An exploration into common mechanisms of oxidative damage in models of demyelinating disease and neurotrauma

This thesis explored common mechanisms of oxidative damage in multiple sclerosis and neurotrauma and encompasses work that has been published in four journal articles. Initially, the effectiveness of a combinatorial ion channel inhibitor treatment was established in models of both disorders. The cup...

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Main Author: Toomey, Lillian May
Format: Thesis
Published: Curtin University 2022
Online Access:http://hdl.handle.net/20.500.11937/90843
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author Toomey, Lillian May
author_facet Toomey, Lillian May
author_sort Toomey, Lillian May
building Curtin Institutional Repository
collection Online Access
description This thesis explored common mechanisms of oxidative damage in multiple sclerosis and neurotrauma and encompasses work that has been published in four journal articles. Initially, the effectiveness of a combinatorial ion channel inhibitor treatment was established in models of both disorders. The cuprizone model of demyelinating disease was also optimised, followed by explorations into the relationship between oxidative DNA damage, cell proliferation and blood vessel dysfunction in models of neurotrauma and multiple sclerosis.
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format Thesis
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institution Curtin University Malaysia
institution_category Local University
last_indexed 2025-11-14T11:35:19Z
publishDate 2022
publisher Curtin University
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spelling curtin-20.500.11937-908432023-03-13T04:06:40Z An exploration into common mechanisms of oxidative damage in models of demyelinating disease and neurotrauma Toomey, Lillian May This thesis explored common mechanisms of oxidative damage in multiple sclerosis and neurotrauma and encompasses work that has been published in four journal articles. Initially, the effectiveness of a combinatorial ion channel inhibitor treatment was established in models of both disorders. The cuprizone model of demyelinating disease was also optimised, followed by explorations into the relationship between oxidative DNA damage, cell proliferation and blood vessel dysfunction in models of neurotrauma and multiple sclerosis. 2022 Thesis http://hdl.handle.net/20.500.11937/90843 Curtin University fulltext
spellingShingle Toomey, Lillian May
An exploration into common mechanisms of oxidative damage in models of demyelinating disease and neurotrauma
title An exploration into common mechanisms of oxidative damage in models of demyelinating disease and neurotrauma
title_full An exploration into common mechanisms of oxidative damage in models of demyelinating disease and neurotrauma
title_fullStr An exploration into common mechanisms of oxidative damage in models of demyelinating disease and neurotrauma
title_full_unstemmed An exploration into common mechanisms of oxidative damage in models of demyelinating disease and neurotrauma
title_short An exploration into common mechanisms of oxidative damage in models of demyelinating disease and neurotrauma
title_sort exploration into common mechanisms of oxidative damage in models of demyelinating disease and neurotrauma
url http://hdl.handle.net/20.500.11937/90843