Angiogenesis in the pathogenesis of inflammatory joint and lung diseases

This paper reviews hypotheses about roles of angiogenesis in the pathogenesis of inflammatory disease in two organs, the synovial joint and the lung. Neovascularisation is a fundamental process for growth and tissue repair after injury. Nevertheless, it may contribute to a variety of chronic inflamm...

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Main Authors: Walsh, D.A., Pearson, C.I.
Format: Article
Published: BioMEd Central 2001
Online Access:https://eprints.nottingham.ac.uk/126/
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author Walsh, D.A.
Pearson, C.I.
author_facet Walsh, D.A.
Pearson, C.I.
author_sort Walsh, D.A.
building Nottingham Research Data Repository
collection Online Access
description This paper reviews hypotheses about roles of angiogenesis in the pathogenesis of inflammatory disease in two organs, the synovial joint and the lung. Neovascularisation is a fundamental process for growth and tissue repair after injury. Nevertheless, it may contribute to a variety of chronic inflammatory diseases, including rheumatoid arthritis, osteoarthritis, asthma, and pulmonary fibrosis. Inflammation can promote angiogenesis, and new vessels may enhance tissue inflammation. Angiogenesis in inflammatory disease may also contribute to tissue growth, disordered tissue perfusion, abnormal ossification, and enhanced responses to normal or pathological stimuli. Angiogenesis inhibitors may reduce inflammation and may also help to restore appropriate tissue structure and function
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spelling nottingham-1262020-05-04T20:32:49Z https://eprints.nottingham.ac.uk/126/ Angiogenesis in the pathogenesis of inflammatory joint and lung diseases Walsh, D.A. Pearson, C.I. This paper reviews hypotheses about roles of angiogenesis in the pathogenesis of inflammatory disease in two organs, the synovial joint and the lung. Neovascularisation is a fundamental process for growth and tissue repair after injury. Nevertheless, it may contribute to a variety of chronic inflammatory diseases, including rheumatoid arthritis, osteoarthritis, asthma, and pulmonary fibrosis. Inflammation can promote angiogenesis, and new vessels may enhance tissue inflammation. Angiogenesis in inflammatory disease may also contribute to tissue growth, disordered tissue perfusion, abnormal ossification, and enhanced responses to normal or pathological stimuli. Angiogenesis inhibitors may reduce inflammation and may also help to restore appropriate tissue structure and function BioMEd Central 2001 Article PeerReviewed Walsh, D.A. and Pearson, C.I. (2001) Angiogenesis in the pathogenesis of inflammatory joint and lung diseases. Arthritis Research, 3 (3). pp. 147-153.
spellingShingle Walsh, D.A.
Pearson, C.I.
Angiogenesis in the pathogenesis of inflammatory joint and lung diseases
title Angiogenesis in the pathogenesis of inflammatory joint and lung diseases
title_full Angiogenesis in the pathogenesis of inflammatory joint and lung diseases
title_fullStr Angiogenesis in the pathogenesis of inflammatory joint and lung diseases
title_full_unstemmed Angiogenesis in the pathogenesis of inflammatory joint and lung diseases
title_short Angiogenesis in the pathogenesis of inflammatory joint and lung diseases
title_sort angiogenesis in the pathogenesis of inflammatory joint and lung diseases
url https://eprints.nottingham.ac.uk/126/