Pro-inflammatory cytokines can act as intracellular modulators of commensal bacterial virulence

Interactions between commensal pathogens and hosts are critical for disease development but the underlying mechanisms for switching between the commensal and virulent states are unknown. We show that the human pathogen Neisseria meningitidis, the leading cause of pyogenic meningitis, can modulate ge...

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Main Authors: Mahdavi, Jafar, Royer, Pierre-Joseph, Sjolinder, Hong S., Azimi, Sheyda, Self, Tim, Stoof, Jeroen, Wheldon, Lee M., Brannstrom, Kristoffer, Wilson, Raymond, Moreton, Joanna, Moir, James W.B., Sihlbom, Carina, Boren, Thomas, Jonssen, Ann-Beth, Soultanas, Panos, Ala'Aldeen, Dlawer A.A.
Format: Article
Published: Royal Society Publishing 2013
Online Access:https://eprints.nottingham.ac.uk/31165/
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author Mahdavi, Jafar
Royer, Pierre-Joseph
Sjolinder, Hong S.
Azimi, Sheyda
Self, Tim
Stoof, Jeroen
Wheldon, Lee M.
Brannstrom, Kristoffer
Wilson, Raymond
Moreton, Joanna
Moir, James W.B.
Sihlbom, Carina
Boren, Thomas
Jonssen, Ann-Beth
Soultanas, Panos
Ala'Aldeen, Dlawer A.A.
author_facet Mahdavi, Jafar
Royer, Pierre-Joseph
Sjolinder, Hong S.
Azimi, Sheyda
Self, Tim
Stoof, Jeroen
Wheldon, Lee M.
Brannstrom, Kristoffer
Wilson, Raymond
Moreton, Joanna
Moir, James W.B.
Sihlbom, Carina
Boren, Thomas
Jonssen, Ann-Beth
Soultanas, Panos
Ala'Aldeen, Dlawer A.A.
author_sort Mahdavi, Jafar
building Nottingham Research Data Repository
collection Online Access
description Interactions between commensal pathogens and hosts are critical for disease development but the underlying mechanisms for switching between the commensal and virulent states are unknown. We show that the human pathogen Neisseria meningitidis, the leading cause of pyogenic meningitis, can modulate gene expression via uptake of host pro-inflammatory cytokines leading to increased virulence. This uptake is mediated by type IV pili (Tfp) and reliant on the PilT ATPase activity. Two Tfp subunits, PilE and PilQ, are identified as the ligands for TNF-a and IL-8 in a glycan-dependent manner, and their deletion results in decreased virulence and increased survival in a mouse model. We propose a novel mechanism by which pathogens use the twitching motility mode of the Tfp machinery for sensing and importing host elicitors, aligning with the inflamed environment and switching to the virulent state.
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publishDate 2013
publisher Royal Society Publishing
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spelling nottingham-311652020-05-04T16:39:26Z https://eprints.nottingham.ac.uk/31165/ Pro-inflammatory cytokines can act as intracellular modulators of commensal bacterial virulence Mahdavi, Jafar Royer, Pierre-Joseph Sjolinder, Hong S. Azimi, Sheyda Self, Tim Stoof, Jeroen Wheldon, Lee M. Brannstrom, Kristoffer Wilson, Raymond Moreton, Joanna Moir, James W.B. Sihlbom, Carina Boren, Thomas Jonssen, Ann-Beth Soultanas, Panos Ala'Aldeen, Dlawer A.A. Interactions between commensal pathogens and hosts are critical for disease development but the underlying mechanisms for switching between the commensal and virulent states are unknown. We show that the human pathogen Neisseria meningitidis, the leading cause of pyogenic meningitis, can modulate gene expression via uptake of host pro-inflammatory cytokines leading to increased virulence. This uptake is mediated by type IV pili (Tfp) and reliant on the PilT ATPase activity. Two Tfp subunits, PilE and PilQ, are identified as the ligands for TNF-a and IL-8 in a glycan-dependent manner, and their deletion results in decreased virulence and increased survival in a mouse model. We propose a novel mechanism by which pathogens use the twitching motility mode of the Tfp machinery for sensing and importing host elicitors, aligning with the inflamed environment and switching to the virulent state. Royal Society Publishing 2013-10-09 Article PeerReviewed Mahdavi, Jafar, Royer, Pierre-Joseph, Sjolinder, Hong S., Azimi, Sheyda, Self, Tim, Stoof, Jeroen, Wheldon, Lee M., Brannstrom, Kristoffer, Wilson, Raymond, Moreton, Joanna, Moir, James W.B., Sihlbom, Carina, Boren, Thomas, Jonssen, Ann-Beth, Soultanas, Panos and Ala'Aldeen, Dlawer A.A. (2013) Pro-inflammatory cytokines can act as intracellular modulators of commensal bacterial virulence. Open Biology, 3 . e130048/1-e130048/15. ISSN 2046-2441 http://rsob.royalsocietypublishing.org/content/3/10/130048 doi:10.1098/rsob.130048 doi:10.1098/rsob.130048
spellingShingle Mahdavi, Jafar
Royer, Pierre-Joseph
Sjolinder, Hong S.
Azimi, Sheyda
Self, Tim
Stoof, Jeroen
Wheldon, Lee M.
Brannstrom, Kristoffer
Wilson, Raymond
Moreton, Joanna
Moir, James W.B.
Sihlbom, Carina
Boren, Thomas
Jonssen, Ann-Beth
Soultanas, Panos
Ala'Aldeen, Dlawer A.A.
Pro-inflammatory cytokines can act as intracellular modulators of commensal bacterial virulence
title Pro-inflammatory cytokines can act as intracellular modulators of commensal bacterial virulence
title_full Pro-inflammatory cytokines can act as intracellular modulators of commensal bacterial virulence
title_fullStr Pro-inflammatory cytokines can act as intracellular modulators of commensal bacterial virulence
title_full_unstemmed Pro-inflammatory cytokines can act as intracellular modulators of commensal bacterial virulence
title_short Pro-inflammatory cytokines can act as intracellular modulators of commensal bacterial virulence
title_sort pro-inflammatory cytokines can act as intracellular modulators of commensal bacterial virulence
url https://eprints.nottingham.ac.uk/31165/
https://eprints.nottingham.ac.uk/31165/
https://eprints.nottingham.ac.uk/31165/