Recombination is a key driver of genomic and phenotypic diversity in a Pseudomonas aeruginosa population during cystic fibrosis infection

The Cystic Fibrosis (CF) lung harbors a complex, polymicrobial ecosystem, in which Pseudomonas aeruginosa is capable of sustaining chronic infections, which are highly resistant to multiple antibiotics. Here, we investigate the phenotypic and genotypic diversity of 44 morphologically identical P. ae...

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Main Authors: Darch, Sophie E., McNally, Alan, Harrison, Freya, Corander, Jukka, Barr, Helen L., Paszkiewicz, Konrad, Holden, Stephen, Fogarty, Andrew W., Crusz, Shanika A., Diggle, Stephen P.
Format: Article
Published: Nature Publishing Group 2015
Online Access:https://eprints.nottingham.ac.uk/31951/
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author Darch, Sophie E.
McNally, Alan
Harrison, Freya
Corander, Jukka
Barr, Helen L.
Paszkiewicz, Konrad
Holden, Stephen
Fogarty, Andrew W.
Crusz, Shanika A.
Diggle, Stephen P.
author_facet Darch, Sophie E.
McNally, Alan
Harrison, Freya
Corander, Jukka
Barr, Helen L.
Paszkiewicz, Konrad
Holden, Stephen
Fogarty, Andrew W.
Crusz, Shanika A.
Diggle, Stephen P.
author_sort Darch, Sophie E.
building Nottingham Research Data Repository
collection Online Access
description The Cystic Fibrosis (CF) lung harbors a complex, polymicrobial ecosystem, in which Pseudomonas aeruginosa is capable of sustaining chronic infections, which are highly resistant to multiple antibiotics. Here, we investigate the phenotypic and genotypic diversity of 44 morphologically identical P. aeruginosa isolates taken from a single CF patient sputum sample. Comprehensive phenotypic analysis of isolates revealed large variances and trade-offs in growth, virulence factors and quorum sensing (QS) signals. Whole genome analysis of 22 isolates revealed high levels of intra-isolate diversity ranging from 5 to 64 SNPs and that recombination and not spontaneous mutation was the dominant driver of diversity in this population. Furthermore, phenotypic differences between isolates were not linked to mutations in known genes but were statistically associated with distinct recombination events. We also assessed antibiotic susceptibility of all isolates. Resistance to antibiotics significantly increased when multiple isolates were mixed together. Our results highlight the significant role of recombination in generating phenotypic and genetic diversification during in vivo chronic CF infection. We also discuss (i) how these findings could influence how patient-to-patient transmission studies are performed using whole genome sequencing, and (ii) the need to refine antibiotic susceptibility testing in sputum samples taken from patients with CF.
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spelling nottingham-319512020-05-04T17:01:07Z https://eprints.nottingham.ac.uk/31951/ Recombination is a key driver of genomic and phenotypic diversity in a Pseudomonas aeruginosa population during cystic fibrosis infection Darch, Sophie E. McNally, Alan Harrison, Freya Corander, Jukka Barr, Helen L. Paszkiewicz, Konrad Holden, Stephen Fogarty, Andrew W. Crusz, Shanika A. Diggle, Stephen P. The Cystic Fibrosis (CF) lung harbors a complex, polymicrobial ecosystem, in which Pseudomonas aeruginosa is capable of sustaining chronic infections, which are highly resistant to multiple antibiotics. Here, we investigate the phenotypic and genotypic diversity of 44 morphologically identical P. aeruginosa isolates taken from a single CF patient sputum sample. Comprehensive phenotypic analysis of isolates revealed large variances and trade-offs in growth, virulence factors and quorum sensing (QS) signals. Whole genome analysis of 22 isolates revealed high levels of intra-isolate diversity ranging from 5 to 64 SNPs and that recombination and not spontaneous mutation was the dominant driver of diversity in this population. Furthermore, phenotypic differences between isolates were not linked to mutations in known genes but were statistically associated with distinct recombination events. We also assessed antibiotic susceptibility of all isolates. Resistance to antibiotics significantly increased when multiple isolates were mixed together. Our results highlight the significant role of recombination in generating phenotypic and genetic diversification during in vivo chronic CF infection. We also discuss (i) how these findings could influence how patient-to-patient transmission studies are performed using whole genome sequencing, and (ii) the need to refine antibiotic susceptibility testing in sputum samples taken from patients with CF. Nature Publishing Group 2015-01-12 Article PeerReviewed Darch, Sophie E., McNally, Alan, Harrison, Freya, Corander, Jukka, Barr, Helen L., Paszkiewicz, Konrad, Holden, Stephen, Fogarty, Andrew W., Crusz, Shanika A. and Diggle, Stephen P. (2015) Recombination is a key driver of genomic and phenotypic diversity in a Pseudomonas aeruginosa population during cystic fibrosis infection. Scientific Reports, 5 . 7649/1-7649/12. ISSN 2045-2322 http://www.nature.com/articles/srep07649 doi:10.1038/srep07649 doi:10.1038/srep07649
spellingShingle Darch, Sophie E.
McNally, Alan
Harrison, Freya
Corander, Jukka
Barr, Helen L.
Paszkiewicz, Konrad
Holden, Stephen
Fogarty, Andrew W.
Crusz, Shanika A.
Diggle, Stephen P.
Recombination is a key driver of genomic and phenotypic diversity in a Pseudomonas aeruginosa population during cystic fibrosis infection
title Recombination is a key driver of genomic and phenotypic diversity in a Pseudomonas aeruginosa population during cystic fibrosis infection
title_full Recombination is a key driver of genomic and phenotypic diversity in a Pseudomonas aeruginosa population during cystic fibrosis infection
title_fullStr Recombination is a key driver of genomic and phenotypic diversity in a Pseudomonas aeruginosa population during cystic fibrosis infection
title_full_unstemmed Recombination is a key driver of genomic and phenotypic diversity in a Pseudomonas aeruginosa population during cystic fibrosis infection
title_short Recombination is a key driver of genomic and phenotypic diversity in a Pseudomonas aeruginosa population during cystic fibrosis infection
title_sort recombination is a key driver of genomic and phenotypic diversity in a pseudomonas aeruginosa population during cystic fibrosis infection
url https://eprints.nottingham.ac.uk/31951/
https://eprints.nottingham.ac.uk/31951/
https://eprints.nottingham.ac.uk/31951/