The role of signal transducer and activator of transcription 3 (STAT3) and its targeted inhibition in hematological malignancies

© 2018 by the authors. Licensee MDPI, Basel, Switzerland. Signal transducer and activator of transcription 3 (STAT3), a member of the STAT protein family, can be phosphorylated by receptor-associated Janus kinases (JAKs) in response to stimulation by cytokines and growth factors. It forms homo-or he...

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Main Authors: Arora, L., Kumar, Alan Prem, Arfuso, Frank, Chng, W., Sethi, G.
Format: Journal Article
Published: MDPI AG 2018
Online Access:http://hdl.handle.net/20.500.11937/72647
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author Arora, L.
Kumar, Alan Prem
Arfuso, Frank
Chng, W.
Sethi, G.
author_facet Arora, L.
Kumar, Alan Prem
Arfuso, Frank
Chng, W.
Sethi, G.
author_sort Arora, L.
building Curtin Institutional Repository
collection Online Access
description © 2018 by the authors. Licensee MDPI, Basel, Switzerland. Signal transducer and activator of transcription 3 (STAT3), a member of the STAT protein family, can be phosphorylated by receptor-associated Janus kinases (JAKs) in response to stimulation by cytokines and growth factors. It forms homo-or heterodimers that can translocate to the cell nucleus where they act as transcription activators. Constitutive activation of STAT3 has been found to be associated with initiation and progression of various cancers. It can exert proliferative as well as anti-apoptotic effects. This review focuses on the role of STAT3 in pathogenesis i.e., proliferation, differentiation, migration, and apoptosis of hematological malignancies viz. leukemia, lymphoma and myeloma, and briefly highlights the potential therapeutic approaches developed against STAT3 activation pathway.
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institution Curtin University Malaysia
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publishDate 2018
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spelling curtin-20.500.11937-726472021-01-05T08:07:08Z The role of signal transducer and activator of transcription 3 (STAT3) and its targeted inhibition in hematological malignancies Arora, L. Kumar, Alan Prem Arfuso, Frank Chng, W. Sethi, G. © 2018 by the authors. Licensee MDPI, Basel, Switzerland. Signal transducer and activator of transcription 3 (STAT3), a member of the STAT protein family, can be phosphorylated by receptor-associated Janus kinases (JAKs) in response to stimulation by cytokines and growth factors. It forms homo-or heterodimers that can translocate to the cell nucleus where they act as transcription activators. Constitutive activation of STAT3 has been found to be associated with initiation and progression of various cancers. It can exert proliferative as well as anti-apoptotic effects. This review focuses on the role of STAT3 in pathogenesis i.e., proliferation, differentiation, migration, and apoptosis of hematological malignancies viz. leukemia, lymphoma and myeloma, and briefly highlights the potential therapeutic approaches developed against STAT3 activation pathway. 2018 Journal Article http://hdl.handle.net/20.500.11937/72647 10.3390/cancers10090327 http://creativecommons.org/licenses/by/4.0/ MDPI AG fulltext
spellingShingle Arora, L.
Kumar, Alan Prem
Arfuso, Frank
Chng, W.
Sethi, G.
The role of signal transducer and activator of transcription 3 (STAT3) and its targeted inhibition in hematological malignancies
title The role of signal transducer and activator of transcription 3 (STAT3) and its targeted inhibition in hematological malignancies
title_full The role of signal transducer and activator of transcription 3 (STAT3) and its targeted inhibition in hematological malignancies
title_fullStr The role of signal transducer and activator of transcription 3 (STAT3) and its targeted inhibition in hematological malignancies
title_full_unstemmed The role of signal transducer and activator of transcription 3 (STAT3) and its targeted inhibition in hematological malignancies
title_short The role of signal transducer and activator of transcription 3 (STAT3) and its targeted inhibition in hematological malignancies
title_sort role of signal transducer and activator of transcription 3 (stat3) and its targeted inhibition in hematological malignancies
url http://hdl.handle.net/20.500.11937/72647