The acute impact of a hematopoietic allograft on lung function and inflammation: a prospective observational study

Background: No studies have investigated the immediate impact of receiving an allogeneic hematopoietic stem cell transplant (HSCT) on pulmonary inflammation or lung function. Methods: Using a prospective study design, we quantified the changes in these outcome measures in eligible adult individu...

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Main Authors: Enocson, Alexandra, Hubbard, Richard, McKeever, Tricia M., Russell, Nigel, Byrne, Jennifer, Das-Gupta, Emma, Watson, Lynne, Fogarty, Andrew W.
Format: Article
Published: BioMed Central 2013
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Online Access:https://eprints.nottingham.ac.uk/2934/
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author Enocson, Alexandra
Hubbard, Richard
McKeever, Tricia M.
Russell, Nigel
Byrne, Jennifer
Das-Gupta, Emma
Watson, Lynne
Fogarty, Andrew W.
author_facet Enocson, Alexandra
Hubbard, Richard
McKeever, Tricia M.
Russell, Nigel
Byrne, Jennifer
Das-Gupta, Emma
Watson, Lynne
Fogarty, Andrew W.
author_sort Enocson, Alexandra
building Nottingham Research Data Repository
collection Online Access
description Background: No studies have investigated the immediate impact of receiving an allogeneic hematopoietic stem cell transplant (HSCT) on pulmonary inflammation or lung function. Methods: Using a prospective study design, we quantified the changes in these outcome measures in eligible adult individuals in the first six months after receiving an allogeneic hematopoietic stem cell transplant. Results: Between January 2007 and December 2008, 72 patients were eligible to participate in the cohort, and of these 68 (94%) were included in the study. Compared to baseline, pulmonary inflammation as measured by exhaled nitric oxide increased after receiving a HSCT with the largest increment seen at three months (+6.0ppb, 95%CI: +0.4 to +11.5), and this was sustained at six months. Percent predicted forced expiratory volume in one second decreased over the same period, with the largest decrease observed at six weeks (−5.9%, 95% CI: -8.9 to −2.9), and this was also sustained over a six month period. Similar associations were observed for FVC. A larger increase in exhaled nitric oxide from baseline at six weeks and three months may be associated with decreased mortality (p=0.06, p=0.04 respectively). Conclusion: Our data demonstrate that recipients of an allogeneic HSCT experience an increase in biomarkers of pulmonary inflammation and a decrease in lung function in the first six months after the procedure. If independently validated in other study populations, these observations could have potential as a prognostic biomarker for this patient group.
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spelling nottingham-29342020-05-04T16:35:31Z https://eprints.nottingham.ac.uk/2934/ The acute impact of a hematopoietic allograft on lung function and inflammation: a prospective observational study Enocson, Alexandra Hubbard, Richard McKeever, Tricia M. Russell, Nigel Byrne, Jennifer Das-Gupta, Emma Watson, Lynne Fogarty, Andrew W. Background: No studies have investigated the immediate impact of receiving an allogeneic hematopoietic stem cell transplant (HSCT) on pulmonary inflammation or lung function. Methods: Using a prospective study design, we quantified the changes in these outcome measures in eligible adult individuals in the first six months after receiving an allogeneic hematopoietic stem cell transplant. Results: Between January 2007 and December 2008, 72 patients were eligible to participate in the cohort, and of these 68 (94%) were included in the study. Compared to baseline, pulmonary inflammation as measured by exhaled nitric oxide increased after receiving a HSCT with the largest increment seen at three months (+6.0ppb, 95%CI: +0.4 to +11.5), and this was sustained at six months. Percent predicted forced expiratory volume in one second decreased over the same period, with the largest decrease observed at six weeks (−5.9%, 95% CI: -8.9 to −2.9), and this was also sustained over a six month period. Similar associations were observed for FVC. A larger increase in exhaled nitric oxide from baseline at six weeks and three months may be associated with decreased mortality (p=0.06, p=0.04 respectively). Conclusion: Our data demonstrate that recipients of an allogeneic HSCT experience an increase in biomarkers of pulmonary inflammation and a decrease in lung function in the first six months after the procedure. If independently validated in other study populations, these observations could have potential as a prognostic biomarker for this patient group. BioMed Central 2013-01-11 Article PeerReviewed Enocson, Alexandra, Hubbard, Richard, McKeever, Tricia M., Russell, Nigel, Byrne, Jennifer, Das-Gupta, Emma, Watson, Lynne and Fogarty, Andrew W. (2013) The acute impact of a hematopoietic allograft on lung function and inflammation: a prospective observational study. BMC Pulmonary Medicine, 13 (Januar). 6/1-6/6. ISSN 1471-2466 Lung function Inflammation Haematopoietic transplant http://www.biomedcentral.com/1471-2466/13/2 doi:10.1186/1471-2466-13-2 doi:10.1186/1471-2466-13-2
spellingShingle Lung function
Inflammation
Haematopoietic transplant
Enocson, Alexandra
Hubbard, Richard
McKeever, Tricia M.
Russell, Nigel
Byrne, Jennifer
Das-Gupta, Emma
Watson, Lynne
Fogarty, Andrew W.
The acute impact of a hematopoietic allograft on lung function and inflammation: a prospective observational study
title The acute impact of a hematopoietic allograft on lung function and inflammation: a prospective observational study
title_full The acute impact of a hematopoietic allograft on lung function and inflammation: a prospective observational study
title_fullStr The acute impact of a hematopoietic allograft on lung function and inflammation: a prospective observational study
title_full_unstemmed The acute impact of a hematopoietic allograft on lung function and inflammation: a prospective observational study
title_short The acute impact of a hematopoietic allograft on lung function and inflammation: a prospective observational study
title_sort acute impact of a hematopoietic allograft on lung function and inflammation: a prospective observational study
topic Lung function
Inflammation
Haematopoietic transplant
url https://eprints.nottingham.ac.uk/2934/
https://eprints.nottingham.ac.uk/2934/
https://eprints.nottingham.ac.uk/2934/