Cluster-assembled zirconia substrates promote long-term differentiation and functioning of human islets of Langerhans

Ex vivo expansion and differentiation of human pancreatic ß-cell are enabling steps of paramount importance for accelerating the development of therapies for diabetes. The success of regenerative strategies depends on their ability to reproduce the chemical and biophysical properties of the microenv...

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Main Authors: Galli, A., Maffioli, E., Sogne, E., Moretti, S., Di Cairano, E., Negri, A., Nonnis, S., Norata, Giuseppe, Bonacina, F., Borghi, F., Podestà, A., Bertuzzi, F., Milani, P., Lenardi, C., Tedeschi, G., Perego, C.
Format: Journal Article
Published: Nature Publishing Group 2018
Online Access:http://hdl.handle.net/20.500.11937/70116
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author Galli, A.
Maffioli, E.
Sogne, E.
Moretti, S.
Di Cairano, E.
Negri, A.
Nonnis, S.
Norata, Giuseppe
Bonacina, F.
Borghi, F.
Podestà, A.
Bertuzzi, F.
Milani, P.
Lenardi, C.
Tedeschi, G.
Perego, C.
author_facet Galli, A.
Maffioli, E.
Sogne, E.
Moretti, S.
Di Cairano, E.
Negri, A.
Nonnis, S.
Norata, Giuseppe
Bonacina, F.
Borghi, F.
Podestà, A.
Bertuzzi, F.
Milani, P.
Lenardi, C.
Tedeschi, G.
Perego, C.
author_sort Galli, A.
building Curtin Institutional Repository
collection Online Access
description Ex vivo expansion and differentiation of human pancreatic ß-cell are enabling steps of paramount importance for accelerating the development of therapies for diabetes. The success of regenerative strategies depends on their ability to reproduce the chemical and biophysical properties of the microenvironment in which ß-cells develop, proliferate and function. In this paper we focus on the biophysical properties of the extracellular environment and exploit the cluster-assembled zirconia substrates with tailored roughness to mimic the nanotopography of the extracellular matrix. We demonstrate that ß-cells can perceive nanoscale features of the substrate and can convert these stimuli into mechanotransductive processes which promote long-term in vitro human islet culture, thus preserving ß-cell differentiation and function. Proteomic and quantitative immunofluorescence analyses demonstrate that the process is driven by nanoscale topography, via remodelling of the actin cytoskeleton and nuclear architecture. These modifications activate a transcriptional program which stimulates an adaptive metabolic glucose response. Engineered cluster-assembled substrates coupled with proteomic approaches may provide a useful strategy for identifying novel molecular targets for treating diabetes mellitus and for enhancing tissue engineering in order to improve the efficacy of islet cell transplantation therapies.
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spelling curtin-20.500.11937-701162018-08-23T03:34:44Z Cluster-assembled zirconia substrates promote long-term differentiation and functioning of human islets of Langerhans Galli, A. Maffioli, E. Sogne, E. Moretti, S. Di Cairano, E. Negri, A. Nonnis, S. Norata, Giuseppe Bonacina, F. Borghi, F. Podestà, A. Bertuzzi, F. Milani, P. Lenardi, C. Tedeschi, G. Perego, C. Ex vivo expansion and differentiation of human pancreatic ß-cell are enabling steps of paramount importance for accelerating the development of therapies for diabetes. The success of regenerative strategies depends on their ability to reproduce the chemical and biophysical properties of the microenvironment in which ß-cells develop, proliferate and function. In this paper we focus on the biophysical properties of the extracellular environment and exploit the cluster-assembled zirconia substrates with tailored roughness to mimic the nanotopography of the extracellular matrix. We demonstrate that ß-cells can perceive nanoscale features of the substrate and can convert these stimuli into mechanotransductive processes which promote long-term in vitro human islet culture, thus preserving ß-cell differentiation and function. Proteomic and quantitative immunofluorescence analyses demonstrate that the process is driven by nanoscale topography, via remodelling of the actin cytoskeleton and nuclear architecture. These modifications activate a transcriptional program which stimulates an adaptive metabolic glucose response. Engineered cluster-assembled substrates coupled with proteomic approaches may provide a useful strategy for identifying novel molecular targets for treating diabetes mellitus and for enhancing tissue engineering in order to improve the efficacy of islet cell transplantation therapies. 2018 Journal Article http://hdl.handle.net/20.500.11937/70116 10.1038/s41598-018-28019-3 http://creativecommons.org/licenses/by/4.0/ Nature Publishing Group fulltext
spellingShingle Galli, A.
Maffioli, E.
Sogne, E.
Moretti, S.
Di Cairano, E.
Negri, A.
Nonnis, S.
Norata, Giuseppe
Bonacina, F.
Borghi, F.
Podestà, A.
Bertuzzi, F.
Milani, P.
Lenardi, C.
Tedeschi, G.
Perego, C.
Cluster-assembled zirconia substrates promote long-term differentiation and functioning of human islets of Langerhans
title Cluster-assembled zirconia substrates promote long-term differentiation and functioning of human islets of Langerhans
title_full Cluster-assembled zirconia substrates promote long-term differentiation and functioning of human islets of Langerhans
title_fullStr Cluster-assembled zirconia substrates promote long-term differentiation and functioning of human islets of Langerhans
title_full_unstemmed Cluster-assembled zirconia substrates promote long-term differentiation and functioning of human islets of Langerhans
title_short Cluster-assembled zirconia substrates promote long-term differentiation and functioning of human islets of Langerhans
title_sort cluster-assembled zirconia substrates promote long-term differentiation and functioning of human islets of langerhans
url http://hdl.handle.net/20.500.11937/70116