SREBP-1c as a molecular bridge between lipogenesis and cell cycle progression of clear cell renal carcinoma

Sterol regulatory element binding protein 1c (SREBP-1c) promotes lipogenesis and tumor growth in various cancers. It is well known that clear cell renal cell carcinoma (ccRCC), a major subtype of the kidney cancers, exhibits elevated lipid accumulation. However, it has not been fully understood how...

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Main Authors: Sethi, G., Shanmugam, M., Kumar, Alan Prem
Format: Journal Article
Published: 2017
Online Access:http://hdl.handle.net/20.500.11937/62753
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author Sethi, G.
Shanmugam, M.
Kumar, Alan Prem
author_facet Sethi, G.
Shanmugam, M.
Kumar, Alan Prem
author_sort Sethi, G.
building Curtin Institutional Repository
collection Online Access
description Sterol regulatory element binding protein 1c (SREBP-1c) promotes lipogenesis and tumor growth in various cancers. It is well known that clear cell renal cell carcinoma (ccRCC), a major subtype of the kidney cancers, exhibits elevated lipid accumulation. However, it has not been fully understood how lipid metabolism might be associated with cell cycle regulation in ccRCC. In a recent issue, Lee et al. (Molecular and Cellular Biology (2017) pii: MCB.00265-17) demonstrate that SREBP-1c is up-regulated in ccRCC by ring finger protein 20 (RNF20) down-regulation, leading to aberrant lipid storage and pituitary tumor transforming gene 1 (PTTG1)-dependent cell cycle progression. These findings suggest that SREBP-1c serves as a molecular bridge between lipid metabolism and cell cycle control in ccRCC tumorigenesis.
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spelling curtin-20.500.11937-627532018-02-01T05:58:35Z SREBP-1c as a molecular bridge between lipogenesis and cell cycle progression of clear cell renal carcinoma Sethi, G. Shanmugam, M. Kumar, Alan Prem Sterol regulatory element binding protein 1c (SREBP-1c) promotes lipogenesis and tumor growth in various cancers. It is well known that clear cell renal cell carcinoma (ccRCC), a major subtype of the kidney cancers, exhibits elevated lipid accumulation. However, it has not been fully understood how lipid metabolism might be associated with cell cycle regulation in ccRCC. In a recent issue, Lee et al. (Molecular and Cellular Biology (2017) pii: MCB.00265-17) demonstrate that SREBP-1c is up-regulated in ccRCC by ring finger protein 20 (RNF20) down-regulation, leading to aberrant lipid storage and pituitary tumor transforming gene 1 (PTTG1)-dependent cell cycle progression. These findings suggest that SREBP-1c serves as a molecular bridge between lipid metabolism and cell cycle control in ccRCC tumorigenesis. 2017 Journal Article http://hdl.handle.net/20.500.11937/62753 10.1042/BSR20171270 unknown
spellingShingle Sethi, G.
Shanmugam, M.
Kumar, Alan Prem
SREBP-1c as a molecular bridge between lipogenesis and cell cycle progression of clear cell renal carcinoma
title SREBP-1c as a molecular bridge between lipogenesis and cell cycle progression of clear cell renal carcinoma
title_full SREBP-1c as a molecular bridge between lipogenesis and cell cycle progression of clear cell renal carcinoma
title_fullStr SREBP-1c as a molecular bridge between lipogenesis and cell cycle progression of clear cell renal carcinoma
title_full_unstemmed SREBP-1c as a molecular bridge between lipogenesis and cell cycle progression of clear cell renal carcinoma
title_short SREBP-1c as a molecular bridge between lipogenesis and cell cycle progression of clear cell renal carcinoma
title_sort srebp-1c as a molecular bridge between lipogenesis and cell cycle progression of clear cell renal carcinoma
url http://hdl.handle.net/20.500.11937/62753