Retagging identifies dendritic cell-specific intercellular adhesion molecule-3 (ICAM3)-grabbing non-integrin (DC-SIGN) protein as a novel receptor for a major allergen from house dust mite

Dendritic cells (DCs) have been shown to play a key role in the initiation and maintenance of immune responses to microbial pathogens as well as to allergens, but the exact mechanisms of their involvement in allergic responses and Th2 cell differentiation have remained elusive. Using retagging, we i...

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Main Authors: Emara, Mohamed, Royer, Pierre-Joseph, Mahdavi, Jafar, Shakib, Farouk, Ghaemmaghami, Amir M.
Format: Article
Published: American Society for Biochemistry and Molecular Biology 2012
Subjects:
Online Access:https://eprints.nottingham.ac.uk/2297/
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author Emara, Mohamed
Royer, Pierre-Joseph
Mahdavi, Jafar
Shakib, Farouk
Ghaemmaghami, Amir M.
author_facet Emara, Mohamed
Royer, Pierre-Joseph
Mahdavi, Jafar
Shakib, Farouk
Ghaemmaghami, Amir M.
author_sort Emara, Mohamed
building Nottingham Research Data Repository
collection Online Access
description Dendritic cells (DCs) have been shown to play a key role in the initiation and maintenance of immune responses to microbial pathogens as well as to allergens, but the exact mechanisms of their involvement in allergic responses and Th2 cell differentiation have remained elusive. Using retagging, we identified DCSIGN as a novel receptor involved in the initial recognition and uptake of the major house dust mite and dog allergens Der p 1 and Can f 1, respectively. To confirm this, we used gene silencing to specifically inhibit DC-SIGN expression by DCs followed by allergen uptake studies. Binding and uptake of Der p 1 and Can f 1 allergens was assessed by ELISA and flow cytometry. Intriguingly, our data showed that silencing DC-SIGN on DCs promotes a Th2 phenotype in DC/T cell co-cultures. These findings should lead to better understanding of the molecular basis of allergen-induced Th2 cell polarization and in doing so paves the way for the rational design of novel intervention strategies by targeting allergen receptors on innate immune cells or their carbohydrate counterstructures on allergens.
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spelling nottingham-22972020-05-04T16:32:30Z https://eprints.nottingham.ac.uk/2297/ Retagging identifies dendritic cell-specific intercellular adhesion molecule-3 (ICAM3)-grabbing non-integrin (DC-SIGN) protein as a novel receptor for a major allergen from house dust mite Emara, Mohamed Royer, Pierre-Joseph Mahdavi, Jafar Shakib, Farouk Ghaemmaghami, Amir M. Dendritic cells (DCs) have been shown to play a key role in the initiation and maintenance of immune responses to microbial pathogens as well as to allergens, but the exact mechanisms of their involvement in allergic responses and Th2 cell differentiation have remained elusive. Using retagging, we identified DCSIGN as a novel receptor involved in the initial recognition and uptake of the major house dust mite and dog allergens Der p 1 and Can f 1, respectively. To confirm this, we used gene silencing to specifically inhibit DC-SIGN expression by DCs followed by allergen uptake studies. Binding and uptake of Der p 1 and Can f 1 allergens was assessed by ELISA and flow cytometry. Intriguingly, our data showed that silencing DC-SIGN on DCs promotes a Th2 phenotype in DC/T cell co-cultures. These findings should lead to better understanding of the molecular basis of allergen-induced Th2 cell polarization and in doing so paves the way for the rational design of novel intervention strategies by targeting allergen receptors on innate immune cells or their carbohydrate counterstructures on allergens. American Society for Biochemistry and Molecular Biology 2012-02-17 Article PeerReviewed Emara, Mohamed, Royer, Pierre-Joseph, Mahdavi, Jafar, Shakib, Farouk and Ghaemmaghami, Amir M. (2012) Retagging identifies dendritic cell-specific intercellular adhesion molecule-3 (ICAM3)-grabbing non-integrin (DC-SIGN) protein as a novel receptor for a major allergen from house dust mite. Journal of Biological Chemistry, 287 (8). pp. 5756-5763. ISSN 0021-9258 Immunology Glycobiology Extracellular Matrices http://www.jbc.org/content/287/8/5756 doi:10.1074/jbc.M111.312520 doi:10.1074/jbc.M111.312520
spellingShingle Immunology Glycobiology
Extracellular Matrices
Emara, Mohamed
Royer, Pierre-Joseph
Mahdavi, Jafar
Shakib, Farouk
Ghaemmaghami, Amir M.
Retagging identifies dendritic cell-specific intercellular adhesion molecule-3 (ICAM3)-grabbing non-integrin (DC-SIGN) protein as a novel receptor for a major allergen from house dust mite
title Retagging identifies dendritic cell-specific intercellular adhesion molecule-3 (ICAM3)-grabbing non-integrin (DC-SIGN) protein as a novel receptor for a major allergen from house dust mite
title_full Retagging identifies dendritic cell-specific intercellular adhesion molecule-3 (ICAM3)-grabbing non-integrin (DC-SIGN) protein as a novel receptor for a major allergen from house dust mite
title_fullStr Retagging identifies dendritic cell-specific intercellular adhesion molecule-3 (ICAM3)-grabbing non-integrin (DC-SIGN) protein as a novel receptor for a major allergen from house dust mite
title_full_unstemmed Retagging identifies dendritic cell-specific intercellular adhesion molecule-3 (ICAM3)-grabbing non-integrin (DC-SIGN) protein as a novel receptor for a major allergen from house dust mite
title_short Retagging identifies dendritic cell-specific intercellular adhesion molecule-3 (ICAM3)-grabbing non-integrin (DC-SIGN) protein as a novel receptor for a major allergen from house dust mite
title_sort retagging identifies dendritic cell-specific intercellular adhesion molecule-3 (icam3)-grabbing non-integrin (dc-sign) protein as a novel receptor for a major allergen from house dust mite
topic Immunology Glycobiology
Extracellular Matrices
url https://eprints.nottingham.ac.uk/2297/
https://eprints.nottingham.ac.uk/2297/
https://eprints.nottingham.ac.uk/2297/