cause of the decreased conduction velocity and the conduction block observed after acoustic overexposure (AOE).
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spelling nottingham-353772020-05-04T16:49:58Z https://eprints.nottingham.ac.uk/35377/ Computational modeling of the effects of auditory nerve dysmyelination Brown, Angus M. Hamann, Martine Our previous study showed that exposure to loud sound leading to hearing loss elongated the auditory nerve (AN) nodes of Ranvier and triggered notable morphological changes at paranodes and juxtaparanodes. Here we used computational modeling to examine how theoretical redistribution of voltage gated Na+, Kv3.1, and Kv1.1 channels along the AN may be responsible for the alterations of conduction property following acoustic over-exposure. Our modeling study infers that changes related to Na+ channel density (rather than the redistribution of voltage gated Na+, Kv3.1, and Kv1.1 channels) is the likely cause of the decreased conduction velocity and the conduction block observed after acoustic overexposure (AOE). Frontiers Media 2014-08-01 Article PeerReviewed Brown, Angus M. and Hamann, Martine (2014) Computational modeling of the effects of auditory nerve dysmyelination. Frontiers in Neuroanatomy, 8 (73). pp. 1-7. ISSN 1662-5129 myelin sheath hearing loss node of Ranvier conduction velocity conduction block deafness myelin domains action potential http://journal.frontiersin.org/article/10.3389/fnana.2014.00073/full doi:10.3389/fnana.2014.00073 doi:10.3389/fnana.2014.00073
spellingShingle myelin sheath
hearing loss
node of Ranvier
conduction velocity
conduction block
deafness
myelin domains
action potential
Brown, Angus M.
Hamann, Martine
Computational modeling of the effects of auditory nerve dysmyelination
title Computational modeling of the effects of auditory nerve dysmyelination
title_full Computational modeling of the effects of auditory nerve dysmyelination
title_fullStr Computational modeling of the effects of auditory nerve dysmyelination
title_full_unstemmed Computational modeling of the effects of auditory nerve dysmyelination
title_short Computational modeling of the effects of auditory nerve dysmyelination
title_sort computational modeling of the effects of auditory nerve dysmyelination
topic myelin sheath
hearing loss
node of Ranvier
conduction velocity
conduction block
deafness
myelin domains
action potential
url https://eprints.nottingham.ac.uk/35377/
https://eprints.nottingham.ac.uk/35377/
https://eprints.nottingham.ac.uk/35377/