A RNA interference screen identifies the protein phosphatase 2A subunit PR55γ as a stress-sensitive inhibitor of c-SRC

© 2007 Eichhorn et al. Protein Phosphatase type 2A (PP2A) represents a family of holoenzyme complexes with diverse biological activities. Specific holoenzyme complexes are thought to be deregulated during oncogenic transformation and oncogeneinduced signaling. Since most studies on the role of...

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Main Authors: Eichhorn, Pieter, Creyghton, M.P., Wilhelmsen, K., Van Dam, H., Bernards, R.
Format: Journal Article
Language:English
Published: PUBLIC LIBRARY SCIENCE 2007
Subjects:
Online Access:http://hdl.handle.net/20.500.11937/81903
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author Eichhorn, Pieter
Creyghton, M.P.
Wilhelmsen, K.
Van Dam, H.
Bernards, R.
author_facet Eichhorn, Pieter
Creyghton, M.P.
Wilhelmsen, K.
Van Dam, H.
Bernards, R.
author_sort Eichhorn, Pieter
building Curtin Institutional Repository
collection Online Access
description © 2007 Eichhorn et al. Protein Phosphatase type 2A (PP2A) represents a family of holoenzyme complexes with diverse biological activities. Specific holoenzyme complexes are thought to be deregulated during oncogenic transformation and oncogeneinduced signaling. Since most studies on the role of this phosphatase family have relied on the use of generic PP2A inhibitors, the contribution of individual PP2A holoenzyme complexes in PP2A-controlled signaling pathways is largely unclear. To gain insight into this, we have constructed a set of shRNA vectors targeting the individual PP2A regulatory subunits for suppression by RNA interference. Here, we identify PR55γ and PR55δ as inhibitors of c-Jun NH 2-terminal kinase (JNK) activation by UV irradiation. We show that PR55γ binds c-SRC and modulates the phosphorylation of serine 12 of c-SRC, a residue we demonstrate to be required for JNK activation by c-SRC. We also find that the physical interaction between PR55γ and c-SRC is sensitive to UV irradiation. Our data reveal a novel mechanism of c-SRC regulation whereby in response to stress c-SRC activity is regulated, at least in part, through loss of the interaction with its inhibitor, PR55γ.
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spelling curtin-20.500.11937-819032021-01-08T05:04:21Z A RNA interference screen identifies the protein phosphatase 2A subunit PR55γ as a stress-sensitive inhibitor of c-SRC Eichhorn, Pieter Creyghton, M.P. Wilhelmsen, K. Van Dam, H. Bernards, R. Science & Technology Life Sciences & Biomedicine Genetics & Heredity GROWTH-FACTOR RECEPTOR N-TERMINAL KINASE ADENOVIRUS E4ORF4 PROTEIN MIDDLE TUMOR-ANTIGEN OKADAIC ACID BREAST-CANCER SERINE/THREONINE PHOSPHATASE SIGNAL-TRANSDUCTION CSF-1 RECEPTOR CELL-GROWTH © 2007 Eichhorn et al. Protein Phosphatase type 2A (PP2A) represents a family of holoenzyme complexes with diverse biological activities. Specific holoenzyme complexes are thought to be deregulated during oncogenic transformation and oncogeneinduced signaling. Since most studies on the role of this phosphatase family have relied on the use of generic PP2A inhibitors, the contribution of individual PP2A holoenzyme complexes in PP2A-controlled signaling pathways is largely unclear. To gain insight into this, we have constructed a set of shRNA vectors targeting the individual PP2A regulatory subunits for suppression by RNA interference. Here, we identify PR55γ and PR55δ as inhibitors of c-Jun NH 2-terminal kinase (JNK) activation by UV irradiation. We show that PR55γ binds c-SRC and modulates the phosphorylation of serine 12 of c-SRC, a residue we demonstrate to be required for JNK activation by c-SRC. We also find that the physical interaction between PR55γ and c-SRC is sensitive to UV irradiation. Our data reveal a novel mechanism of c-SRC regulation whereby in response to stress c-SRC activity is regulated, at least in part, through loss of the interaction with its inhibitor, PR55γ. 2007 Journal Article http://hdl.handle.net/20.500.11937/81903 10.1371/journal.pgen.0030218 English http://creativecommons.org/licenses/by/4.0/ PUBLIC LIBRARY SCIENCE fulltext
spellingShingle Science & Technology
Life Sciences & Biomedicine
Genetics & Heredity
GROWTH-FACTOR RECEPTOR
N-TERMINAL KINASE
ADENOVIRUS E4ORF4 PROTEIN
MIDDLE TUMOR-ANTIGEN
OKADAIC ACID
BREAST-CANCER
SERINE/THREONINE PHOSPHATASE
SIGNAL-TRANSDUCTION
CSF-1 RECEPTOR
CELL-GROWTH
Eichhorn, Pieter
Creyghton, M.P.
Wilhelmsen, K.
Van Dam, H.
Bernards, R.
A RNA interference screen identifies the protein phosphatase 2A subunit PR55γ as a stress-sensitive inhibitor of c-SRC
title A RNA interference screen identifies the protein phosphatase 2A subunit PR55γ as a stress-sensitive inhibitor of c-SRC
title_full A RNA interference screen identifies the protein phosphatase 2A subunit PR55γ as a stress-sensitive inhibitor of c-SRC
title_fullStr A RNA interference screen identifies the protein phosphatase 2A subunit PR55γ as a stress-sensitive inhibitor of c-SRC
title_full_unstemmed A RNA interference screen identifies the protein phosphatase 2A subunit PR55γ as a stress-sensitive inhibitor of c-SRC
title_short A RNA interference screen identifies the protein phosphatase 2A subunit PR55γ as a stress-sensitive inhibitor of c-SRC
title_sort rna interference screen identifies the protein phosphatase 2a subunit pr55γ as a stress-sensitive inhibitor of c-src
topic Science & Technology
Life Sciences & Biomedicine
Genetics & Heredity
GROWTH-FACTOR RECEPTOR
N-TERMINAL KINASE
ADENOVIRUS E4ORF4 PROTEIN
MIDDLE TUMOR-ANTIGEN
OKADAIC ACID
BREAST-CANCER
SERINE/THREONINE PHOSPHATASE
SIGNAL-TRANSDUCTION
CSF-1 RECEPTOR
CELL-GROWTH
url http://hdl.handle.net/20.500.11937/81903