Mitochondrial function and increased convective O2 transport: Implications for the assessment of mitochondrial respiration in vivo

Although phosphorus magnetic resonance spectroscopy ( 31 P-MRS)- based evidence suggests that in vivo peak mitochondrial respiration rate in young untrained adults is limited by the intrinsic mitochondrial capacity of ATP synthesis, it remains unknown whether a large, locally targeted increase in co...

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Main Authors: Layec, G., Haseler, Luke, Trinity, J., Hart, C., Liu, X., Fur, Y., Jeong, E., Richardson, R.
Format: Journal Article
Published: The American Physiological Society 2013
Online Access:http://hdl.handle.net/20.500.11937/58274
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author Layec, G.
Haseler, Luke
Trinity, J.
Hart, C.
Liu, X.
Fur, Y.
Jeong, E.
Richardson, R.
author_facet Layec, G.
Haseler, Luke
Trinity, J.
Hart, C.
Liu, X.
Fur, Y.
Jeong, E.
Richardson, R.
author_sort Layec, G.
building Curtin Institutional Repository
collection Online Access
description Although phosphorus magnetic resonance spectroscopy ( 31 P-MRS)- based evidence suggests that in vivo peak mitochondrial respiration rate in young untrained adults is limited by the intrinsic mitochondrial capacity of ATP synthesis, it remains unknown whether a large, locally targeted increase in convective O2 delivery would alter this interpretation. Consequently, we examined the effect of superimposing reactive hyperemia (RH), induced by a period of brief ischemia during the last minute of exercise, on oxygen delivery and mitochondrial function in the calf muscle of nine young adults compared with free-flow conditions (FF). To this aim, we used an integrative experimental approach combining 31 P-MRS, Doppler ultrasound imaging, and near-infrared spectroscopy. Limb blood flow [area under the curve (AUC), 1.4 ± 0.8 liters in FF and 2.5 ± 0.3 liters in RH, P < 0.01] and convective O2 delivery (AUC, 0.30 ± 0.16 liters in FF and 0.54 ± 0.05 liters in RH, P < 0.01), were significantly increased in RH compared with FF. RH was also associated with significantly higher capillary blood flow (P < 0.05) and faster tissue reoxygenation mean response times (70 ± 15 s in FF and 24 ± 15 s in RH, P < 0.05). This resulted in a 43% increase in estimated peak mitochondrial ATP synthesis rate (29 ± 13 mM/min in FF and 41 ± 14 mM/min in RH, P < 0.05) whereas the phosphocreatine (PCr) recovery time constant in RH was not significantly different (P = 0.22). This comprehensive assessment of local skeletal muscle O2 availability and utilization in untrained subjects reveals that mitochondrial function, assessed in vivo by 31 P-MRS, is limited by convective O2 delivery rather than an intrinsic mitochondrial limitation. © 2013 the American Physiological Society.
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spelling curtin-20.500.11937-582742023-02-22T06:24:20Z Mitochondrial function and increased convective O2 transport: Implications for the assessment of mitochondrial respiration in vivo Layec, G. Haseler, Luke Trinity, J. Hart, C. Liu, X. Fur, Y. Jeong, E. Richardson, R. Although phosphorus magnetic resonance spectroscopy ( 31 P-MRS)- based evidence suggests that in vivo peak mitochondrial respiration rate in young untrained adults is limited by the intrinsic mitochondrial capacity of ATP synthesis, it remains unknown whether a large, locally targeted increase in convective O2 delivery would alter this interpretation. Consequently, we examined the effect of superimposing reactive hyperemia (RH), induced by a period of brief ischemia during the last minute of exercise, on oxygen delivery and mitochondrial function in the calf muscle of nine young adults compared with free-flow conditions (FF). To this aim, we used an integrative experimental approach combining 31 P-MRS, Doppler ultrasound imaging, and near-infrared spectroscopy. Limb blood flow [area under the curve (AUC), 1.4 ± 0.8 liters in FF and 2.5 ± 0.3 liters in RH, P < 0.01] and convective O2 delivery (AUC, 0.30 ± 0.16 liters in FF and 0.54 ± 0.05 liters in RH, P < 0.01), were significantly increased in RH compared with FF. RH was also associated with significantly higher capillary blood flow (P < 0.05) and faster tissue reoxygenation mean response times (70 ± 15 s in FF and 24 ± 15 s in RH, P < 0.05). This resulted in a 43% increase in estimated peak mitochondrial ATP synthesis rate (29 ± 13 mM/min in FF and 41 ± 14 mM/min in RH, P < 0.05) whereas the phosphocreatine (PCr) recovery time constant in RH was not significantly different (P = 0.22). This comprehensive assessment of local skeletal muscle O2 availability and utilization in untrained subjects reveals that mitochondrial function, assessed in vivo by 31 P-MRS, is limited by convective O2 delivery rather than an intrinsic mitochondrial limitation. © 2013 the American Physiological Society. 2013 Journal Article http://hdl.handle.net/20.500.11937/58274 10.1152/japplphysiol.00257.2013 The American Physiological Society unknown
spellingShingle Layec, G.
Haseler, Luke
Trinity, J.
Hart, C.
Liu, X.
Fur, Y.
Jeong, E.
Richardson, R.
Mitochondrial function and increased convective O2 transport: Implications for the assessment of mitochondrial respiration in vivo
title Mitochondrial function and increased convective O2 transport: Implications for the assessment of mitochondrial respiration in vivo
title_full Mitochondrial function and increased convective O2 transport: Implications for the assessment of mitochondrial respiration in vivo
title_fullStr Mitochondrial function and increased convective O2 transport: Implications for the assessment of mitochondrial respiration in vivo
title_full_unstemmed Mitochondrial function and increased convective O2 transport: Implications for the assessment of mitochondrial respiration in vivo
title_short Mitochondrial function and increased convective O2 transport: Implications for the assessment of mitochondrial respiration in vivo
title_sort mitochondrial function and increased convective o2 transport: implications for the assessment of mitochondrial respiration in vivo
url http://hdl.handle.net/20.500.11937/58274